J. Black

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Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy.

S. Waxman, J. Kocsis, J. Black, 1994, Journal of neurophysiology.

Structure of oxyntic cell membranes during conditions of rest and secretion of HCl as revealed by freeze‐fracture

J. G. Forte, T. Forte, J. Black, 1980, The Anatomical record.

A painful neuropathy-associated Nav1.7 mutant leads to time-dependent degeneration of small-diameter axons associated with intracellular Ca2+ dysregulation and decrease in ATP levels

S. Waxman, G. Lauria, J. Hoeijmakers, 2016, Molecular pain.

Sodium channels and pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Sodium channels in normal and pathological pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 2010, Annual review of neuroscience.

Sodium Channels Contribute to Degeneration of Dorsal Root Ganglion Neurites Induced by Mitochondrial Dysfunction in an In Vitro Model of Axonal Injury

S. Waxman, Insil Kim, M. Estacion, 2013, The Journal of Neuroscience.

Rescue of alpha-SNS sodium channel expression in small dorsal root ganglion neurons after axotomy by nerve growth factor in vivo.

S. Dib-Hajj, S. Waxman, A. Kenney, 1998, Journal of neurophysiology.

The NaV1.7 sodium channel: from molecule to man

S. Dib-Hajj, S. Waxman, Yang Yang, 2012, Nature Reviews Neuroscience.

NaV1.9: a sodium channel linked to human pain

S. Dib-Hajj, S. Waxman, J. Black, 2015, Nature Reviews Neuroscience.

Two tetrodotoxin‐resistant sodium channels in human dorsal root ganglion neurons

S. Dib-Hajj, S. Waxman, J. A. Black, 1999, FEBS letters.

Sodium channels contribute to microglia/macrophage activation and function in EAE and MS

A. Lo, S. Waxman, J. Newcombe, 2005, Glia.

A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis

S. Dib-Hajj, S. Waxman, S. Shields, 2012, Annals of neurology.

Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosis.

J Newcombe, S G Waxman, S. Dib-Hajj, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Transfection of rat or mouse neurons by biolistics or electroporation

S. Dib-Hajj, S. Waxman, T. Cummins, 2009, Nature Protocols.

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger.

Jia Newcombe, Stephen G Waxman, S. Waxman, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Sodium channels and their genes: dynamic expression in the normal nervous system, dysregulation in disease states 1 1 Published on the World Wide Web on 15 August 2000.

S. Dib-Hajj, S. Waxman, T. Cummins, 2000, Brain Research.

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy.

S. Dib-Hajj, S. Waxman, J. A. Black, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Schwann cells modulate sodium channel expression in spinal sensory neurons in vitro

S. Dib-Hajj, S. Waxman, J. A. Black, 1997, Glia.

Sodium channel α-subunit mRNAs I, II, III, NaG, Na6 and hNE (PN1): Different expression patterns in developing rat nervous system

S. Dib-Hajj, S. Waxman, P. Felts, 1997 .

Spinal sensory neurons express multiple sodium channel α-subunit mRNAs

S. Dib-Hajj, S. Waxman, J. Kocsis, 1996 .

Sodium channel mRNA in the B104 neuroblastoma cell line

S. Dib-Hajj, A. Hinson, S. Waxman, 1996, FEBS letters.

Type II brain sodium channel expression in non-neuronal cells: embryonic rat osteoblasts.

S. Waxman, W. Catterall, J. A. Black, 1995, Brain research. Molecular brain research.

Expression of sodium channel α- and β-subunits in the nervous system of themyelin-deficient rat

S. Waxman, J. A. Black, P. Felts, 1995, Journal of neurocytology.

Isoform‐specific expression of sodium channels in astrocytes in vitro: Immunocytochemical observations

S. Waxman, W. Catterall, B. Ransom, 1995, Glia.

Differential up-regulation of sodium channel α- and β1-subunit mRNAs in cultured embryonic DRG neurons following exposure to NGF

S. Waxman, K. Zur, J. A. Black, 1995 .

Sodium channel mRNAs in cultured spinal cord astrocytes: in situ hybridization in identified cell types.

S. Waxman, K. Zur, H. Sontheimer, 1994, Brain research. Molecular brain research.

The expression of rat brain voltage-sensitive Na+ channel mRNAs in astrocytes.

