Zi‐Qiang Zhou

发表

A critical role for Mnt in Myc-driven T-cell proliferation and oncogenesis

Colin J. Daniel, R. Sears, P. Hurlin, 2012, Proceedings of the National Academy of Sciences.

Cloning and characterization of the promoter of baboonXRCC1, a gene involved in DNA strand-break repair

C. Walter, Zi‐Qiang Zhou, 1998, Somatic cell and molecular genetics.

Loss of the Max-interacting protein Mnt in mice results in decreased viability, defective embryonic growth and craniofacial defects: relevance to Miller-Dieker syndrome.

R. Eisenman, M. Rosenfeld, A. Wynshaw-Boris, 2004, Human molecular genetics.

Deletion of Mnt leads to disrupted cell cycle control and tumorigenesis

A. Wynshaw-Boris, P. Hurlin, S. Ota, 2003, The EMBO journal.

Activities of N-Myc in the developing limb link control of skeletal size with digit separation

P. Hurlin, D. Keene, S. Ota, 2007, Development.

Mnt–Max to Myc–Max complex switching regulates cell cycle entry

A. Wynshaw-Boris, P. Hurlin, S. Ota, 2005, The Journal of cell biology.

Disruption of a Sox9-β-catenin circuit by mutant Fgfr3 in thanatophoric dysplasia type II.

P. Hurlin, D. Keene, S. Ota, 2012, Human molecular genetics.

The role of senescence and prosurvival signaling in controlling the oncogenic activity of FGFR2 mutants associated with cancer and birth defects

P. Hurlin, S. Ota, Jason M. Link, 2009, Human molecular genetics.

Sequential and Coordinated Actions of c-Myc and N-Myc Control Appendicular Skeletal Development

H. Akiyama, P. Hurlin, D. Keene, 2011, PloS one.

Health Span and Life Span in Transgenic Mice with Modulated DNA Repair

C. McMahan, M. Hanes, D. Herbert, 2001, Annals of the New York Academy of Sciences.

Suppression of FGFR3- and MYC-dependent oncogenesis by tubacin: association with HDAC6-dependent and independent activities

P. Hurlin, S. Ota, Zi-Qiang Zhou, 2017, Oncotarget.

miR-637 Prevents Glioblastoma Progression by Interrupting ZEB2/WNT/β-catenin Cascades

Di Wu, W. Wang, Shuai Han, 2021, Cellular and Molecular Neurobiology.

Testis and somaticXrcc-1 DNA repair gene expression

L. Thompson, Zi-Qiang Zhou, J. McCarrey, 1994, Somatic cell and molecular genetics.

Mutant activated FGFR3 impairs endochondral bone growth by preventing SOX9 downregulation in differentiating chondrocytes.

H. Akiyama, P. Hurlin, C. Deng, 2015, Human molecular genetics.

Spontaneous hepatocellular carcinoma is reduced in transgenic mice overexpressing human O6- methylguanine-DNA methyltransferase

M. Hanes, D. Herbert, Kristen Kewitt, 2001, Proceedings of the National Academy of Sciences of the United States of America.

The human XRCC 9 gene corrects chromosomal instability and mutagen sensitivities in CHO UV 40 cells

J. Albala, J. Lamerdin, L. Thompson, 1997 .

HDAC6 deficiency or inhibition blocks FGFR3 accumulation and improves bone growth in a model of achondroplasia.

P. Hurlin, S. Ota, Zi-Qiang Zhou, 2016, Human molecular genetics.

The human XRCC9 gene corrects chromosomal instability and mutagen sensitivities in CHO UV40 cells.

J. Albala, J. Lamerdin, L. Thompson, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Preparation and characterization of a novel injectable strontium-containing calcium phosphate cement with collagen.

Zi-Qiang Zhou, Dongping Ye, Bin Wang, 2015, Chinese journal of traumatology = Zhonghua chuang shang za zhi.

ERCC4 (XPF) encodes a human nucleotide excision repair protein with eukaryotic recombination homologs

J. Lamerdin, K. Brookman, L. Thompson, 1996, Molecular and cellular biology.

XRCC2 and XRCC3, new human Rad51-family members, promote chromosome stability and protect against DNA cross-links and other damages.

J. Lamerdin, D. Schild, R. Tebbs, 1998, Molecular cell.