G. Gleich

Substance P-induced airway hyperreactivity is mediated by neuronal M(2) receptor dysfunction.

A. Fryer, C. M. Evans, K. Belmonte, 2000, American journal of physiology. Lung cellular and molecular physiology.

Eosinophil-mediated cholinergic nerve remodeling.

I. Hall, M. Walsh, J. Blusztajn, 2006, American journal of respiratory cell and molecular biology.

Eosinophil major basic protein activates human cord blood mast cells primed with fibroblast membranes by integrin‐β1

I. Bachelet, F. Levi-Schaffer, M. Ben-Zimra, 2013, Allergy.

Pretreatment with antibody to eosinophil major basic protein prevents hyperresponsiveness by protecting neuronal M2 muscarinic receptors in antigen-challenged guinea pigs.

A. Fryer, C. M. Evans, D. Jacoby, 1997, The Journal of clinical investigation.

Eosinophils and airway nerves in asthma.

A. Fryer, D. Jacoby, R. Costello, 2000, Histology and histopathology.

Localization of eosinophils to airway nerves and effect on neuronal M2 muscarinic receptor function.

G. Gleich, A. Fryer, D. Jacoby, 1997, The American journal of physiology.

Repeat treatment of acute hereditary angioedema attacks with open‐label icatibant in the FAST‐1 trial

W. Lumry, B. Ritchie, J. Hébert, 2014, Clinical and experimental immunology.

Eosinophil peroxidase induces the expression and function of acid‐sensing ion channel‐3 in allergic rhinitis: in vitro evidence in cultured epithelial cells

M. Walsh, B. Harvey, S. McQuaid, 2012, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

Human eosinophil major basic protein is an endogenous allosteric antagonist at the inhibitory muscarinic M2 receptor.

G. Gleich, A. Fryer, D. Jacoby, 1993, The Journal of clinical investigation.

Ozone-induced hyperresponsiveness and blockade of M2 muscarinic receptors by eosinophil major basic protein.

A. Fryer, B. L. Yost, G. J. Gleich, 1999, Journal of applied physiology.