S. Waxman, Y. Oh, J. Black, 1994, Brain research. Molecular brain research.

Sodium channel mRNAs I, II and III in the CNS: cell-specific expression.

S G Waxman, S. Waxman, H. Higashida, 1994, Brain research. Molecular brain research.

Primary motor neurons fail to up‐regulate voltage‐gated sodium channel Nav1.3/brain type III following axotomy resulting from spinal cord injury

S. Waxman, J. A. Black, B. C. Hains, 2002, Journal of neuroscience research.

Sodium channels and microglial function

S. Waxman, J. Black, 2012, Experimental Neurology.

Sodium channel activity modulates multiple functions in microglia

S. Waxman, Shujun Liu, J. Black, 2009, Glia.

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination.

Stephen G Waxman, A. Lo, S. Waxman, 2003, Brain : a journal of neurology.

From genes to pain: Nav1.7 and human pain disorders

Sulayman D. Dib-Hajj, Stephen G. Waxman, S. Dib-Hajj, 2007, Trends in Neurosciences.

Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury

Stephen G Waxman, S. Waxman, J. A. Black, 2003, The Journal of comparative neurology.

Multiple sodium channel isoforms and mitogen‐activated protein kinases are present in painful human neuromas

S. Waxman, L. Nikolajsen, T. S. Jensen, 2008, Annals of neurology.

Voltage-gated sodium channel expression in rat and human epidermal keratinocytes: Evidence for a role in pain

S. Dib-Hajj, S. Waxman, F. Rice, 2008, PAIN.

Distribution of sodium channels in chronically demyelinated spinal cord axons: immuno-ultrastructural localization and electrophysiological observations

S. Waxman, J. Kocsis, K. Smith, 1991, Brain Research.

The perinodal astrocyte

S. Waxman, J. Black, 1988, Glia.

Effects of delayed myelination by oligodendrocytes and Schwann cells on the macromolecular structure of axonal membrane in rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1986, Journal of neurocytology.

A quantitative study of developing axons and glia following altered gliogenesis in rat optic nerve

B. Ransom, S. G. Waxman, J. Black, 1986, Brain Research.

Molecular structure of the axolemma of developing axons following altered gliogenesis in rat optic nerve.

S. Waxman, J. A. Black, J. Black, 1986, Developmental biology.

Perinodal astrocytic processes at nodes of ranvier in developing normal and glial cell deficient rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1985, Brain Research.

Rat optic nerve: Disruption of gliogenesis with 5-azacytidine during early postnatal development

S. Waxman, B. Ransom, J. A. Black, 1985, Brain Research.

Membrane ultrastructure of developing axons in glial cell deficient rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1985, Journal of neurocytology.

Correlation of Nav1.8 and Nav1.9 sodium channel expression with neuropathic pain in human subjects with lingual nerve neuromas

S. Waxman, J. A. Black, F. Boissonade, 2013, Molecular pain.

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Masaki Tanaka, S. Waxman, J. A. Black, 2004, Pain.

Molecular identities of two tetrodotoxin-resistant sodium channels in corneal axons.

S. Waxman, J. Black, 2002, Experimental eye research.

Transient presence and functional interaction of endogenous GABA and GABAA receptors in developing rat optic nerve

K. Sakatani, Kaoru Sakatani, J. Kocsis, 1992, Proceedings of the Royal Society of London. Series B: Biological Sciences.

NaV1.7 accumulates and co-localizes with phosphorylated ERK1/2 within transected axons in early experimental neuromas

S. Waxman, A. Gasser, A. Persson, 2011, Experimental Neurology.

Neuroma Removal for Neuropathic Pain: Efficacy and Predictive Value of Lidocaine Infusion

S. Waxman, J. A. Black, T. Jensen, 2010, The Clinical journal of pain.

SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model

S. Dib-Hajj, S. Waxman, T. Cummins, 1998, Neuroreport.

Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury

S. Waxman, B. C. Hains, C. Saab, 2003, The Journal of Neuroscience.

Sodium channels, excitability of primary sensory neurons, and the molecular basis of pain

S. Dib-Hajj, S. Waxman, J. Fjell, 1999, Muscle & nerve.

Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs.

S. Dib-Hajj, S. Waxman, J. Kocsis, 1996, Brain research. Molecular brain research.

Voltage-gated sodium channels: therapeutic targets for pain.

