Jayden A. Clark

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

J. Chuckowree, C. Blizzard, T. Dickson, 2020, Neural regeneration research.

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1G93A mouse model of amyotrophic lateral sclerosis

C. Blizzard, Katherine A. Southam, A. King, 2016, Journal of Chemical Neuroanatomy.

A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease

J. Chuckowree, C. Blizzard, T. Dickson, 2016, Front. Cell. Neurosci..

Neurons to Excitotoxicity Determine the Selective Vulnerability of Motor GluR2-Dependent Properties of AMPA Receptors

G. Callewaert, C. Blizzard, P. Damme, 2015 .

Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis – vulnerability of lower motor neurons to proximal excitotoxicity

C. Blizzard, Katherine A. Southam, A. King, 2015, Disease Models & Mechanisms.

Epothilone D alters normal growth, viability and microtubule dependent intracellular functions of cortical neurons in vitro

C. Blizzard, T. Dickson, J. A. Clark, 2020, Scientific Reports.

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1G93A mouse model of amyotrophic lateral sclerosis

C. Blizzard, Katherine A. Southam, A. King, 2016, Journal of Chemical Neuroanatomy.

Epothilone D accelerates disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis

J. Chuckowree, C. Blizzard, T. Dickson, 2018, Neuropathology and applied neurobiology.