Lu‐Hai Wang

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Receptor for RACK1 Mediates Activation of JNK by Protein Kinase C

Z. Ronai, Lu-Hai Wang, A. Bhoumik, 2005 .

RACK1, an Insulin-Like Growth Factor I (IGF-I) Receptor-Interacting Protein, Modulates IGF-I-Dependent Integrin Signaling and Promotes Cell Spreading and Contact with Extracellular Matrix

Weiqun Li, U. Hermanto, C. S. Zong, 2002, Molecular and Cellular Biology.

Protein Kinase C-δ Is an Important Signaling Molecule in Insulin-Like Growth Factor I Receptor-Mediated Cell Transformation

L. Flechner, Weiqun Li, Lu-Hai Wang, 1998, Molecular and Cellular Biology.

Dual mechanism of signal transducer and activator of transcription 5 activation by the insulin receptor.

R. Kohanski, H. Sadowski, Maithao N Le, 2002, Molecular endocrinology.

Insulin Induction of SOCS-2 and SOCS-3 mRNA Expression in C2C12 Skeletal Muscle Cells Is Mediated by Stat5*

Maithao Le, H. Sadowski, T. Wheeler, 2001, The Journal of Biological Chemistry.

Effect of Tyrosine Mutations on the Kinase Activity and Transforming Potential of an Oncogenic Human Insulin-like Growth Factor I Receptor (*)

C. S. Zong, J. Chan, Yixing Jiang, 1996, The Journal of Biological Chemistry.

Advances of AKT pathway in human oncogenesis and as a target for anti-cancer drug discovery.

W. Kong, Lili He, J. Cheng, 2008, Current cancer drug targets.

Differential Requirement for Rho Family GTPases in an Oncogenic Insulin-like Growth Factor-I Receptor-induced Cell Transformation*

Weiqun Li, Lu-Hai Wang, T. Miki, 2001, The Journal of Biological Chemistry.

Vav3 Mediates Receptor Protein Tyrosine Kinase Signaling, Regulates GTPase Activity, Modulates Cell Morphology, and Induces Cell Transformation

John D. Welsh, T. Trenkle, J. Chan, 2000, Molecular and Cellular Biology.

Vav3 oncogene is overexpressed and regulates cell growth and androgen receptor activity in human prostate cancer.

M. Revelo, Z. Dong, Sha Lu, 2006, Molecular endocrinology.

Mutations of Ros Differentially Effecting Signal Transduction Pathways Leading to Cell Growth Versus Transformation*

C. S. Zong, J. Chan, Lu‐Hai Wang, 1997, The Journal of Biological Chemistry.

Rapamycin by itself and additively in combination with carboplatin inhibits the growth of ovarian cancer cells.

Lu-Hai Wang, Wei Li, J. Chan, 2009, Gynecologic oncology.

Twist Is Transcriptionally Induced by Activation of STAT3 and Mediates STAT3 Oncogenic Function*

D. Coppola, J. Cheng, Mei Sun, 2008, Journal of Biological Chemistry.

Trefoil factor family-1 mutations enhance gastric cancer cell invasion through distinct signaling pathways.

H. Weinstein, S. Itzkowitz, X. Yio, 2006, Gastroenterology.

RACK1 Recruits STAT3 Specifically to Insulin and Insulin-Like Growth Factor 1 Receptors for Activation, Which Is Important for Regulating Anchorage-Independent Growth

Z. Ronai, U. Hermanto, C. S. Zong, 2006, Molecular and Cellular Biology.

Stat3 Plays an Important Role in Oncogenic Ros- and Insulin-like Growth Factor I Receptor-induced Anchorage-independent Growth*

C. S. Zong, Lu-Hai Wang, H. Sadowski, 1998, The Journal of Biological Chemistry.

Molecular signaling regulating anchorage-independent growth of cancer cells.

Lu‐Hai Wang, 2004, The Mount Sinai journal of medicine, New York.

Twist transcriptionally up-regulates AKT2 in breast cancer cells leading to increased migration, invasion, and resistance to paclitaxel.

G. Z. Cheng, Lu-Hai Wang, George Z Cheng, 2007, Cancer research.

The Role of Phosphatidylinositol 3-Kinase, Rho Family GTPases, and STAT3 in Ros-induced Cell Transformation*

Maithao Le, C. S. Zong, K. Nguyen, 2002, The Journal of Biological Chemistry.