S. Dib-Hajj, S. Waxman, J. Black, 2009, Pain medicine.

Voltage-gated sodium channels: therapeutic targets for pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 2009, Pain medicine.

Sodium channel Nav1.7 in vascular myocytes, endothelium, and innervating axons in human skin

S. Waxman, F. Rice, J. A. Black, 2015, Molecular pain.

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons.

A. M. Rush, S. Dib-Hajj, S. Waxman, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Noncanonical Roles of Voltage-Gated Sodium Channels

S. Waxman, J. Black, 2013, Neuron.

Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons

S. Dib-Hajj, S. Waxman, L. Tyrrell, 2011, Molecular pain.

Differential expression of sodium channel genes in retinal ganglion cells.

S. Dib-Hajj, S. Waxman, J. Fjell, 1997, Brain research. Molecular brain research.

The Presence and Role of the Tetrodotoxin-Resistant Sodium Channel Nav1.9 (NaN) in Nociceptive Primary Afferent Neurons

S. Dib-Hajj, S. Waxman, L. Djouhri, 2002, The Journal of Neuroscience.

Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy

S. Dib-Hajj, S. Waxman, T. Cummins, 2000, The Journal of Neuroscience.

Na+ channel beta 1 subunit mRNA: differential expression in rat spinal sensory neurons.

S. Waxman, J. A. Black, Y. Oh, 1995, Brain research. Molecular brain research.

The voltage-dependent sodium channel in mammalian CNS and PNS: antibody characterization and immunocytochemical localization

S. Waxman, J. A. Black, K. Angelides, 1990, Brain Research.

Sodium-calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals

S. Waxman, J. A. Black, A. Gasser, 2010, Molecular pain.

Na(+)-current expression in rat hippocampal astrocytes in vitro: alterations during development.

A. Cornell-Bell, S. Waxman, J. A. Black, 1991, Journal of neurophysiology.

In situ demonstration of mature oligodendrocytes and their processes: An immunocytochemical study with a new monoclonal antibody, Rip

S. Hockfield, S. Waxman, B. Friedman, 1989, Glia.

CAP-1A is a novel linker that binds clathrin and the voltage-gated sodium channel Nav1.8

S. Dib-Hajj, S. Waxman, T. Cummins, 2005, Molecular and Cellular Neuroscience.

Selective Expression of a Persistent Tetrodotoxin-Resistant Na+ Current and NaV1.9 Subunit in Myenteric Sensory Neurons

Stephen G. Waxman, Patrick Delmas, S. Waxman, 2003, The Journal of Neuroscience.

Apoptosis of vasopressinergic hypothalamic neurons in chronic diabetes mellitus

S. Waxman, B. C. Hains, M. Craner, 2004, Neurobiology of Disease.

Sodium channels in astroglia and microglia

S. Waxman, J. Black, L. Pappalardo, 2016, Glia.

Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

Stephen G Waxman, A. Lo, S. Waxman, 2004, Brain : a journal of neurology.

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

R. LaMotte, S. Dib-Hajj, S. Waxman, 1999, PAIN®.

Preferential expression of IGF-I in small DRG neurons and down-regulation following injury

S. Waxman, M. Craner, J. Black, 2002, Neuroreport.

Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn

S. Dib-Hajj, S. Waxman, N. Frézel, 2012, Molecular pain.

Changes of sodium channel expression in experimental painful diabetic neuropathy

S. Waxman, M. Renganathan, M. Craner, 2002, Annals of neurology.

Changes in expression of voltage‐gated potassium channels in dorsal root ganglion neurons following axotomy

S. Waxman, K. Ishikawa, J. Black, 1999, Muscle & nerve.

Nav1.5 in astrocytes plays a sex‐specific role in clinical outcomes in a mouse model of multiple sclerosis

S. Waxman, O. A. Samad, Shujun Liu, 2018, Glia.

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

S. Dib-Hajj, S. Waxman, D. Vasylyev, 2014, Experimental Neurology.

ERK1/2 Mitogen-Activated Protein Kinase Phosphorylates Sodium Channel Nav1.7 and Alters Its Gating Properties

