M. Morán

发表

Neural inputs into the temporopolar cortex of the rhesus monkey

M. Mesulam, M. Mesulam, E. Mufson, 1987, The Journal of comparative neurology.

GSK3β overexpression induces neuronal death and a depletion of the neurogenic niches in the dentate gyrus

J. García-Verdugo, J. Lucas, J. Ávila, 2010, Hippocampus.

Decreased nuclear β‐catenin, tau hyperphosphorylation and neurodegeneration in GSK‐3β conditional transgenic mice

René Hen, Jesús Avila, R. Hen, 2001 .

Relative growth and maturation of axon size and myelin thickness in the tibial nerve of the rat

D. O'Leary, R. King, P. Thomas, 2004, Acta Neuropathologica.

Relative growth and maturation of axin size and myelin thickness in the tibial nerve of the rat

D. O'Leary, R. King, P. Thomas, 2004, Acta Neuropathologica.

Hippocampal neuronal subpopulations are differentially affected in double transgenic mice overexpressing frontotemporal dementia and parkinsonism linked to chromosome 17 tau and glycogen synthase kinase-3β

J. Lucas, F. Hernández, T. Engel, 2008, Neuroscience.

Cooexpression of FTDP-17 tau and GSK-3β in transgenic mice induce tau polymerization and neurodegeneration

J. Lucas, J. Ávila, F. Hernández, 2006, Neurobiology of Aging.

Ultrastructural localization of fructose‐1,6‐bisphosphatase in mouse brain

J. Ávila, M. Morán, Z. Velásquez, 2011, Microscopy research and technique.

Ultrastructural localization of acetylcholinesterase in neurofibrillary tangles, neuropil threads and senile plaques in aged and Alzheimer's brain

E. Mufson, P. Gómez-Ramos, M. Morán, 1992, Brain Research.

Full Motor Recovery Despite Striatal Neuron Loss and Formation of Irreversible Amyloid-Like Inclusions in a Conditional Mouse Model of Huntington's Disease

J. Lucas, J. Alberch, M. Diaz-hernandez, 2005, The Journal of Neuroscience.

α-Synuclein accumulates in huntingtin inclusions but forms independent filaments and its deficiency attenuates early phenotype in a mouse model of Huntington's disease.

J. Lucas, I. Ferrer, M. Miras-Portugal, 2012, Human molecular genetics.

Alpha-synuclein accumulates in huntingtin inclusions but forms independent filaments and its deficiency attenuates early phenotype in a mouse model of Huntington ́ s disease

J. Lucas, I. Ferrer, M. Morán, 2011 .

Accelerated amyloid deposition, neurofibrillary degeneration and neuronal loss in double mutant APP/tau transgenic mice

I. Ferrer, T. Gómez-Isla, B. Puig, 2005, Neurobiology of Disease.

Decreased nuclear beta-catenin, tau hyperphosphorylation and neurodegeneration in GSK-3beta conditional transgenic mice.

R. Hen, J. Lucas, J. Ávila, 2001, The EMBO journal.

Microtubule-associated protein MAP1B showing a fetal phosphorylation pattern is present in sites of neurofibrillary degeneration in brains of Alzheimer's disease patients.

J. Ávila, L. Ulloa, E. Montejo de Garcíni, 1994, Brain research. Molecular brain research.

Phosphorylated tau in neuritic plaques of APPsw/Tauvlw transgenic mice and Alzheimer disease

J. Ávila, I. Ferrer, Mar Pérez, 2008, Acta Neuropathologica.

Characterization of a double (amyloid precursor protein-tau) transgenic: Tau phosphorylation and aggregation

J. Ávila, I. Ferrer, E. Ribe, 2005, Neuroscience.

FTDP-17 Mutations in tau Transgenic Mice Provoke Lysosomal Abnormalities and Tau Filaments in Forebrain

J. Lucas, J. Ávila, F. Hernández, 2001, Molecular and Cellular Neuroscience.

Ultrastructural aspects of neurofibrillary tangle formation in aging and Alzheimer's disease

P. Gómez-Ramos, M. Morán, Pilar Gómez‐Ramos, 1998, Microscopy research and technique.

Ultrastructural localization of butyrylcholinesterase in senile plaques in the brains of aged and Alzheimer disease patients.

P. Gómez-Ramos, M. Morán, 1997, Molecular and chemical neuropathology.

Neuronal Induction of the Immunoproteasome in Huntington's Disease

Isidro Ferrer, I. Ferrer, J. Lucas, 2003, The Journal of Neuroscience.

Colocalization of cholinesterases with β amyloid protein in aged and Alzheimer's brains

E. Mufson, P. Gómez-Ramos, M. Morán, 2004, Acta Neuropathologica.

Cholinesterases colocalize with sites of neurofibrillary degeneration in aged and Alzheimer's brains

E. Mufson, P. Gómez-Ramos, M. Morán, 1994, Acta Neuropathologica.

Association of cholinesterases with amyloid in extracellular neurofibrillary tangles

J. Cebrian, P. Gómez-Ramos, M. Morán, 1997, Brain Research.

Inhibition of 26S proteasome activity by huntingtin filaments but not inclusion bodies isolated from mouse and human brain

Beatriz Alvarez-Castelao, J. Lucas, F. Hernández, 2006, Journal of neurochemistry.

Biochemical, Ultrastructural, and Reversibility Studies on Huntingtin Filaments Isolated from Mouse and Human Brain

Isidro Ferrer, Jesús Avila, I. Ferrer, 2004, The Journal of Neuroscience.

Relative growth and maturation of axon size and myelin thickness in the tibial nerve of the rat. 2. Effect of streptozotocin-induced diabetes.

D. O'Leary, R. King, P. Thomas, 1990, Acta neuropathologica.

Apolipoprotein E immunoreactivity in neurons and neurofibrillary degeneration of aged non‐demented and alzheimer's disease patients

E. Mufson, P. Gómez-Ramos, M. Morán, 2001, Microscopy research and technique.

Glial Changes in Aging and Alzheimer’s Disease

P. Gómez-Ramos, M. Morán, 1998 .

Ubiquitinated granular structures and initial neurofibrillary changes in the human brain

P. Gómez-Ramos, M. Morán, M. L. G. Gil, 2001, Journal of the Neurological Sciences.

Topographical organization of the thalamic afferent connections to the motor cortex in the cat

F. Reinoso-suárez, M. Morán, 1988, The Journal of comparative neurology.

Alzheimer's disease-type neurofibrillary degeneration in verrucose dysplasias of the cerebral cortex

A. Probst, C. Navarro, P. Gómez-Ramos, 2004, Acta Neuropathologica.

[Diagnosis of Alzheimer's disease. Evaluation of senile plaques of the diffuse type].

E. Mufson, A. Cabello, S. Madero, 1992, Medicina clinica.