Gisela Schubert

The Tumor Necrosis Factor-related Apoptosis-inducing Ligand Receptors TRAIL-R1 and TRAIL-R2 Have Distinct Cross-linking Requirements for Initiation of Apoptosis and Are Non-redundant in JNK Activation*

J. Tschopp, P. Scheurich, H. Wajant, 2000, The Journal of Biological Chemistry.

Inhibition of Death Receptor-mediated Gene Induction by a Cycloheximide-sensitive Factor Occurs at the Level of or Upstream of Fas-associated Death Domain Protein (FADD)*

P. Scheurich, H. Wajant, M. Grell, 2000, The Journal of Biological Chemistry.

TWEAK can induce cell death via endogenous TNF and TNF receptor 1

J. Tschopp, P. Scheurich, H. Wajant, 1999, European journal of immunology.

TRAIL/Apo2L Activates c-Jun NH2-terminal Kinase (JNK) via Caspase-dependent and Caspase-independent Pathways*

P. Scheurich, H. Wajant, M. Grell, 1998, The Journal of Biological Chemistry.

Dominant-negative FADD inhibits TNFR60-, Fas/Apo1- and TRAIL-R/Apo2-mediated cell death but not gene induction

P. Scheurich, H. Wajant, M. Grell, 1998, Current Biology.

The Human Tumor Necrosis Factor (TNF) Receptor-associated Factor 1 Gene (TRAF1) Is Up-regulated by Cytokines of the TNF Ligand Family and Modulates TNF-induced Activation of NF-κB and c-Jun N-terminal Kinase*

Peter Scheurich, P. Scheurich, R. Schmid, 1999, The Journal of Biological Chemistry.