S. Dib-Hajj, S. Waxman, Séverine Stamboulian, 2010, The Journal of Neuroscience.

Voltage‐gated sodium channel Nav1.5 contributes to astrogliosis in an in vitro model of glial injury via reverse Na+/Ca2+ exchange

S. Waxman, O. A. Samad, J. Black, 2014, Glia.

Sodium Channel Expression Within Chronic Multiple Sclerosis Plaques

S. Waxman, J. Newcombe, B. Trapp, 2007, Journal of neuropathology and experimental neurology.

NaN/Nav1.9: a sodium channel with unique properties

S. Dib-Hajj, S. Waxman, T. Cummins, 2002, Trends in Neurosciences.

Anoxic injury of rat optic nerve: ultrastructural evidence for coupling between Na+ influx and Ca2+-mediated injury in myelinated CNS axons

S. Waxman, B. Ransom, P. Stys, 1994, Brain Research.

Dynamics of sodium channel Nav1.5 expression in astrocytes in mouse models of multiple sclerosis

S. Waxman, J. A. Black, Shujun Liu, 2014, Neuroreport.

Specificity of cell-cell coupling in rat optic nerve astrocytes in vitro.

S. Waxman, J. A. Black, H. Sontheimer, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Conduction properties of spinal cord axons in the myelin-deficient rat mutant

J. Kocsis, D. Utzschneider, J. Black, 1992, Neuroscience.

Macromolecular structure of axon membrane and action potential conduction in myelin deficient and myelin deficient heterozygote rat optic nerves

S G Waxman, S. Waxman, I. Duncan, 1990, Journal of neurocytology.

Use of Freeze-Fracture in Neurobiology

J. Black, 1990 .

Freeze–fracture studies on unmyelinated axolemma of rat cervical sympathetic trunk : correlation with saxitoxin binding

S. Waxman, J. A. Black, J. Black, 1988, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Membrane specialization and axo-glial association in the rat retinal nerve fibre layer: freeze-fracture observations

S. Waxman, J. A. Black, C. Hildebrand, 1984, Journal of neurocytology.

Voltage‐Gated Sodium Channels: Multiple Roles in the Pathophysiology of Pain

S. Dib-Hajj, S. Waxman, B. C. Hains, 2008 .

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

S. Waxman, J. Newcombe, J. A. Black, 2010, Brain : a journal of neurology.

Ion channels in spinal cord astrocytes in vitro. I. Transient expression of high levels of Na+ and K+ channels.

S. Waxman, J. A. Black, H. Sontheimer, 1992, Journal of neurophysiology.

Nav1.5 sodium channels in macrophages in multiple sclerosis lesions

S. Waxman, J. Newcombe, J. A. Black, 2013, Multiple sclerosis.

Nitric oxide is an autocrine regulator of Na(+) currents in axotomized C-type DRG neurons.

S. Waxman, T. Cummins, M. Renganathan, 2000, Journal of neurophysiology.

Molecular and functional remodeling of electrogenic membrane of hypothalamic neurons in response to changes in their input.

S. Waxman, Y. Ibata, T. Cummins, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Ultrastructural identification of HRP‐injected oligodendrocytes in the intact rat optic nerve

B. Ransom, A. Butt, J. Black, 1991, Glia.

GDNF and NGF reverse changes in repriming of TTX-sensitive Na(+) currents following axotomy of dorsal root ganglion neurons.

S. Dib-Hajj, S. Waxman, J. A. Black, 2002, Journal of neurophysiology.

Down-Regulation of Transcripts for Na Channel α -SNS in Spinal Sensory Neurons Following Axotomy

S. Dib-Hajj, S. Waxman, P. Felts, 1996 .

Down-regulation of transcripts for Na channel alpha-SNS in spinal sensory neurons following axotomy.

S. Dib-Hajj, S. Waxman, J. A. Black, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Phosphorylation of Sodium Channel Nav1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons

A. M. Rush, S. Dib-Hajj, S. Waxman, 2008, The Journal of Neuroscience.

Anoxic injury of mammalian central white matter: Decreased susceptibility in myelin‐deficient optic nerve

S. Waxman, B. Ransom, J. Black, 1990, Annals of neurology.

Freeze-fracture ultrastructure of the perinodal astrocyte and associated glial junctions

S. Waxman, J. Black, 1984, Brain Research.

Unmyelinated and myelinated axon membrane from rat corpus callosum: differences in macromolecular structure

Stephen G. Waxman, S. Waxman, Joel A. Black, 1988, Brain Research.