Mechanism of STAT3 Activation by Insulin-like Growth Factor I Receptor*

D. Levy, C. Horvath, L. Wang, 2000, The Journal of Biological Chemistry.

Differential requirements of the MAP kinase and PI3 kinase signaling pathways in Src- versus insulin and IGF-1 receptors-induced growth and transformation of rat intestinal epithelial cells

K. Nguyen, J. Chan, Lu‐Hai Wang, 2000, Oncogene.

Effect of Dimerization on Signal Transduction and Biological Function of Oncogenic Ros, Insulin, and Insulin-like Growth Factor I Receptors*

Lu-Hai Wang, Michael Lai, J. Chan, 1997, The Journal of Biological Chemistry.

Insulin-Like Growth Factor I Synergizes with Interleukin 4 for Hematopoietic Cell Proliferation Independent of Insulin Receptor Substrate Expression

L. Flechner, J. Gutkind, Weiqun Li, 1999, Molecular and Cellular Biology.

Rapamycin together with herceptin significantly increased anti‐tumor efficacy compared to either alone in ErbB2 over expressing breast cancer cells

Wei Li, J. Chan, Lu‐Hai Wang, 2007, International journal of cancer.

Resistance to Paclitaxel Cells Leading to Increased Migration , Invasion , and Twist Transcriptionally Upregulates AKT 2 in Breast Cancer

G. Z. Cheng, Lu-Hai Wang, Qi Wang, 2007 .

Transcriptional profile of Rous Sarcoma Virus transformed chicken embryo fibroblasts reveals new signaling targets of viral src

M. Sudol, D. Carey, Weizhou Zhang, 2007, Virology.

Interferon-β Signaling Contributes to Ras Transformation

S. Pestka, L. Runnels, Leroy F. Liu, 2011, PloS one.

Matrix-independent Activation of Phosphatidylinositol 3-Kinase, Stat3, and Cyclin A-associated Cdk2 Is Essential for Anchorage-independent Growth of v-Ros-transformed Chicken Embryo Fibroblasts*

C. S. Zong, Lu-Hai Wang, S. Uttamsingh, 2003, Journal of Biological Chemistry.

- Ros: c-Ros protein-tyrosine kinase (vertebrates)

Lu‐Hai Wang, 1995 .

Regulation of cancer cell survival, migration, and invasion by Twist: AKT2 comes to interplay.

G. Z. Cheng, Lu-Hai Wang, Weizhou Zhang, 2008, Cancer research.

RACK1 and CIS Mediate the Degradation of BimEL in Cancer Cells*

J. Cheng, G. Z. Cheng, U. Hermanto, 2008, Journal of Biological Chemistry.

RACK1 mediates activation of JNK by protein kinase C [corrected].

Z. Ronai, Lu-Hai Wang, A. Bhoumik, 2005, Molecular cell.

ErbB2-overexpressing human mammary carcinoma cells display an increased requirement for the phosphatidylinositol 3-kinase signaling pathway in anchorage-independent growth

U. Hermanto, C. S. Zong, Lu-Hai Wang, 2001, Oncogene.

Stat5 is a physiological substrate of the insulin receptor.

L. Wang, R. Kohanski, H. Sadowski, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Proto-oncogene c-ros codes for a molecule with structural features common to those of growth factor receptors and displays tissue specific and developmentally regulated expression

M. Shibuya, L. Wang, M. Hsu, 1986, Molecular and cellular biology.

Inhibition of mitogen-activated protein kinase kinase selectively inhibits cell proliferation in human breast cancer cells displaying enhanced insulin-like growth factor I-mediated mitogen-activated protein kinase activation.

L. Wang, U. Hermanto, C. S. Zong, 2000, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Twist Is Transcriptionally Induced by Activation of STAT3 and Mediates STAT3 Oncogenic Function*

D. Coppola, J. Cheng, Mei Sun, 2008, Journal of Biological Chemistry.

Distinct Role of Phosphatidylinositol 3-Kinase and Rho Family GTPases in Vav3-induced Cell Transformation, Cell Motility, and Morphological Changes*

L. Wang, L. Zeng, P. Sachdev, 2002, The Journal of Biological Chemistry.

Rapamycin by itself and additively in combination with carboplatin inhibits the growth of ovarian cancer cells.

Wei Li, J. Chan, P. Schlosshauer, 2009, Gynecologic oncology.