M. O. Nordkamp

Interactions of host defense and hyper-keratinization in psoriasis

Runyue Huang, W. Tao, Chuanjian Lu, 2021, bioRxiv.

Multi‐omics integration reveals a core network involved in host defence and hyperkeratinization in psoriasis

W. Tao, Jingwen Deng, Chuanjiang Lu, 2022, Clinical and translational medicine.

ZFP36 family members regulate the pro-inflammatory features of psoriatic dermal fibroblasts.

T. Radstake, B. Giovannone, F. van Wijk, 2021, The Journal of investigative dermatology.

Impaired proteolysis by SPPL2a causes CD74 fragment accumulation that can be recognized by anti‐CD74 autoantibodies in human ankylosing spondylitis

M. Boes, T. Radstake, R. Lebbink, 2019, European journal of immunology.

Suppression of IL‐12/IL‐23 p40 subunit in the skin and blood of psoriasis patients by Tofacitinib is dependent on active interferon‐γ signaling in dendritic cells: Implications for the treatment of psoriasis and interferon‐driven diseases

P. Welsing, E. Leijten, S. Silva‐Cardoso, 2022, Experimental dermatology.

Prophylactic vaccines are potent activators of monocyte-derived dendritic cells and drive effective anti-tumor responses in melanoma patients at the cost of toxicity

W. Oyen, C. Figdor, C. Punt, 2016, Cancer Immunology, Immunotherapy.

Multi-omics approach identifies PI3 as a biomarker for disease severity and hyper-keratinization in psoriasis.

J. Borghans, W. Tao, Chuanjian Lu, 2023, Journal of dermatological science.