Rat optic nerve: Freeze-fracture studies during development of myelinated axons

Robert E. Foster, Stephen G. Waxman, S. Waxman, 1982, Brain Research.

Slowly Progressive Axonal Degeneration in a Rat Model of Chronic, Nonimmune-Mediated Demyelination

S. Waxman, I. Duncan, A. Compston, 2010, Journal of neuropathology and experimental neurology.

Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo.

A. Lo, S. Waxman, C. Saab, 2003, Journal of neurophysiology.

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

S G Waxman, S. Dib-Hajj, S. Waxman, 1999, The Journal of Neuroscience.

Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

S. Waxman, J. A. Black, J. P. Klein, 2002, Neuroreport.

Freeze-fracture ultrastructure of rat C.N.S. and P.N.S. nonmyelinated axolemma

S. Waxman, J. A. Black, R. Foster, 1981, Journal of neurocytology.

Voltage-gated Na+ channels in glia: properties and possible functions

Harald Sontheimer, Stephen G Waxman, S. Waxman, 1996, Trends in Neurosciences.

Upregulation of a silent sodium channel after peripheral, but not central, nerve injury in DRG neurons.

Y. H. Chen, S. Waxman, J. A. Black, 1999, Journal of neurophysiology.

Sodium channel alpha-subunit mRNAs I, II, III, NaG, Na6 and hNE (PN1): different expression patterns in developing rat nervous system.

S. Dib-Hajj, S. Waxman, P. Felts, 1997, Brain research. Molecular brain research.

Sodium channel expression: a dynamic process in neurons and non-neuronal cells.

S. Waxman, J. Black, 1996, Developmental neuroscience.

Ca 2 toxicity due to reverse Na / Ca 2 exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav 1 . 7 mutation

S. Waxman, J. A. Black, G. Lauria, 2015 .

Sodium Channels, Mitochondria, and Axonal Degeneration in Peripheral Neuropathy.

S. Waxman, M. Estacion, J. Hoeijmakers, 2016, Trends in molecular medicine.

Neuropathy‐associated NaV1.7 variant I228M impairs integrity of dorsal root ganglion neuron axons

S. Waxman, I. Merkies, C. Faber, 2013, Annals of neurology.

Differential modulation of sodium channel Nav1.6 by two members of the fibroblast growth factor homologous factor 2 subfamily

A. M. Rush, S. Dib-Hajj, S. Waxman, 2006, The European journal of neuroscience.

Contactin Associates with Sodium Channel Nav1.3 in Native Tissues and Increases Channel Density at the Cell Surface

A. M. Rush, S. Dib-Hajj, S. Waxman, 2004, The Journal of Neuroscience.

Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar purkinje cells

S. Waxman, M. Gelderblom, M Renganathan, 2003, Brain Research.

Localization of the tetrodotoxin‐resistant sodium channel NaN in nociceptors

S. Dib-Hajj, S. Waxman, J. Fjell, 2000, Neuroreport.

Low density of sodium channels supports action potential conduction in axons of neonatal rat optic nerve.

S G Waxman, S. Waxman, J. Kocsis, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Differential role of GDNF and NGF in the maintenance of two TTX-resistant sodium channels in adult DRG neurons.

S. Dib-Hajj, S. Waxman, J. Fjell, 1999, Brain research. Molecular brain research.

Three types of sodium channels in adult rat dorsal root ganglion neurons

S. Waxman, J. Kocsis, J. M. Caffrey, 1992, Brain Research.

Sodium channels in astrocytes of rat optic nerve in situ: Immuno‐electron microscopic studies

S. Waxman, J. A. Black, K. Angelides, 1999, Glia.

Immuno-ultrastructural localization of sodium channels at nodes of Ranvier and perinodal astrocytes in rat optic nerve

S. Waxman, K. Angelides, B. Friedman, 1989, Proceedings of the Royal Society of London. B. Biological Sciences.

Macromolecular structure of the Schwann cell membrane Perinodal microvilli

S. Waxman, J. A. Black, J. Black, 1987, Journal of the Neurological Sciences.

Immuno-Localization of Sodium Channels in Axon Membrane and Astrocytes and Schwann Cells in vivo and in vitro

