B Stoyanov

Phosphatidylinositol 3-kinase gamma mediates shear stress-dependent activation of JNK in endothelial cells.

H. Jo, R. Wetzker, H. Park, 1998, The American journal of physiology.

Cloning and characterization of a G protein-activated human phosphoinositide-3 kinase.

S. Volinia, B. Vanhaesebroeck, B. Nürnberg, 1995, Science.

Both SH2 Domains Are Involved in Interaction of SHP-1 with the Epidermal Growth Factor Receptor but Cannot Confer Receptor-directed Activity to SHP-1/SHP-2 Chimera*

A. Ullrich, S. Tomić, T. Tenev, 1997, The Journal of Biological Chemistry.

Tumor necrosis factor-alpha- and hyperglycemia-induced insulin resistance. Evidence for different mechanisms and different effects on insulin signaling.

A. Ullrich, H. Häring, E. Capp, 1996, The Journal of clinical investigation.

Serine residues 1177/78/82 of the insulin receptor are required for substrate phosphorylation but not autophosphorylation.

A. Ullrich, H. Klein, J. Krützfeldt, 2000, Diabetes.

Impact of mutations at different serine residues on the tyrosine kinase activity of the insulin receptor.

H. Häring, V. Strack, B. Bossenmaier, 1997, Biochemical and biophysical research communications.

The inhibitory effect of 2‐deoxyglucose on insulin receptor autophosphorylation does not depend on known serine phosphorylation sites or other conserved serine residues of the receptor β‐subunit

H. Häring, V. Strack, B. Bossenmaier, 1999, FEBS letters.