M. D. de Beer

发表

Characterization of the inbred CE/J mouse strain as amyloid resistant.

J. Sipe, F. D. de Beer, I. Carreras, 1993, The American journal of pathology.

Serum amyloid A impairs the antiinflammatory properties of HDL.

S. Subramanian, A. Chait, K. Elkon, 2016, The Journal of clinical investigation.

Serum amyloid A impairs the antiinflammatory properties of HDL.

S. Subramanian, A. Chait, K. Elkon, 2016, The Journal of clinical investigation.

Serum Amyloid A Facilitates the Binding of High-Density Lipoprotein From Mice Injected With Lipopolysaccharide to Vascular Proteoglycans

A. Chait, T. Vaisar, J. Heinecke, 2011, Arteriosclerosis, thrombosis, and vascular biology.

The role of androgens in female vertebrates.

M. de Beer, M. Beer, N. L. Staub, 1997, General and comparative endocrinology.

SAA does not induce cytokine production in physiological conditions.

N. Webb, F. D. de Beer, Myung-hee Kim, 2013, Cytokine.

In vitro stimulation of immune spleen cells enhances the number of anti-lipid A-producing hybridomas.

J. Verhoef, R. Torensma, B. Dekker, 1989, Journal of immunological methods.

Serum amyloid A3 is pro-atherogenic.

P. Shridas, N. Webb, F. D. de Beer, 2018, Atherosclerosis.

Structural prerequisites for serum amyloid A fibril formation.

C. Kay, M. Kindy, F. D. de Beer, 1993, The Journal of biological chemistry.

Mouse serum amyloid A protein. Complete amino acid sequence and mRNA analysis of a new isoform.

J. Sipe, F. D. de Beer, I. Carreras, 1992, The Biochemical journal.

High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

P. Shridas, N. Webb, M. de Beer, 2018, The Journal of Biological Chemistry.

Hepatocytes direct the formation of a pro-metastatic niche in the liver.

M. Borad, Dexi Liu, S. Yee, 2019, Nature.

Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

A. Chait, P. Shridas, N. Webb, 2017, Journal of Lipid Research.

Serum amyloid A (SAA): influence on HDL-mediated cellular cholesterol efflux.

L. Curtiss, M. Kindy, F. D. de Beer, 1995, Journal of lipid research.

Role of group II secretory phospholipase A2 in atherosclerosis: 1. Increased atherogenesis and altered lipoproteins in transgenic mice expressing group IIa phospholipase A2.

J. Qiao, A. Lusis, L. Castellani, 1999, Arteriosclerosis, thrombosis, and vascular biology.

High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Roger E Bumgarner, Masashi Suzuki, R. Beyer, 2010, Circulation.

Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation

D. Kell, E. Pretorius, T. Nell, 2019, Scientific Reports.

ApoA-II modulates the association of HDL with class B scavenger receptors SR-BI and CD36 Published, JLR Papers in Press, January 16, 2004. DOI 10.1194/jlr.M300417-JLR200

A. Stromberg, L. Castellani, F. D. de Beer, 2004, Journal of Lipid Research.

Apolipoprotein A-II Modulates the Binding and Selective Lipid Uptake of Reconstituted High Density Lipoprotein by Scavenger Receptor BI*

A. Jonas, N. Webb, F. D. de Beer, 2001, The Journal of Biological Chemistry.

Overexpression of SR-BI by adenoviral vector promotes clearance of apoA-I, but not apoB, in human apoB transgenic mice DOI 10.1194/jlr.M200026-JLR200

A. Daugherty, M. Kindy, N. Webb, 2002, Journal of Lipid Research.

Comparative analysis of lipid composition of normal and acute-phase high density lipoproteins.

W. Pruzanski, A. Kuksis, A. Ravandi, 2000, Journal of lipid research.

ApoB-containing lipoproteins in apoE-deficient mice are not metabolized by the class B scavenger receptor BI Published, JLR Papers in Press, October 16, 2003. DOI 10.1194/jlr.M300319-JLR200

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2004, Journal of Lipid Research.