A. Cornell-Bell, S. Waxman, J. A. Black, 1990 .

Glial cells have heart: rH1 Na+ channel mRNA and protein in spinal cord astrocytes

S. Dib-Hajj, S. Waxman, A. Hinson, 1998, Glia.

Ion channel organization of the myelinated fiber

S. Waxman, J. Kocsis, J. Black, 1990, Trends in Neurosciences.

Upregulation and colocalization of p75 and Nav1.8 in Purkinje neurons in experimental autoimmune encephalomyelitis

Stephen G. Waxman, S. Waxman, Joel A. Black, 2004, Neuroscience Letters.

Sodium channels and the molecular pathophysiology of pain.

S. Dib-Hajj, S. Waxman, T. Cummins, 2000, Progress in brain research.

Endogenous NMDA-receptor activation regulates glutamate release in cultured spinal neurons.

S. Waxman, J. A. Black, A. Robert, 1998, Journal of neurophysiology.

Tapered withdrawal of phenytoin removes protective effect in EAE without inflammatory rebound and mortality

S. Waxman, J. A. Black, S. Liu, 2014, Journal of the Neurological Sciences.

Exacerbation of experimental autoimmune encephalomyelitis after withdrawal of phenytoin and carbamazepine

S. Waxman, J. A. Black, Shujun Liu, 2007, Annals of neurology.

Subcellular Distribution of UDP‐Galactose:Ceramide Galactosyltransferase in Rat Brain Oligodendroglia

J. A. Black, R. Yu, C. Sato, 1988, Journal of neurochemistry.

Fibroblast growth factor homologous factor 2 (FGF-13) associates with Nav1.7 in DRG neurons and alters its current properties in an isoform-dependent manner

S. Dib-Hajj, S. Waxman, A. Lampert, 2019, Neurobiology of pain.

Ontogenesis of the axolemma and axoglial relationships in myelinated fibers: electrophysiological and freeze-fracture correlates of membrane plasticity.

S. Waxman, R. Foster, J. Black, 1983, International review of neurobiology.

Na+ channel expression along axons in multiple sclerosis and its models.

Stephen G Waxman, S. Waxman, Matthew J Craner, 2004, Trends in pharmacological sciences.

Abnormal expression of SNS/PN3 sodium channel in cerebellar Purkinje cells following loss of myelin in the taiep rat.

S. Dib-Hajj, S. Waxman, I. Duncan, 1999, Neuroreport.

Sodium channel Na(v)1.6 is expressed along nonmyelinated axons and it contributes to conduction.

S. Waxman, M. Renganathan, J. Black, 2002, Brain research. Molecular brain research.

Contribution of sodium channels to lamellipodial protrusion and Rac1 and ERK1/2 activation in ATP‐stimulated microglia

S. Waxman, M. Estacion, H. Ahn, 2014, Glia.

Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis

Stephen G Waxman, A. Lo, S. Waxman, 2003, Journal of neuropathology and experimental neurology.

Molecular Reconstruction of Nodes of Ranvier after Remyelination by Transplanted Olfactory Ensheathing Cells in the Demyelinated Spinal Cord

S. Waxman, J. A. Black, J. Kocsis, 2006, The Journal of Neuroscience.

Remyelination of dorsal column axons by endogenous Schwann cells restores the normal pattern of Nav1.6 and Kv1.2 at nodes of Ranvier.

S. Waxman, Kenneth J. Smith, J. Black, 2006, Brain : a journal of neurology.

Retinal involvement in multiple sclerosis

S. Waxman, J. A. Black, J. Black, 2007, Neurology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.

Stephen G Waxman, S. Waxman, J. A. Black, 2006, Brain : a journal of neurology.

Expression of mRNA for a sodium channel in subfamily 2 in spinal sensory neurons

S. Waxman, J. Black, 1996, Neurochemical Research.

Na+ channel β1 subunit mRNA expression in developing rat central nervous system

S. Waxman, Y. Oh, S. Sashihara, 1995 .

Neuroprotection of axons with phenytoin in experimental allergic encephalomyelitis

A. Lo, S. Waxman, J. A. Black, 2002, Neuroreport.

Introduction. Astrocytes in the injured brain: is gliosis adaptive or maladaptive?

