B. Oen

发表

The effect of anions on bound acetylcholine in frog sartorius muscle.

P. Molenaar, R. L. Polak, B. Ceccarelli, 1989, The Journal of physiology.

A myasthenic syndrome with congenital paucity of secondary synaptic clefts: CPSC Syndrome

G. Hageman, P. Molenaar, F. Jennekens, 1988, Muscle & nerve.

A Congenital Myasthenic Disorder with Paucity of Secondary Synaptic Clefts: Deficiency and Altered Distribution of Acetylcholine Receptors a

P. Molenaar, F. Jennekens, L. Smit, 1987, Annals of the New York Academy of Sciences.

Congenital paucity of secondary synaptic clefts (CPSC) syndrome in 2 adult sibs

J. Wokke, P. Molenaar, F. Jennekens, 1989, Neurology.

The slow channel syndrome. Two new cases.

A. Vincent, J. Wokke, T. V. Weerden, 1987, Brain : a journal of neurology.

A non-immunogenic myasthenia gravis model and its application in a study of transsynaptic regulation at the neuromuscular junction.

P. Molenaar, J. Plomp, F. Jennekens, 1991, European journal of pharmacology.

Analysis of quantal acetylcholine noise at end‐plates of frog muscle during rapid transmitter secretion.

P. Molenaar, B. Oen, 1988, The Journal of physiology.

Surplus acetylcholine and acetylcholine release in the rat diaphragm.

R. L. Polak, P. Molenaar, B. Oen, 1987, The Journal of physiology.

Effect of chloride ions on giant miniature end‐plate potentials at the frog neuromuscular junction.

R. L. Polak, P. Molenaar, B. Oen, 1987, The Journal of physiology.