R. Mor‐cohen

发表

Utilization of machine-learning models to accurately predict the risk for critical COVID-19

Yair Neuman, Dany H. Assaf, G. Rahav, 2020, Internal and Emergency Medicine.

Disulfide Bonds as Regulators of Integrin Function in Thrombosis and Hemostasis.

R. Mor-cohen, R. Mor‐cohen, 2014, Antioxidants & redox signaling.

Identification and Functional Analysis of Two Novel Mutations in the Multidrug Resistance Protein 2 Gene in Israeli Patients with Dubin-Johnson Syndrome*

S. Muallem, N. Rosenberg, U. Seligsohn, 2001, The Journal of Biological Chemistry.

Mechano-redox control of integrin de-adhesion

F. Gräter, P. Hogg, C. Aponte-Santamaría, 2018, eLife.

Age estimates of ancestral mutations causing factor VII deficiency and Dubin–Johnson syndrome in Iranian and Moroccan Jews are consistent with ancient Jewish migrations

N. Rosenberg, U. Seligsohn, A. Zivelin, 2007, Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis.

A mutation in the β3 cytoplasmic tail causes variant Glanzmann thrombasthenia by abrogating transition of αIIbβ3 to an active state

N. Rosenberg, U. Seligsohn, H. Hauschner, 2012, Journal of thrombosis and haemostasis : JTH.

A novel ancestral splicing mutation in the multidrug resistance protein 2 gene causes Dubin-Johnson syndrome in Ashkenazi Jewish patients.

I. Goldberg, N. Rosenberg, U. Seligsohn, 2005, Hepatology research : the official journal of the Japan Society of Hepatology.

Correlation between the incidence of myotonic dystrophy in different groups in Israel and the number of CTG trinucleotide repeats in the myotonin gene.

N. Gadoth, T. Shohat, M. Shohat, 1997, American journal of medical genetics.

An allosteric disulfide bond is involved in enhanced activation of factor XI by protein disulfide isomerase

A. Yeheskel, U. Seligsohn, R. Mor-cohen, 2016, Journal of thrombosis and haemostasis : JTH.

ONCOGENE PANEL SEQUENCING ANALYSIS IDENTIFIES CANDIDATE ACTIONABLE GENES IN ADVANCED WELL-DIFFERENTIATED GASTROENTEROPANCREATIC NEUROENDOCRINE TUMORS.

J. Walling, Y. Zhu, E. Kebebew, 2019, Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists.

Prothrombin 20210G>A is an ancestral prothrombotic mutation that occurred in whites approximately 24,000 years ago.

R. Bertina, P. Kyrle, J. Emmerich, 2006, Blood.

Disulfide bond disruption by a beta 3-Cys549Arg mutation in six Jordanian families with Glanzmann thrombasthenia causes diminished production of constitutively active alpha IIb beta 3.

M. Landau, B. Coller, N. Rosenberg, 2007, Thrombosis and haemostasis.

Molecular diversity of Glanzmann thrombasthenia in southern India: new insights into mRNA splicing and structure–function correlations of αIIbβ3 integrin (ITGA2B, ITGB3)

M. Landau, B. Coller, A. Srivastava, 2006, Human mutation.

Integrin-mediated cell adhesion requires extracellular disulfide exchange regulated by protein disulfide isomerase.

J. Lahav, N. Rosenberg, R. Mor-cohen, 2019, Experimental cell research.

Specific Cysteines in β3 Are Involved in Disulfide Bond Exchange-dependent and -independent Activation of αIIbβ3*

M. Landau, J. Lahav, N. Rosenberg, 2008, Journal of Biological Chemistry.

Unique Disulfide Bonds in Epidermal Growth Factor (EGF) Domains of β3 Affect Structure and Function of αIIbβ3 and αvβ3 Integrins in Different Manner

M. Landau, W. Mansour, Y. Einav, 2012, The Journal of Biological Chemistry.

E109K Is a SEC23B Founder Mutation among Israeli Moroccan Jewish Patients with Congenital Dyserythropoietic Anemia Type II

A. Berrebi, H. Tamary, R. Mor-cohen, 2011, Acta Haematologica.

The role of protein disulfide isomerase in the post-ligation phase of β3 integrin-dependent cell adhesion.

J. Lahav, R. Ram, N. Rosenberg, 2015, Thrombosis research.

The Role Of Protein Disulfide Isomerase In The Post-Ligation Phase Of β3 Integrin-Dependent Cell Adhesion

J. Lahav, R. Ram, N. Rosenberg, 2013 .

Disulfide bond exchanges in integrins αIIbβ3 and αvβ3 are required for activation and post-ligation signaling during clot retraction.

J. Lahav, N. Rosenberg, U. Seligsohn, 2014, Thrombosis research.

Abnormal cytoplasmic extensions associated with active &agr;IIb&bgr;3 are probably the cause for macrothrombocytopenia in Glanzmann thrombasthenia-like syndrome

A. Savoia, W. Mansour, N. Rosenberg, 2015, Blood Coagulation and Fibrinolysis.

Disulfide bond disruption by a β3-Cys549Arg mutation in six Jordanian families with Glanzmann thrombasthenia causes diminished production of constitutively active αIIbβ3

M. Landau, B. Coller, N. Rosenberg, 2007, Thrombosis and Haemostasis.

A 13‐bp deletion in αIIb gene is a founder mutation that predominates in Palestinian‐Arab patients with Glanzmann thrombasthenia

M. Landau, B. Coller, G. Kenet, 2005, Journal of thrombosis and haemostasis : JTH.

Abnormal cytoplasmic extensions associated with active &agr;IIb&bgr;3 are probably the cause for macrothrombocytopenia in Glanzmann thrombasthenia-like syndrome

A. Savoia, W. Mansour, N. Rosenberg, 2015, Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis.

The reduced form of coagulation factor XI is associated with illness severity and coagulopathy in critically-ill septic patients

Samuel M. Brown, Robert A. Campbell, M. Rondina, 2019, Journal of Thrombosis and Thrombolysis.