George E Barreto, G. Barreto, Joel A Black, 2014, Neuroscience letters.

Voltage-gated sodium channels and the molecular pathogenesis of pain: a review.

S. Dib-Hajj, S. Waxman, T. Cummins, 2000, Journal of rehabilitation research and development.

Sodium channel expression in NGF‐overexpressing transgenic mice

S. Waxman, J. Fjell, K. Fried, 1999, Journal of neuroscience research.

Freeze‐fracture heterogeneity of the axolemma of premyelinated fibers in the CNS

S. Waxman, J. A. Black, R. Foster, 1982, Neurology.

Direct Interaction with Contactin Targets Voltage-gated Sodium Channel Nav1.9/NaN to the Cell Membrane*

Z. Lian, S. Dib-Hajj, S. Waxman, 2001, The Journal of Biological Chemistry.

NGF has opposing effects on Na+ channel III and SNS gene expression in spinal sensory neurons

S. Dib-Hajj, S. Waxman, J. Black, 1997, Neuroreport.

Spinal cord astrocytes in vitro: Phenotypic diversity and sodium channel immunoreactivity

S. Waxman, J. A. Black, H. Sontheimer, 1993, Glia.

Sodium channel expression in optic nerve astrocytes chronically deprived of axonal contact

S. Waxman, B. Ransom, J. A. Black, 1992, Glia.

Tetrodotoxin-resistant sodium channels Nav1.8/SNS and Nav1.9/NaN in afferent neurons innervating urinary bladder in control and spinal cord injured rats

S. Waxman, W. C. Groat, N. Yoshimura, 2003, Brain Research.

Carbonic anhydrase activity develops postnatally in the rat optic nerve.

S. Waxman, B. Ransom, J. A. Black, 1987, Brain research.

Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation.

S. Waxman, J. A. Black, G. Lauria, 2015, Journal of neurophysiology.

Sodium channels and the molecular basis for pain

S. Dib-Hajj, S. Waxman, T. Cummins, 2002 .

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons

S. Dib-Hajj, S. Waxman, T. Cummins, 2000, The Journal of Neuroscience.

NaG: A sodium channel‐like mRNA shared by Schwann cells and other neural crest derivatives

S. Dib-Hajj, P. Felts, S. G. Waxman, 1997, Glia.

Type II sodium channels in spinal cord astrocytes in situ: Immunocytochemical observations

S. Waxman, W. Catterall, B. Ransom, 1994, Glia.

Ultrastructural concomitants of anoxic injury and early post-anoxic recovery in rat optic nerve

Peter K. Stys, Stephen G. Waxman, Bruce R. Ransom, 1992, Brain Research.

Annexin II/p11 is up-regulated in Purkinje cells in EAE and MS

A. Lo, S. Waxman, J. Newcombe, 2003, Neuroreport.

Quantitative analysis of rat synaptosomes and cerebrum using high-resolution 1H magnetic resonance spectroscopy.

O. Petroff, J. Prichard, A. Burlina, 1992, Clinica chimica acta; international journal of clinical chemistry.

In vivo NGF deprivation reduces SNS expression and TTX-R sodium currents in IB4-negative DRG neurons.

S. Waxman, J. Fjell, K. Fried, 1999, Journal of neurophysiology.

NaV1.7: Stress-induced changes in immunoreactivity within magnocellular neurosecretory neurons of the supraoptic nucleus

S. Waxman, J. A. Black, J. Hoeijmakers, 2013, Molecular pain.

Protection of the axonal cytoskeleton in anoxic optic nerve by decreased extracellular calcium

S. Waxman, B. Ransom, J. A. Black, 1993, Brain Research.

Axonal Protection with Sodium Channel Blocking Agents in Models of Multiple Sclerosis

S. Waxman, J. A. Black, Kenneth J. Smith, 2013 .

Sodium channel expression detected with antibody 7493 in A2B5+ and A2B5− astrocytes from rat optic nerve in vitro

S. Waxman, J. A. Black, K. Angelides, 1990, Glia.

Pathophysiology of demyelinated axons

Stephen G. Waxman, S. Waxman, J. Kocsis, 1995 .

