A. Greiner

发表

Additional Evidence Implicating GPD1L in the Pathogenesis of Brugada Syndrome in A Large Multigenerational Family

H. Mehdi, A. Greiner, C. Cevan, 2022, medRxiv.

Rethinking medical oaths using the Physician Charter and ethical virtues

Alexander M Greiner, L. Kaldjian, A. Greiner, 2018, Medical education.

SUMOylation of the Cardiac Sodium Channel NaV1.5 Modifies Inward Current and Cardiac Excitability

K. Irani, A. Vikram, B. London, 2022, bioRxiv.

Modulation of the Cardiac Sodium Channel NaV1.5 Peak and Late Currents by NAD+ Precursors

Mark S. Schmidt, C. Brenner, K. Irani, 2020, bioRxiv.

Functional Analysis of a De Novo ACTB Mutation in a Patient with Atypical Baraitser–Winter Syndrome

Julie C. Sapp, L. Biesecker, J. Johnston, 2013, Human Mutation.

: Additional Evidence Implicating GPD1L in the Pathogenesis of Brugada Syndrome in A Large Multi-1 generational Family

Rebecca Gutmann, A. Greiner, C. Cevan, 2022 .

: Additional Evidence Implicating GPD1L in the Pathogenesis of Brugada Syndrome in A Large Multi-1 generational Family

Rebecca Gutmann, A. Greiner, C. Cevan, 2022 .

SUMOylation of the Cardiac Sodium Channel NaV1.5 Modifies Inward Current and Cardiac Excitability.

K. Irani, A. Vikram, B. London, 2023, Heart rhythm.

The role of GPD1L, a sodium channel interacting gene, in the pathogenesis of Brugada Syndrome

Rebecca Gutmann, A. Greiner, C. Cevan, 2024, Frontiers in medicine.