M. McCallister

Prenatal polycyclic aromatic hydrocarbon exposure leads to behavioral deficits and downregulation of receptor tyrosine kinase, MET.

Marcus C. Ferguson, P. Levitt, M. Aschner, 2010, Toxicological sciences : an official journal of the Society of Toxicology.

Prenatal exposure to benzo(a)pyrene impairs later-life cortical neuronal function.

F. Ebner, M. Aschner, D. Hood, 2008, Neurotoxicology.

Polycyclic aromatic hydrocarbons: implications for developmental, molecular, and behavioral neurotoxicity

R. Savage, Raina Rhoades, D. Hood, 2015 .

Down-regulation of early ionotrophic glutamate receptor subunit developmental expression as a mechanism for observed plasticity deficits following gestational exposure to benzo(a)pyrene.

M. Aschner, D. Hood, A. Ramesh, 2007, Neurotoxicology.

Revealing Behavioral Learning Deficit Phenotypes Subsequent to In Utero Exposure to Benzo(a)pyrene.

M. C. Newland, D. Hood, A. Ramesh, 2016, Toxicological sciences : an official journal of the Society of Toxicology.

PAH particles perturb prenatal processes and phenotypes: protection from deficits in object discrimination afforded by dampening of brain oxidoreductase following in utero exposure to inhaled benzo(a)pyrene.

M. Aschner, S. Safe, Raina Rhoades, 2012, Toxicological sciences : an official journal of the Society of Toxicology.

Polycyclic aromatic hydrocarbons: implications for developmental, molecular, and behavioral neurotoxicity

R. Savage, Raina Rhoades, D. Hood, 2020, Handbook of Toxicology of Chemical Warfare Agents.