Neutrophil association and degradation of normal and acute-phase high-density lipoprotein 3.

G. Coetzee, F. D. de Beer, D. R. van der Westhuyzen, 1987, The Biochemical journal.

Scavenger receptor SR-BI in macrophage lipid metabolism.

Jason M. Meyer, N. Webb, D. R. van der Westhuyzen, 2011, Atherosclerosis.

Structure, organization, and chromosomal mapping of the gene encoding macrosialin, a macrophage-restricted protein.

F. D. de Beer, W. D. de Villiers, D. R. van der Westhuyzen, 1998, Genomics.

Scavenger Receptor BI Mediates the Selective Uptake of Oxidized Cholesterol Esters by Rat Liver*

P. Connell, T. V. van Berkel, K. Fluiter, 1999, The Journal of Biological Chemistry.

SR-BI Selective Lipid Uptake: Subsequent Metabolism of Acute Phase HDL

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2009, Arteriosclerosis, thrombosis, and vascular biology.

HDL modification by secretory phospholipase A(2) promotes scavenger receptor class B type I interaction and accelerates HDL catabolism.

P. Connell, N. Webb, F. D. de Beer, 2000, Journal of lipid research.

Characterization of constitutive human serum amyloid A protein (SAA4) as an apolipoprotein.

M. Kindy, F. D. de Beer, B. Asztalos, 1995, Journal of Lipid Research.

ATP binding cassette G1-dependent cholesterol efflux during inflammation1

A. Vaughan, N. Webb, F. D. de Beer, 2011, Journal of Lipid Research.

Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI[S]

N. Webb, D. R. van der Westhuyzen, A. Ji, 2012, Journal of Lipid Research.

The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

Jason M. Meyer, N. Webb, F. D. de Beer, 2013, Journal of lipids.

Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake*

F. D. de Beer, D. R. van der Westhuyzen, L. Cai, 2005, Journal of Biological Chemistry.

HDL Remodeling During the Acute Phase Response

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Impact of serum amyloid A on high density lipoprotein composition and levels

D. Rateri, N. Webb, F. D. de Beer, 2010, Journal of Lipid Research.

Serum amyloid A protein enhances the activity of secretory non-pancreatic phospholipase A2.

P. Vadas, W. Pruzanski, F. D. de Beer, 1995, The Biochemical journal.

Mouse serum amyloid A protein (SAA5) structure and expression.

William Arbuthnot Sir Lane, M. Kindy, F. D. de Beer, 1994, Journal of Biological Chemistry.

Identification of a novel serum amyloid A protein in BALB/c mice.

S. Shedlofsky, F. D. de Beer, M. de Beer, 1991, The Biochemical journal.

Human serum amyloid A protein. The assignment of the six major isoforms to three published gene sequences and evidence for two genetic loci.

G. Coetzee, F. D. de Beer, A. Strachan, 1989, The Journal of biological chemistry.

Identification of novel members of the serum amyloid A protein superfamily as constitutive apolipoproteins of high density lipoprotein.

William Arbuthnot Sir Lane, J. Lélias, A. Whitehead, 1992, The Journal of biological chemistry.

Secretory non-pancreatic phospholipase A2: influence on lipoprotein metabolism.

A. Lusis, L. Castellani, F. D. de Beer, 1997, Journal of lipid research.

Lipoproteins are substrates for human secretory group IIA phospholipase A2: preferential hydrolysis of acute phase HDL.