Phenytoin protects central axons in experimental autoimmune encephalomyelitis

S. Waxman, J. A. Black, J. Black, 2008, Journal of the Neurological Sciences.

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

S. Dib-Hajj, D. Vasylyev, J. Black, 2014, Experimental Neurology.

The expression of sodium channels in astrocytes in situ and in vitro.

S. Waxman, J. A. Black, H. Sontheimer, 1992, Progress in brain research.

Sodium channels and pain.

S. Dib-Hajj, S. Waxman, T. Cummins, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Freeze-fracture ultrastructure of developing and adult non-myelinated ganglion cell axolemma in the retinal nerve fibre layer

S. Waxman, J. A. Black, R. Foster, 1983, Journal of neurocytology.

Membrane‐Associated Sodium Channels and Cytoplasmic Precursors in Glial Cells

S. Waxman, J. A. Black, K. Angelides, 1991, Annals of the New York Academy of Sciences.

Specialization of astrocytic membrane at glia limitans in rat optic nerve: Freeze-fracture observations

S. Waxman, J. A. Black, J. Black, 1985, Neuroscience Letters.

Sodium channels in the cytoplasm of Schwann cells.

S. Waxman, J. A. Black, K. Angelides, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Dynamic aspects of sodium channel expression in astrocytes.

S. Waxman, J. A. Black, H. Sontheimer, 1993, Advances in neurology.

Macromolecular Structure of Axonal Membrane during Acute Experimental Allergic Encephalomyelitis in Rat and Guinea Pig Spinal Cord

S. Waxman, J. A. Black, M. Smith, 1987, Journal of neuropathology and experimental neurology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE. Commentary

S. Waxman, B. C. Hains, Kenneth J. Smith, 2006 .

Axotomy does not up-regulate expression of sodium channel Na(v)1.8 in Purkinje cells.

C. Sotelo, S. Waxman, I. Dusart, 2002, Brain research. Molecular brain research.

Voltage-gated sodium channels and pain associated with nerve injury and neuropathies

S. Dib-Hajj, S. Waxman, B. C. Hains, 2005 .

Molecular Contactin Associates with Sodium Channel Nav 1 . 3 in Native Tissues and Increases Channel Density at the Cell Surface

S. Dib-Hajj, S. Waxman, L. Tyrrell, 2004 .

Emotion , Cognition , and Mental State Representation in Amygdala and Prefrontal Cortex

R. Mcinnes, S. Waxman, R. Watts, 2012 .

Expression of skeletal muscle‐type voltage‐gated Na+ channel in rat and human prostate cancer cell lines

S. Dib-Hajj, S. Waxman, J. A. Black, 1998, FEBS letters.

The effects of microfilament disrupting agents on HCl secretion and ultrastructure of piglet gastric oxyntic cells.

J. A. Black, J. G. Forte, T. Forte, 1982, Gastroenterology.

Fibroblast growth factor homologous factor 2 (FGF-13) associates with Nav1.7 in DRG neurons and alters its current properties in an isoform-dependent manner

S. Dib-Hajj, S. Waxman, A. Lampert, 2019, Neurobiology of pain.

Filipin-cholesterol binding in CNS axons prior to myelination: evidence for microheterogeneity in premyelinated axolemma

S. Waxman, R. Fields, J. A. Black, 1987, Brain Research.

Cell Coupling Is Restricted to Subpopulations of Astrocytes Cultured from Rat Hippocampus and Optic Nerve

A. Cornell-Bell, H. Sontheimer, J. Minturn, 1991, Annals of the New York Academy of Sciences.

Membrane structure of vesiculotubular complexes in developing axons in rat optic nerve: freeze—fracture evidence for sequential membrane assembly

S. Waxman, J. A. Black, J. Black, 1985, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Axo-glial relations in the retina-optic nerve junction of the adult rat: freeze-fracture observations on axon membrane structure

S. Waxman, J. A. Black, C. Hildebrand, 1985, Journal of neurocytology.

Two types of Na +‐currents in cultured rat optic nerve astrocytes: Changes with time in culture and with age of culture derivation

S. Waxman, B. Ransom, J. A. Black, 1991, Journal of neuroscience research.

Injury-Induced Gliosis In Vitro After TTX-Sensitive to TTX-Resistant Sodium Currents Spinal Cord Astrocytes Display a Switch From

S. Waxman, J. Newcombe, H. Sontheimer, 2015 .