G. Oltmans

发表

Food intake and body weight: Effects of specific and non-specific lesions in the midbrain path of the ascending noradrenergic neurons of the rat

D. Margules, J. Lorden, G. Oltmans, 1977, Brain Research.

Alterations in the noradrenergic projection to the cerebellum of the dystonic (dt) rat

J. Lorden, M. Beales, T. W. Mckeon, 1986, Brain Research.

The effect of selective lesions of brain noradrenergic systems on the development of barbiturate tolerance in rats

R. Ritzmann, B. Tabakoff, G. Oltmans, 1979, Brain Research.

LH syndrome and brain catecholamine levels after lesions of the nigrostriatal bundle.

J. Harvey, G. A. Oltmans, G. Oltmans, 1972, Physiology & behavior.

ai-Adrenergic Receptor Binding in the Spontaneously Hypertensive Rat

S. Berenbaum, T. R. Hansen, G. Pullen, 2005 .

alpha 1-Adrenergic receptor binding in the spontaneously hypertensive rat.

S. Berenbaum, T. R. Hansen, G. Pullen, 1985, Hypertension.

Central noradrenergic neurons: differential effects on body weight of electrolytic and 6-hydroxydopamine lesions in rats.

D. Margules, J. Lorden, G. Oltmans, 1976, Journal of comparative and physiological psychology.

Alterations in cerebellar glutamic acid decarboxylase (GAD) activity in a genetic model of torsion dystonia (rat)

J. Gordon, J. Lorden, M. Beales, 1984, Experimental Neurology.

Purkinje cell activity in rats following chronic treatment with harmaline

L. Mays, J. Lorden, S. Stratton, 1988, Neuroscience.

Evaluation of the non-specific effects of catecholamine and serotonin neurotoxins by injection into the medial forebrain bundle of the rat

J. Lorden, R. Dawson, G. Oltmans, 1979, Pharmacology Biochemistry and Behavior.

Lesions of the inferior olive increase glutamic acid decar☐ylase activity in the deep cerebellar nuclei of the rat

J. Lorden, M. Beales, G. Oltmans, 1985, Brain Research.

Effects of food restriction and mutation on central catecholamine levels in genetically obese mice

D. Margules, J. Lorden, G. Oltmans, 1976, Pharmacology Biochemistry and Behavior.

Tolerance to the tremorogenic effects of harmaline: Evidence for altered olivo-cerebellar function

J. Lorden, M. Beales, G. Oltmans, 1988, Neuropharmacology.

Increased glutamic acid decarboxylase (GAD) mRNA and GAD activity in cerebellar Purkinje cells following lesion-induced increases in cell firing

M. Mercugliano, J. Litwak, M. Chesselet, 1990, Neuroscience Letters.

Neuropharmacological correlates of the motor syndrome of the genetically dystonic (dt) rat.

L. Mays, J. Lorden, S. Stratton, 1988, Advances in neurology.

Decreased catalepsy response to haloperidol in the genetically dystonic (dt) rat

S. Walkley, J. Lorden, M. Beales, 1984, Brain Research.

Decreased cerebellar 3',5'-cyclic guanosine monophosphate levels and insensitivity to harmaline in the genetically dystonic rat (dt)

J. Lorden, M. Beales, T. W. Mckeon, 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Yohimbine and rauwolscine reduce food intake of genetically obese (obob) and lean mice

M. Callahan, G. A. Oltmans, M. Beales, 1984, Pharmacology Biochemistry and Behavior.

Lateral hypothalamic syndrome in rats: a comparison of the behavioral and neurochemical effects of lesions placed in the lateral hypothalamus and nigrostriatal bundle.

J. Harvey, G. Oltmans, 1976, Journal of comparative and physiological psychology.

Quantitative autoradiography reveals selective changes in cerebellar GABA receptors of the rat mutant dystonic

M. Beales, E. Walz, G. Oltmans, 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Norepinephrine and dopamine levels in hypothalamic nuclei of the genetically obese mouse (ob/ob)

G. A. Oltmans, G. Oltmans, 1983, Brain Research.

Hypothalamic catecholamine systems in genetically obese mice (obob): Decreased sensitivity to reserpine treatment

J. Comaty, G. Oltmans, R. Olsauskas, 1980, Neuropharmacology.

Amphetamine Anorexia and Hypothalamic catecholamines in genetically obese mice (obob)

G. A. Oltmans, Roma Kuprys, G. Oltmans, 1982, Pharmacology Biochemistry and Behavior.

Increases in α-adrenergic receptors in the hypothalamus of the genetically obese mouse (ob/ob)

J. Fields, M. Callahan, J. Lorden, 1981, Brain Research.

Hypothalamic and pituitary catecholamine levels in genetically obese mice (obob)

J. Lorden, G. Oltmans, 1977, Brain Research.

Central catecholamine turnover in genetically obese mice (obob)

D. Margules, J. Lorden, G. Oltmans, 1976, Brain Research.

Central catecholamine levels in genetically obese mice (obob and dbdb)

D. Margules, J. Lorden, G. A. Oltmans, 1975, Brain Research.

Glutamic acid decar☐ylase activity in micropunches of the deep cerebellar nuclei of the genetically dystonic (dt) rat

J. Lorden, M. Beales, G. Oltmans, 1986, Brain Research.

Acute and chronic effects of climbing fiber lesions on cerebellar cyclic guanosine monophosphate

J. Lorden, M. Beales, G. Oltmans, 1987, Brain Research.

Temporal sequence of motor disturbances and increased cerebellar glutamic acid decar☐ylase activity following 3-acetylpyridine lesions in adult rats

J. Lorden, M. Beales, S. Stratton, 1987, Brain Research.

Adrenergic agents inhibit rapid increases in cerebellar Purkinje cell glutamic acid decarboxylase (GAD67) mRNA levels after climbing fiber lesions or reserpine treatment

G. Oltmans, JF Lorden, GA Oltmans, 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Alteration of the characteristics of learned taste aversion by manipulation of serotonin levels in the rat

J. Lorden, G. Oltmans, 1978, Pharmacology Biochemistry and Behavior.

Spontaneous and harmaline-stimulated Purkinje cell activity in rats with a genetic movement disorder

L. Mays, J. Lorden, G. A. Oltmans, 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Rapid increases in cerebellar Purkinje cell glutamic acid decar☐ylase (GAD67) mRNA after lesion-induced increases in cell firing

S. Drengler, G. Oltmans, 1993, Brain Research.