D. Wilson

V. Bohr, B. Berquist, M. Scheibye-Knudsen, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Direct and indirect roles of RECQL4 in modulating base excision repair capacity.

David M. Wilson, K. Becker, M. Hedayati, 2009, Human molecular genetics.

Effect of protein binding on ultrafast DNA dynamics: characterization of a DNA:APE1 complex.

R. S. Coleman, L. Gearheart, Sobhan Sen, 2005, Biophysical journal.

Repair of persistent strand breaks in the mitochondrial genome

David M. Wilson, V. Bohr, P. Sykora, 2012, Mechanisms of Ageing and Development.

Aprataxin localizes to mitochondria and preserves mitochondrial function

David M. Wilson, V. Bohr, D. Croteau, 2011, Proceedings of the National Academy of Sciences.

Synthetic lethal targeting of DNA double‐strand break repair deficient cells by human apurinic/apyrimidinic endonuclease inhibitors

C. Laughton, P. Fischer, C. Seedhouse, 2012, International journal of cancer.

Variation in base excision repair capacity.

A. Sigurdson, B. Berquist, Daemyung Kim, 2011, Mutation research.

Removal of Oxidative DNA Damage via FEN1-Dependent Long-Patch Base Excision Repair in Human Cell Mitochondria

David M. Wilson, V. Bohr, D. Bogenhagen, 2008, Molecular and Cellular Biology.

Functional Assessment of Population and Tumor-Associated APE1 Protein Variants

J. Illuzzi, A. Drohat, Mengxia Li, 2013, PloS one.

Nucleotide Sequence and DNA Secondary Structure, as Well as Replication Protein A, Modulate the Single-stranded Abasic Endonuclease Activity of APE1*

David M. Wilson, Jinshui Fan, Y. Matsumoto, 2006, Journal of Biological Chemistry.

Identification and Quantification of DNA Repair Protein Apurinic/Apyrimidinic Endonuclease 1 (APE1) in Human Cells by Liquid Chromatography/Isotope-Dilution Tandem Mass Spectrometry

David M. Wilson, A. Tona, M. Dizdaroglu, 2013, PloS one.

Reduced Nuclease Activity of Apurinic/Apyrimidinic Endonuclease (APE1) Variants on Nucleosomes

Peng Mao, D. R. McNeill, J. M. Hinz, 2015, The Journal of Biological Chemistry.

Microsoft Word-APE1 Variants Nucleosomes MS.vJBC3.docx

David M. Wilson, Peng Mao, D. R. McNeill, 2015 .

Identification and Characterization of Inhibitors of Human Apurinic/apyrimidinic Endonuclease APE1

David M. Wilson, A. Jadhav, C. Austin, 2009, PloS one.

Lead promotes abasic site accumulation and co‐mutagenesis in mammalian cells by inhibiting the major abasic endonuclease Ape1

H. Wong, D. R. McNeill, D. Wilson, 2007, Molecular carcinogenesis.

Small Molecule Inhibitors of the Human Apurinic/apyrimidinic Endonuclease 1 (APE1)

David M. Wilson, A. Jadhav, A. Simeonov, 2013 .

New insights into the structure of abasic DNA from molecular dynamics simulations

Alexander D. MacKerell, Nicolas Foloppe, Daniel Barsky, 2000, Nucleic Acids Res..

Lucanthone and Its Derivative Hycanthone Inhibit Apurinic Endonuclease-1 (APE1) by Direct Protein Binding

Samuel H. Wilson, David M. Wilson, M. Mezei, 2011, PloS one.

Investigation of the role of the histidine-aspartate pair in the human exonuclease III-like abasic endonuclease, Ape1.

D. Hoyt, D. F. Lowry, J. Bagu, 2003, Journal of molecular biology.

Non-muscle invasive bladder cancer tissues have increased base excision repair capacity

A. Kural, Y. Sağlıcan, C. Obek, 2020, Scientific Reports.

Inhibitors of the apurinic/apyrimidinic endonuclease 1 (APE1)/nucleophosmin (NPM1) interaction that display anti‐tumor properties

David M. Wilson, A. Jadhav, A. Simeonov, 2016, Molecular carcinogenesis.

XRCC1 down‐regulation in human cells leads to DNA‐damaging agent hypersensitivity, elevated sister chromatid exchange, and reduced survival of BRCA2 mutant cells

Jinshui Fan, H. Wong, P. F. Wilson, 2007, Environmental and molecular mutagenesis.

Serum APE1 as a predictive marker for platinum-based chemotherapy of non-small cell lung cancer patients

G. Tell, Chuan Chen, Dong Wang, 2016, Oncotarget.

Integrated computational and experimental identification of p53, KRAS and VHL mutant selection associated with CRISPR-Cas9 editing

Mark D. M. Leiserson, E. Ruppin, J. Lee, 2018, bioRxiv.

The excitatory neurotransmitter glutamate stimulates DNA repair to increase neuronal resiliency

M. Mattson, V. Bohr, Jenq-Lin Yang, 2011, Mechanisms of Ageing and Development.

Serum APE 1 as a predictive marker for platinum-based chemotherapy of non-small cell lung cancer patients

G. Tell, Chuan Chen, Dong Wang, 2016 .

Intrusion of a DNA Repair Protein in the RNome World: Is This the Beginning of a New Era?

David M. Wilson, G. Tell, Chow H Lee, 2009, Molecular and Cellular Biology.

DNA damage repair response in mesenchymal stromal cells: From cellular senescence and aging to apoptosis and differentiation ability

David M. Wilson, N. Samadi, M. Hejazi, 2020, Ageing Research Reviews.

