Clara Zettelmeyer

Targeting natural splicing plasticity of APOBEC3B restricts its expression and mutagenic activity

L. Prokunina-Olsson, V. Pathak, L. Dyrskjøt, 2021, Communications biology.

Isoform-specific characterization implicates alternative splicing in APOBEC3B as a mechanism restricting APOBEC-mediated mutagenesis

L. Prokunina-Olsson, V. Pathak, L. Dyrskjøt, 2020, bioRxiv.

APOBEC3B expression in breast cancer cell lines and tumors depends on the estrogen receptor status.

L. Prokunina-Olsson, A. R. Banday, Krizia-Ivana Udquim, 2020, Carcinogenesis.