N. A. Temiz

发表

Optimization of minimum set of protein–DNA interactions: a quasi exact solution with minimum over-fitting

Oleg A. Prokopyev, Andrew C. Trapp, Carlos J. Camacho, 2009, Bioinform..

APOBEC3B is an enzymatic source of mutation in breast cancer

N. A. Temiz, Jason B. Nikas, R. Harris, 2013, Nature.

Evidence for APOBEC3B mutagenesis in multiple human cancers

N. A. Temiz, R. Harris, M. Burns, 2013, Nature Genetics.

Inhibitor binding alters the directions of domain motions in HIV‐1 reverse transcriptase

Ivet Bahar, N. A. Temiz, I. Bahar, 2002, Proteins.

Novel modulation factor quantifies the role of water molecules in protein interactions

N. A. Temiz, C. Camacho, M. Bueno, 2010, Proteins.

Experimentally based contact energies decode interactions responsible for protein–DNA affinity and the role of molecular waters at the binding interface

Carlos J. Camacho, N. A. Temiz, C. Camacho, 2009, Nucleic acids research.

Decoding the protein-DNA recognition rules

N. A. Temiz, N. Temiz, 2009 .

The Role of Methylation in the Intrinsic Dynamics of B- and Z-DNA

N. A. Temiz, D. Donohue, B. Luke, 2012, PloS one.

Abstract A242: R-spondin 2 drives Wnt signaling and tumor formation in breast and liver cancer.

N. A. Temiz, D. Largaespada, R. Harris, 2013 .

The somatic autosomal mutation matrix in cancer genomes

N. A. Temiz, R. Stephens, M. Yi, 2015, Human Genetics.

R-Spondins 2 and 3 Are Overexpressed in a Subset of Human Colon and Breast Cancers.

N. A. Temiz, D. Largaespada, R. Harris, 2020, DNA and cell biology.

Mutational impact of APOBEC3B and APOBEC3A in a human cell line

N. A. Temiz, M. Carpenter, D. Yee, 2022 .