MINIREVIEW Intrusion of a DNA Repair Protein in the RNome World: Is This the Beginning of a New Era? (cid:1)

G. Tell, Chow H Lee, D. Wilson, 2009 .

Tumor‐associated APE1 variant exhibits reduced complementation efficiency but does not promote cancer cell phenotypes

K. Becker, F. Bunz, R. Wersto, 2017, Environmental and molecular mutagenesis.

A High Fat Diet and NAD+ Rescue Premature Aging in Cockayne Syndrome

David M. Wilson, Bohr, M. Mattson, 2014 .

The major human abasic endonuclease: formation, consequences and repair of abasic lesions in DNA.

D. Wilson, D. Barsky, D. Wilson, 2001, Mutation research.

Mapping the protein-DNA interface and the metal-binding site of the major human apurinic/apyrimidinic endonuclease.

J. P. Erzberger, D. Wilson, D. Barsky, 2000, Journal of molecular biology.

XRCC1 haploinsufficiency in mice has little effect on aging, but adversely modifies exposure-dependent susceptibility

P. Pistell, K. Fishbein, R. Spencer, 2011, Nucleic acids research.

Two divalent metal ions in the active site of a new crystal form of human apurinic/apyrimidinic endonuclease, Ape1: implications for the catalytic mechanism.

B. Rupp, M. Hadi, B. Segelke, 2001, Journal of molecular biology.

Functional characterization of Ape1 variants identified in the human population.

K. Fidelis, H. Mohrenweiser, M. Hadi, 2000, Nucleic acids research.

The role of Mg2+ and specific amino acid residues in the catalytic reaction of the major human abasic endonuclease: new insights from EDTA-resistant incision of acyclic abasic site analogs and site-directed mutagenesis.

D. Wilson, J. Erzberger, D. Wilson, 1999, Journal of molecular biology.

Androgen receptor-binding sites are highly mutated in prostate cancer

M. Gönen, Tunc Morova, N. Lack, 2020, Nature Communications.

Participation of DNA repair in the response to 5-fluorouracil

M. Wyatt, D. Wilson, D. Wilson, 2009, Cellular and Molecular Life Sciences.

Kinetic and Mechanistic Studies of the Terminal Uridylyltransferase, Zcchc11 (TUT4).

David M. Wilson, R. Stein, D. Wilson, 2022, Biochemistry.

Regulation of the Intranuclear Distribution of the Cockayne Syndrome Proteins

David M. Wilson, V. Bohr, Mustafa N Okur, 2018, Scientific Reports.

Werner syndrome protein 1367 variants and disposition towards coronary artery disease in Caucasian patients

E. Metter, M. Gray, V. Bohr, 2004, Mechanisms of Ageing and Development.

New areas of focus at workshop on human diseases involving DNA repair deficiency and premature aging

D. Wilson, 2007 .

Development of a Cell-Based Assay for Measuring Base Excision Repair Responses

David M. Wilson, D. R. McNeill, R. Sobol, 2017, Scientific Reports.

DNA Polymerase Beta Participates in Mitochondrial DNA Repair

A. Yasui, V. Bohr, B. A. Baptiste, 2017, Molecular and Cellular Biology.

Neil1 is a genetic modifier of somatic and germline CAG trinucleotide repeat instability in R6/1 mice

David M. Wilson, M. Bjørås, A. Klungland, 2012, Human molecular genetics.

3'-phosphodiesterase activity of human apurinic/apyrimidinic endonuclease at DNA double-strand break ends.

L. Povirk, D. Suh, D. Wilson, 1997, Nucleic acids research.

Identification of factors interacting with hMSH2 in the fetal liver utilizing the yeast two-hybrid system. In vivo interaction through the C-terminal domains of hEXO1 and hMSH2 and comparative expression analysis.

L. Rasmussen, F. Nielsen, M. Rasmussen, 2000, Mutation research.

DNA repair mechanisms in dividing and non-dividing cells.

T. Iyama, D. Wilson, 2013, DNA repair.

The nucleotide sequence, DNA damage location, and protein stoichiometry influence the base excision repair outcome at CAG/CTG repeats.

A. Tomkinson, C. E. Pearson, K. Merienne, 2012, Biochemistry.

Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane

V. Bohr, B. Berquist, M. Scheibye-Knudsen, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

DNA damage, DNA repair, ageing and age-related disease

V. Bohr, P. Mckinnon, D. Wilson, 2008, Mechanisms of Ageing and Development.

Intrusion of a DNA Repair Protein in the RNome World: Is This the Beginning of a New Era?

G. Tell, Chow H Lee, D. Wilson, 2009, Molecular and Cellular Biology.

Aprataxin localizes to mitochondria and preserves mitochondrial function

V. Bohr, D. Croteau, P. Sykora, 2011, Proceedings of the National Academy of Sciences.

A high-fat diet and NAD(+) activate Sirt1 to rescue premature aging in cockayne syndrome.

R. de Cabo, M. Mattson, V. Bohr, 2014, Cell metabolism.

The excitatory neurotransmitter glutamate stimulates DNA repair to increase neuronal resiliency

M. Mattson, V. Bohr, Jenq-Lin Yang, 2011, Mechanisms of Ageing and Development.

Genomic and protein expression analysis reveals flap endonuclease 1 (FEN1) as a key biomarker in breast and ovarian cancer

G. Ball, I. Ellis, C. Caldas, 2014, Molecular oncology.

Inhibitors of the apurinic/apyrimidinic endonuclease 1 (APE1)/nucleophosmin (NPM1) interaction that display anti‐tumor properties

A. Jadhav, A. Simeonov, C. Vascotto, 2016, Molecular carcinogenesis.

Impairment of APE1 Function Enhances Cellular Sensitivity to Clinically Relevant Alkylators and Antimetabolites

T. DeWeese, D. R. McNeill, W. Lam, 2009, Molecular Cancer Research.