Talia Adi

Resilience to Pain: A Peripheral Component Identified Using Induced Pluripotent Stem Cells and Dynamic Clamp

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2018, The Journal of Neuroscience.

Atypical changes in DRG neuron excitability and complex pain phenotype associated with a Nav1.7 mutation that massively hyperpolarizes activation

S. Dib-Hajj, S. Waxman, S. Walker, 2018, Scientific Reports.

Reverse pharmacogenomics: carbamazepine normalizes activation and attenuates thermal hyperexcitability of sensory neurons due to Nav1.7 mutation I234T

S. Dib-Hajj, S. Waxman, Yang Yang, 2018, British journal of pharmacology.

A novel gain-of-function Nav1.7 mutation in a carbamazepine-responsive patient with adult-onset painful peripheral neuropathy

S. Dib-Hajj, S. Waxman, M. Estacion, 2018, Molecular pain.

Building sensory axons: Delivery and distribution of NaV1.7 channels and effects of inflammatory mediators

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2019, Science Advances.