Mosi Lin

发表

The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia.

D. Thomas, L. Mantell, Ashwini Gore, 2020, Biochemical pharmacology.

Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

L. Mantell, M. Lin, Maleka T. Stewart, 2022, Free Radical Biology and Medicine.

The Positive Allosteric Modulation of alpha7-Nicotinic Cholinergic Receptors by GAT107 Increases Bacterial Lung Clearance in Hyperoxic Mice by Decreasing Oxidative Stress in Macrophages

G. Thakur, Abhijit R. Kulkarni, C. Ashby, 2021, Antioxidants.

Nuclear Damage Associated Molecular Pattern (DAMP) Molecule HMGB1 in the Pathogenesis of Lung Diseases.

C. Ashby, L. Mantell, Mao Wang, 2019, Antioxidants & redox signaling.

The Role of HMGB1, a Nuclear Damage-Associated Molecular Pattern Molecule, in the Pathogenesis of Lung Diseases

L. Mantell, Mao Wang, Joanna Woo, 2019 .

From nicotine to the cholinergic anti-inflammatory reflex – Can nicotine alleviate the dysregulated inflammation in COVID-19?

C. Ashby, T. Kennedy, L. Mantell, 2021, Journal of immunotoxicology.

The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

V. Pavlov, K. Tracey, C. Ashby, 2020, Molecular Medicine.

Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

C. Ashby, M. Espey, D. Thomas, 2020, International journal of molecular sciences.

GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

G. Thakur, Abhijit R. Kulkarni, L. Mantell, 2023, Redox biology.

Regulation of Basal Lateral Membrane Mobility and Permeability to Divalent Cations by Membrane Associated-Protein Kinase C

Dali Luo, Yuan-yuan Zheng, Chao Zhang, 2013, PloS one.

The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

V. Pavlov, K. Tracey, C. Ashby, 2020, Molecular medicine.

GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

C. Ashby, L. Mantell, R. Sitapara, 2020, Molecular Medicine.

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

C. Ashby, L. Mantell, R. Sitapara, 2020, Molecular medicine.

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

C. Ashby, L. Mantell, R. Sitapara, 2021, Molecular medicine.

2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Haichao Wang, T. Talele, C. Ashby, 2021, Molecular medicine.

A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

M. Yimam, Q. Jia, M. Hong, 2023, Molecules.

GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

C. Ashby, L. Mantell, R. Sitapara, 2020, Molecular Medicine.