Judy U Earley

发表

Experimental Modeling Supports a Role for MyBP-HL as a Novel Myofilament Component in Arrhythmia and Dilated Cardiomyopathy

Lorenzo L. Pesce, E. A. Waters, Andy H. Vo, 2017, Circulation.

Ets1 is required for proper migration and differentiation of the cardiac neural crest

Gene Kim, E. Svensson, A. Mackinnon, 2010, Development.

Impaired muscle growth and response to insulin-like growth factor 1 in dysferlin-mediated muscular dystrophy.

E. McNally, J. Fahrenbach, P. Pytel, 2011, Human molecular genetics.

Translational Control of FOG-2 Expression in Cardiomyocytes by MicroRNA-130a

Gene Kim, E. Svensson, S. Samant, 2009, PloS one.

FOG-2 attenuates endothelial-to-mesenchymal transformation in the endocardial cushions of the developing heart.

E. Svensson, J. Earley, A. Flagg, 2007, Developmental biology.

Dysferlin and myoferlin regulate transverse tubule formation and glycerol sensitivity.

A. E. Rossi, G. Walter, R. Vohra, 2014, The American journal of pathology.

Myoferlin is required for insulin‐like growth factor response and muscle growth

E. McNally, P. Pytel, J. Earley, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

19th International Symposium on Calcium Binding Proteins and Calcium Function In Health and Disease

A. Daugherty, D. Rateri, M. Hofmann, 2015 .

Disruption of nesprin-1 produces an Emery Dreifuss muscular dystrophy-like phenotype in mice.

Yuan Zhang, D. Hodzic, J. Holaska, 2008, Human molecular genetics.

Normal myoblast fusion requires myoferlin

D. Davis, E. McNally, J. Earley, 2005, Development.

FOG-2 mediated recruitment of the NuRD complex regulates cardiomyocyte proliferation during heart development.

M. Broman, E. Svensson, Zhiguang Gao, 2011, Developmental biology.

Transplanted hematopoietic stem cells demonstrate impaired sarcoglycan expression after engraftment into cardiac and skeletal muscle.

E. McNally, A. Ma, M. Chien, 2004, The Journal of clinical investigation.

Nesprin-1 mutations in human and murine cardiomyopathy.

Yuan Zhang, Gene Kim, J. Holaska, 2010, Journal of molecular and cellular cardiology.

Genetic modifiers of muscular dystrophy act on sarcolemmal resealing and recovery from injury

Andy H. Vo, E. McNally, M. Quattrocelli, 2017, PLoS genetics.

Abcc9 is required for the transition to oxidative metabolism in the newborn heart

Gene Kim, J. Makielski, E. McNally, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Overexpression of Latent TGFβ Binding Protein 4 in Muscle Ameliorates Muscular Dystrophy through Myostatin and TGFβ

Andy H. Vo, J. Molkentin, E. McNally, 2016, PLoS genetics.

Impaired exercise tolerance and skeletal muscle myopathy in sulfonylurea receptor-2 mutant mice.

R. Lang, E. McNally, P. Pytel, 2009, American journal of physiology. Regulatory, integrative and comparative physiology.

Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy.

A. E. Rossi, J. Goldstein, J. Molkentin, 2014, Human molecular genetics.

Mice lacking sulfonylurea receptor 2 (SUR2) ATP-sensitive potassium channels are resistant to acute cardiovascular stress.

J. Makielski, E. McNally, N. Shi, 2007, Journal of molecular and cellular cardiology.

Fog1 is required for cardiac looping in zebrafish.

R. Ho, E. Svensson, A. Bruce, 2006, Developmental biology.

Targeting latent TGFβ release in muscular dystrophy

Lucas R. Smith, J. Molkentin, E. Barton, 2014, Science Translational Medicine.

S100/Calgranulin-Mediated Inflammation Accelerates Left Ventricular Hypertrophy and Aortic Valve Sclerosis in Chronic Kidney Disease in a Receptor for Advanced Glycation End Products–Dependent Manner

Ling Yan, J. Earley, B. Chellan, 2014, Arteriosclerosis, thrombosis, and vascular biology.

Intermittent glucocorticoid steroid dosing enhances muscle repair without eliciting muscle atrophy

Andy H. Vo, E. McNally, M. Quattrocelli, 2017, The Journal of clinical investigation.

Enhanced Muscular Dystrophy from Loss of Dysferlin Is Accompanied by Impaired Annexin A6 Translocation after Sarcolemmal Disruption.

E. McNally, James L. Warner, J. Earley, 2016, The American journal of pathology.

Reengineering a transmembrane protein to treat muscular dystrophy using exon skipping.

C. Bönnemann, J. Goldstein, Matthew J. Wolf, 2015, The Journal of clinical investigation.

Secondary coronary artery vasospasm promotes cardiomyopathy progression.

M. Wheeler, R. Lang, C. Korcarz, 2004, The American journal of pathology.

SMAD signaling drives heart and muscle dysfunction in a Drosophila model of muscular dystrophy.

J. Goldstein, Matthew J. Wolf, E. McNally, 2011, Human molecular genetics.

Nuclear sequestration of delta-sarcoglycan disrupts the nuclear localization of lamin A/C and emerin in cardiomyocytes.

E. McNally, J. Earley, M. Hadhazy, 2007, Human molecular genetics.

lamin A/C and emerin in cardiomyocytes

E. McNally, J. Earley, M. Hadhazy, 2006 .

Reduced life span with heart and muscle dysfunction in Drosophila sarcoglycan mutants.

H. Rockman, G. Bhabha, Matthew J. Wolf, 2007, Human molecular genetics.

Eps 15 Homology Domain (EHD)-1 Remodels Transverse Tubules in Skeletal Muscle

A. E. Rossi, H. Band, E. McNally, 2015, PloS one.

Copper Content and Exchange in Mammalian Hearts

E. Page, L. McCallister, J. Earley, 1974, Circulation research.

Beneficial effects of quinoline-3-carboxamide (ABR-215757) on atherosclerotic plaque morphology in S100A12 transgenic ApoE null mice.

Gene Kim, Ling Yan, J. Gawdzik, 2013, Atherosclerosis.

S100A12 in Vascular Smooth Muscle Accelerates Vascular Calcification in Apolipoprotein E–Null Mice by Activating an Osteogenic Gene Regulatory Program

Gene Kim, P. Toth, A. Husain, 2011, Arteriosclerosis, thrombosis, and vascular biology.

EHD1 mediates vesicle trafficking required for normal muscle growth and transverse tubule development.

H. Band, E. McNally, P. Pytel, 2014, Developmental biology.

Myofibrillar Mass in Rat and Rabbit Heart Muscle: CORRELATION OF MICROCHEMICAL AND STEREOLOGICAL MEASUREMENTS IN NORMAL AND HYPERTROPHIC HEARTS

E. Page, P. Polimeni, R. Zak, 1972, Circulation research.

Downregulation of connexin43 by microRNA-130a in cardiomyocytes results in cardiac arrhythmias.

Gene Kim, M. Broman, T. Calway, 2014, Journal of molecular and cellular cardiology.