K. C. Fernando

Pinocytosis in 2,5-di-tert-butylhydroquinone-stimulated hepatocytes and evaluation of its role in Ca2+ inflow

G. Barritt, K. C. Fernando, 1996, Molecular and Cellular Biochemistry.

Evidence that a low-molecular-mass GTP-binding protein is required for store-activated Ca2+ inflow in hepatocytes.

B. Kemp, G. Barritt, R. Gregory, 1997, The Biochemical journal.

Characterisation of the divalent cation channels of the hepatocyte plasma membrane receptor-activated Ca2+ inflow system using lanthanide ions.

G. Barritt, K. C. Fernando, 1995, Biochimica et biophysica acta.

Activation of the P2Z/P2X7 receptor in human lymphocytes produces a delayed permeability lesion: involvement of phospholipase D.

C. Gargett, J. Wiley, K. C. Fernando, 1999, Archives of biochemistry and biophysics.

Characterisation of the inhibition of the hepatocyte receptor-activated Ca2+ inflow system by gadolinium and SK&F 96365.

G. Barritt, K. C. Fernando, Greg J. Barritt, 1994, Biochimica et biophysica acta.

P2Y(11) receptor expression by human lymphocytes: evidence for two cAMP-linked purinoceptors.

A. Conigrave, J. Wiley, B. Gu, 2001, European journal of pharmacology.

Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondria.

G. Barritt, R. Gregory, K. C. Fernando, 1998, The Biochemical journal.