Isabel Hernández-Porras

发表

The impact of the genetic background in the Noonan syndrome phenotype induced by K-RasV14I

C. Guerra, A. J. Schuhmacher, Isabel Hernández-Porras, 2015, Rare diseases.

Transcriptome Analysis in Prenatal IGF1-Deficient Mice Identifies Molecular Pathways and Target Genes Involved in Distal Lung Differentiation

Nuria Moreno-Barriuso, J. Pichel, I. López, 2013, PloS one.

EGF receptor signaling is essential for k-ras oncogene-driven pancreatic ductal adenocarcinoma.

M. Barbacid, C. Guerra, C. Navas, 2012, Cancer cell.

Noonan syndrome: lessons learned from genetically modified mouse models

C. Guerra, Raquel García-Medina, A. J. Schuhmacher, 2017, Expert review of endocrinology & metabolism.

H-Ras and K-Ras Oncoproteins Induce Different Tumor Spectra When Driven by the Same Regulatory Sequences.

M. Barbacid, C. Guerra, M. Drosten, 2017, Cancer research.

Modeling RASopathies with Genetically Modified Mouse Models.

C. Guerra, Isabel Hernández-Porras, 2017, Methods in molecular biology.

K‐RasV14I‐induced Noonan syndrome predisposes to tumour development in mice

Beatriz Jiménez, C. Guerra, Raquel García-Medina, 2016, The Journal of pathology.

Pancreatitis-induced inflammation contributes to pancreatic cancer by inhibiting oncogene-induced senescence.

M. Barbacid, C. Guerra, M. Serrano, 2011, Cancer cell.

K-RasV14I recapitulates Noonan syndrome in mice

Manuel Desco, Lorena Cussó, M. Barbacid, 2014, Proceedings of the National Academy of Sciences.

EGF receptor signaling is essential for k-ras oncogene-driven pancreatic ductal adenocarcinoma.

M. Barbacid, C. Guerra, C. Navas, 2012, Cancer cell.