F. Parisi

发表

Transformed epithelia trigger non-tissue-autonomous tumor suppressor response by adipocytes via activation of Toll and Eiger/TNF signaling.

Yachuan Yu, M. Vidal, K. Strathdee, 2014, Cell reports.

Epithelial delamination and migration

M. Vidal, F. Parisi, Federica Parisi, 2011, Cell adhesion & migration.

dMyc expression in the fat body affects DILP2 release and increases the expression of the fat desaturase Desat1 resulting in organismal growth.

D. Grifoni, L. Brown, Sheri Zola, 2013, Developmental biology.

Drosophila insulin and target of rapamycin (TOR) pathways regulate GSK3 beta activity to control Myc stability and determine Myc expression in vivo

E. Tabak, Nandini Kundu, A. Pession, 2011, BMC Biology.

Identification of Domains Responsible for Ubiquitin-Dependent Degradation of dMyc by Glycogen Synthase Kinase 3β and Casein Kinase 1 Kinases

P. Pelicci, F. Serras, D. Grifoni, 2009, Molecular and Cellular Biology.

Drosophila Lethal Giant Larvae Neoplastic Mutant as a Genetic Tool for Cancer Modeling

G. Tomba, A. Pession, D. Grifoni, 2008, Current genomics.

aPKCζ cortical loading is associated with Lgl cytoplasmic release and tumor growth in Drosophila and human epithelia

G. Collina, A. Pession, D. Grifoni, 2007, Oncogene.

The Stearoyl-CoA Desaturase-1 (Desat1) in Drosophila cooperated with Myc to Induce Autophagy and Growth, a Potential New Link to Tumor Survival

A. Vingiani, M. Pasini, Sheri Zola, 2017, Genes.

Inositol phosphates induce DAPI fluorescence shift.

A. Saiardi, B. Kolozsvári, F. Parisi, 2014, The Biochemical journal.