C. McKinney

发表

A novel strategy to interfere with human immunodeficiency virus type 1 propagation.

W. Tate, J. A. Horsfield, J. H. Irvine, 1998, The New Zealand medical journal.

Enzymatically inactive U(S)3 protein kinase of Marek's disease virus (MDV) is capable of depolymerizing F-actin but results in accumulation of virions in perinuclear invaginations and reduced virus growth.

N. Osterrieder, B. Kaufer, D. Schumacher, 2008, Virology.

Co-opting the Fanconi anemia genomic stability pathway enables herpesvirus DNA synthesis and productive growth.

J. Yates, I. Mohr, J. Savas, 2014, Molecular cell.

Translational Control of the Abundance of Cytoplasmic Poly(A) Binding Protein in Human Cytomegalovirus-Infected Cells

I. Mohr, C. Pérez, C. McKinney, 2010, Journal of Virology.

The Functionality of Marek's Disease Virus Us3 Protein

C. McKinney, 2007 .

Global reprogramming of the cellular translational landscape facilitates cytomegalovirus replication.

J. Zavadil, I. Mohr, Stuart Brown, 2014, Cell reports.

A new role for the cellular PABP repressor Paip2 as an innate restriction factor capable of limiting productive cytomegalovirus replication

I. Mohr, Dong Yu, C. McKinney, 2013, Genes & development.

Poly(A) binding protein abundance regulates eukaryotic translation initiation factor 4F assembly in human cytomegalovirus-infected cells

I. Mohr, C. Pérez, C. McKinney, 2012, Proceedings of the National Academy of Sciences.

Co-opting the Fanconi anemia genomic stability pathway enables herpesvirus DNA synthesis and productive growth.

J. Yates, I. Mohr, J. Savas, 2014, Molecular cell.

Global Reprogramming of the Cellular Translational Landscape Facilitates Cytomegalovirus Replication.

J. Zavadil, I. Mohr, Stuart Brown, 2014, Cell reports.