Angelique M. L. Scantlebery

β-Cyclodextrin counteracts obesity in Western diet-fed mice but elicits a nephrotoxic effect

P. Ochodnický, S. Florquin, J. Roelofs, 2019, Scientific Reports.

Metabolic Flexibility and Innate Immunity in Renal Ischemia Reperfusion Injury: The Fine Balance Between Adaptive Repair and Tissue Degeneration

J. Kers, S. Florquin, A. Tammaro, 2020, Frontiers in Immunology.

TLR9 Mediates Remote Liver Injury following Severe Renal Ischemia Reperfusion

T. van der Poll, S. Florquin, J. Leemans, 2015, PloS one.

The dysregulation of metabolic pathways and induction of the pentose phosphate pathway in renal ischaemia–reperfusion injury

G. Schmitz, G. Liebisch, S. Florquin, 2020, The Journal of pathology.

Biotechnological production of reduced and oxidized NAD+ precursors.

R. Houtkooper, Bauke V. Schomakers, M. van Weeghel, 2023, Food research international.

Reduced nicotinamide mononucleotide is a new and potent NAD+ precursor in mammalian cells and mice

S. Denis, R. Houtkooper, Bauke V. Schomakers, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Author Correction: Combining streptozotocin and unilateral nephrectomy is an effective method for inducing experimental diabetic nephropathy in the ‘resistant’ C57Bl/6J mouse strain

S. Florquin, J. Roelofs, J. Leemans, 2019, Scientific Reports.

Combining streptozotocin and unilateral nephrectomy is an effective method for inducing experimental diabetic nephropathy in the ‘resistant’ C57Bl/6J mouse strain

S. Florquin, J. Roelofs, J. Leemans, 2018, Scientific Reports.

NLRX1 does not play a role in diabetes nor the development of diabetic nephropathy induced by multiple low doses of streptozotocin

S. Girardin, S. Florquin, J. Roelofs, 2019, PloS one.

TREM1/3 Deficiency Impairs Tissue Repair After Acute Kidney Injury and Mitochondrial Metabolic Flexibility in Tubular Epithelial Cells

M. Colonna, M. Dessing, S. Florquin, 2019, Front. Immunol..

TREM1/3 Deficiency Impairs Tissue Repair After Acute Kidney Injury and Mitochondrial Metabolic Flexibility in Tubular Epithelial Cells

M. Colonna, M. Dessing, S. Florquin, 2019, Front. Immunol..

ACMSD inhibition corrects fibrosis, inflammation, and DNA damage in MASLD/MASH.

R. Houtkooper, J. Ivanisevic, Jonathan Sulc, 2024, Journal of hepatology.