F. Tagliavini

发表

In Situ Tissue Labeling of Cerebral Amyloid Using HIV-Related Tat Peptide

M. Salmona, A. Cagnotto, G. Giaccone, 2018, Molecular Neurobiology.

Intracellular calcium rise through L-type calcium channels, as molecular mechanism for prion protein fragment 106-126-induced astroglial proliferation.

G. Forloni, M. Salmona, O. Bugiani, 1996, Biochemical and biophysical research communications.

Susceptible genes and disease mechanisms identified in frontotemporal dementia and frontotemporal dementia with Amyotrophic Lateral Sclerosis by DNA-methylation and GWAS

E. Haan, P. Pietrini, R. Petersen, 2017, Scientific Reports.

Measurement of intracellular calcium levels by the fluorescent Ca(2+) indicator Calcium-Green.

M. Salmona, O. Bugiani, C. Fabrizi, 2000, Brain research. Brain research protocols.

Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: a study of the BrainNet Europe consortium

I. Ferrer, H. Budka, S. Al-Sarraj, 2008, Acta Neuropathologica.

PrP Peptides as a Tool to Investigate the Pathogenesis of Prion Protein Amyloidoses

B. Ghetti, G. Forloni, F. Tagliavini, 1998 .

Methods for Studying Prion Protein Amyloid

B. Ghetti, G. Forloni, F. Tagliavini, 1996 .

CXCR4 involvement in neurodegenerative diseases

O. Andreassen, G. Schellenberg, A. Dale, 2017, bioRxiv.

Genetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasia

P. Pietrini, R. Desikan, J. Trojanowski, 2019, Scientific Reports.