T. Seck

Two mutations in gB-1 and gD-1 of herpes simplex virus type 1 are involved in the “fusion from without’ phenotype in different cell types

D. Falke, M. Lingen, K. Weise, 2004, Virus Genes.

Single amino acid substitutions in the glycoprotein B carboxy terminus influence the fusion from without property of herpes simplex virus type 1.

D. Falke, M. Lingen, K. Weise, 1995, The Journal of general virology.

Evidence for a multistep mechanism for cell-cell fusion by herpes simplex virus with mutations in the syn 3 locus using heparin derivatives during fusion from within

D. Falke, M. Lingen, K. Weise, 2005, Archives of Virology.

Binding of Filamin to the C-terminal Tail of the Calcitonin Receptor Controls Recycling*

R. Baron, W. Horne, T. Seck, 2003, The Journal of Biological Chemistry.

The Alternatively Spliced Δe13 Transcript of the Rabbit Calcitonin Receptor Dimerizes with the C1a Isoform and Inhibits Its Surface Expression*

Roland Baron, R. Baron, W. Horne, 2003, Journal of Biological Chemistry.

The delta e13 isoform of the calcitonin receptor forms a six-transmembrane domain receptor with dominant-negative effects on receptor surface expression and signaling.

R. Baron, W. Horne, A. Florea, 2005, Molecular endocrinology.