P. Vadas, W. Pruzanski, A. Kuksis, 1998, Journal of lipid research.

Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I*

F. D. de Beer, D. R. van der Westhuyzen, L. Cai, 2005, Journal of Biological Chemistry.

Low-Density Lipoprotein From Apolipoprotein E–Deficient Mice Induces Macrophage Lipid Accumulation in a CD36 and Scavenger Receptor Class A–Dependent Manner

F. D. de Beer, W. D. de Villiers, D. R. van der Westhuyzen, 2004, Arteriosclerosis, thrombosis, and vascular biology.

Lack of a direct role for macrosialin in oxidized LDL metabolism Published, JLR Papers in Press, January 16, 2003. DOI 10.1194/jlr.M200444-JLR200

N. Webb, W. D. de Villiers, D. R. van der Westhuyzen, 2003, Journal of Lipid Research.

Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE−/− Mice From Angiotensin II–Induced Abdominal Aortic Aneurysm Formation

R. Charnigo, A. Daugherty, D. Rateri, 2015, Arteriosclerosis, thrombosis, and vascular biology.

Deficiency of Endogenous Acute Phase Serum Amyloid A Does Not Affect Atherosclerotic Lesions in Apolipoprotein E–Deficient Mice

A. Daugherty, D. Rateri, Deborah A. Howatt, 2014, Arteriosclerosis, thrombosis, and vascular biology.

Expression of mouse acute-phase (SAA1.1) and constitutive (SAA4) serum amyloid A isotypes: influence on lipoprotein profiles.

M. Kindy, F. D. de Beer, J. Yu, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

N. Webb, D. Jennings, A. Ji, 2020, Journal of Lipid Research.

SR-BI-mediated selective lipid uptake segregates apoA-I and apoA-II catabolism Published, JLR Papers in Press, August 1, 2005. DOI 10.1194/jlr.M500068-JLR200

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2005, Journal of Lipid Research.

Remodeling of HDL remnants generated by scavenger receptor class B type I Published, JLR Papers in Press, June 21, 2004. DOI 10.1194/jlr.M400026-JLR200

N. Webb, F. D. de Beer, B. Asztalos, 2004, Journal of Lipid Research.

Adenoviral vector-mediated overexpression of serum amyloid A in apoA-I-deficient mice.

M. Kindy, N. Webb, F. D. de Beer, 1997, Journal of lipid research.

Expression of recombinant human serum amyloid A in mammalian cells and demonstration of the region necessary for high-density lipoprotein binding and amyloid fibril formation by site-directed mutagenesis.

H Patel, H. Patel, J. Bramall, 1996, The Biochemical journal.

Serum Amyloid A Is an Exchangeable Apolipoprotein

P. McNamara, P. Shridas, N. Webb, 2018, Arteriosclerosis, thrombosis, and vascular biology.

Serum Amyloid A Facilitates Early Lesion Development in Ldlr−/− Mice

G. Getz, C. Reardon, J. Lukens, 2015, Journal of the American Heart Association.

Serum amyloid A protein family. Differential induction by oxidized lipids in mouse strains.

A. Lusis, J. Berliner, M. Kindy, 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.

Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

R. Charnigo, N. Webb, F. D. de Beer, 2016, Journal of Lipid Research.

Association of apolipoprotein E with murine amyloid A protein amyloid.

M. Kindy, F. D. de Beer, M. de Beer, 1995, Laboratory investigation; a journal of technical methods and pathology.

Apolipoprotein A-I conformation markedly influences HDL interaction with scavenger receptor BI.

A. Jonas, F. D. de Beer, D. R. van der Westhuyzen, 2001, Journal of lipid research.

Serum Amyloid A Contributes to Chronic Apical Periodontitis via TLR2 and TLR4

H. Sasaki, N. Kawashima, T. V. Van Dyke, 2018, Journal of dental research.

CD36 does not play a direct role in HDL or LDL metabolism.

N. Webb, F. D. de Beer, W. D. de Villiers, 2001, Journal of lipid research.

Syrian and Armenian hamsters differ in serum amyloid A gene expression. Identification of novel Syrian hamster serum amyloid A subtypes.

J. Sipe, F. D. de Beer, I. Carreras, 1993, Journal of immunology.

Structure of the mouse Saa4 gene and its linkage to the serum amyloid A gene family.

M. Kindy, M. Goodson, N. Webb, 1996, Genomics.

Human serum amyloid A protein. Complete amino acid sequence of a new variant.

J. Sipe, F. D. de Beer, M. de Beer, 1992, The Biochemical journal.