C. V. Os

Nephrogenic Diabetes Insipidus: Aquaporin-2 Defect

P. Deen, N. Knoers, C. V. Os, 2009 .

An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex.

P. Sluijs, D. Bichet, P. Deen, 1998, The Journal of clinical investigation.

Requirement of human renal water channel aquaporin-2 for vasopressin-dependent concentration of urine.

B. V. van Oost, B. Wieringa, P. Deen, 1994, Science.

Adenosine triphosphate-dependent transport of anionic conjugates by the rabbit multidrug resistance-associated protein Mrp2 expressed in insect cells.

P. Deen, F. Russel, J. B. Koenderink, 1998, Molecular pharmacology.

The Subcellular Localization of an Aquaporin-2 Tetramer Depends on the Stoichiometry of Phosphorylated and Nonphosphorylated Monomers

P. Deen, C. van Os, E. Kamsteeg, 2000, The Journal of cell biology.

Urinary content of aquaporin 1 and 2 in nephrogenic diabetes insipidus.

P. Deen, C. van Os, R. A. van Aubel, 1996, Journal of the American Society of Nephrology : JASN.

New mutations in the AQP2 gene in nephrogenic diabetes insipidus resulting in functional but misrouted water channels.

P. Deen, N. Knoers, L. Monnens, 1997, Journal of the American Society of Nephrology : JASN.