Diego L. Costa

发表

Protective and Pathological Functions of CD8+ T Cells in Leishmania braziliensis Infection

Adriano Queiroz, P. Scott, L. Carvalho, 2014, Infection and Immunity.

A major role for ferroptosis in Mycobacterium tuberculosis–induced cell death and tissue necrosis

A. Sher, O. Kamenyeva, B. Andrade, 2019, The Journal of experimental medicine.

Mycobacterium tuberculosis Induces Irg1 in Murine Macrophages by a Pathway Involving Both TLR-2 and STING/IFNAR Signaling and Requiring Bacterial Phagocytosis

J. Weiss, A. Sher, R. Chari, 2022, Frontiers in Cellular and Infection Microbiology.

Characterization of regulatory T cell (Treg) function in patients infected with Leishmania braziliensis.

Adriano Queiroz, E. Carvalho, J. Silva, 2013, Human immunology.

Tr-1-like CD4+CD25-CD127-/lowFOXP3- cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis.

Adriano Queiroz, E. Carvalho, J. Silva, 2015, The Journal of infectious diseases.

GPX4 regulates cellular necrosis and host resistance in Mycobacterium tuberculosis infection

A. Sher, D. Barber, R. Wilkinson, 2022, The Journal of experimental medicine.

The lectin-specific activity of Toxoplasma gondii microneme proteins 1 and 4 binds Toll-like receptor 2 and 4 N-glycans to regulate innate immune priming

A. Sher, N. Gay, M. Roque-Barreira, 2019, PLoS pathogens.

Toxoplasma gondii microneme proteins 1 and 4 bind to Toll-like receptors 2 and 4 N-glycans triggering innate immune response

A. Sher, M. Roque-Barreira, D. Soldati-Favre, 2017, bioRxiv.

Longitudinal profiling reveals a persistent intestinal dysbiosis triggered by conventional anti-tuberculosis therapy

A. Sher, M. Glickman, A. Dzutsev, 2017, Microbiome.

Recent Advances in Host-Directed Therapies for Tuberculosis and Malaria

Diego L Costa, K. C. Matteucci, A. S. Correa, 2022, Frontiers in Cellular and Infection Microbiology.

Lutzomyia longipalpis Saliva Induces Heme Oxygenase-1 Expression at Bite Sites

U. Lopes, S. Kamhawi, N. Aronson, 2018, Front. Immunol..

BALB/c Mice Infected with Antimony Treatment Refractory Isolate of Leishmania braziliensis Present Severe Lesions due to IL-4 Production

A. Giudice, E. Carvalho, J. Silva, 2011, PLoS neglected tropical diseases.

Modulation of Inflammation and Immune Responses by Heme Oxygenase-1: Implications for Infection with Intracellular Pathogens

A. Sher, B. Andrade, Diego L Costa, 2020, Antioxidants.

N-acetyl-cysteine exhibits potent anti-mycobacterial activity in addition to its known anti-oxidative functions

A. Sher, B. Andrade, M. Cordeiro-Santos, 2016, BMC Microbiology.

Inflammasome-derived IL-1β production induces nitric oxide–mediated resistance to Leishmania

R. Flavell, M. Bozza, J. Silva, 2013, Nature Medicine.

Heme oxygenase-1 inhibition promotes IFNγ- and NOS2-mediated control of Mycobacterium tuberculosis infection

A. Sher, Sivaranjani Namasivayam, S. Hieny, 2020, Mucosal Immunology.

Heme oxygenase-1 inhibition promotes IFNγ and NOS2 mediated control of Mycobacterium tuberculosis infection

A. Sher, Robert W. Thompson, B. Andrade, 2020, Mucosal immunology.

Systemic toxoplasma infection triggers a long-term defect in the generation and function of naive T lymphocytes

A. Sher, O. Kamenyeva, J. Ashwell, 2016, The Journal of experimental medicine.

Crosstalk between Heme Oxygenase-1 and Iron Metabolism in Macrophages: Implications for the Modulation of Inflammation and Immunity

Joseana de Oliveira, Marina Denadai, Diego L Costa, 2022, Antioxidants.

Longitudinal profiling reveals a persistent intestinal dysbiosis triggered by conventional anti-tuberculosis therapy

A. Sher, M. Glickman, A. Dzutsev, 2017, Microbiome.

Enhancement of CD4+ T Cell Function as a Strategy for Improving Antibiotic Therapy Efficacy in Tuberculosis: Does It Work?

A. Sher, B. Andrade, Sivaranjani Namasivayam, 2021, Frontiers in Cellular and Infection Microbiology.

Reappraisal of the immunopathogenesis of disseminated leishmaniasis: in situ and systemic immune response.

M. Teixeira, P. Machado, E. Carvalho, 2011, Transactions of the Royal Society of Tropical Medicine and Hygiene.

Mycobacterium tuberculosis Induction of Heme Oxygenase-1 Expression Is Dependent on Oxidative Stress and Reflects Treatment Outcomes

A. Sher, A. Kubler, W. Bishai, 2017, Front. Immunol..

The transcription factor BACH1 promotes tissue damage and host susceptibility in Mycobacterium tuberculosis infection by reducing expression of Gpx4, a major negative regulator of ferroptosis

A. Sher, Sivaranjani Namasivayam, C. C. B. Bomfim, 2020, The Journal of Immunology.

The heme oxygenase-1 metalloporphyrin inhibitor stannsoporfin enhances the bactericidal activity of a novel regimen for multidrug-resistant tuberculosis in a murine model

A. Sher, N. Fotouhi, S. Karanika, 2023, bioRxiv.

Pharmacological Inhibition of Host Heme Oxygenase-1 Suppresses Mycobacterium tuberculosis Infection In Vivo by a Mechanism Dependent on T Lymphocytes

A. Sher, C. Goulding, A. Chao, 2016, mBio.

Editorial: Host-Directed Therapies for Tuberculosis

M. Maiga, S. Subbian, Diego L. Costa, 2021, Frontiers in Cellular and Infection Microbiology.

The heme oxygenase-1 metalloporphyrin inhibitor stannsoporfin enhances the bactericidal activity of a novel regimen for multidrug-resistant tuberculosis in a murine model

A. Sher, N. Fotouhi, S. Karanika, 2023, bioRxiv.

BACH1 promotes tissue necrosis and Mycobacterium tuberculosis susceptibility

T. Scriba, K. Hilligan, Sivaranjani Namasivayam, 2023, Nature microbiology.