Alicia S. Gates

发表

Neuregulin-1 inhibits neuroinflammatory responses in a rat model of organophosphate-nerve agent-induced delayed neuronal injury

Pamela J Lein, B. Ford, P. Lein, 2015, Journal of Neuroinflammation.

Expression Analysis Systematic Explorer (EASE) analysis reveals differential gene expression in permanent and transient focal stroke rat models

Byron D. Ford, B. Ford, Gregory D. Ford, 2006, Brain Research.

Agonist mediated internalization of M2 mAChR is β-arrestin-dependent

Alicia S Gates, Maria C Echeverry, V. Mosser, 2006, Journal of molecular signaling.

Neuroprotection by neuregulin-1 following focal stroke is associated with the attenuation of ischemia-induced pro-inflammatory and stress gene expression

B. Ford, Gregory D. Ford, Alicia S Gates, 2005, Neurobiology of Disease.

Gene expression patterns following unilateral traumatic brain injury reveals a local pro-inflammatory and remote anti-inflammatory response

M. LaPlaca, B. Ford, Gregory D. Ford, 2013, BMC Genomics.

Transient hypoxia induces sequestration of M1 and M2 muscarinic acetylcholine receptors

A. Tobin, Alicia S Gates, V. Mosser, 2006, Journal of neurochemistry.

Acute neuronal injury and blood genomic profiles in a nonhuman primate model for ischemic stroke.

B. Ford, E. Kraiselburd, P. Ferchmin, 2012, Comparative medicine.

Bilateral gene interaction hierarchy analysis of the cell death gene response emphasizes the significance of cell cycle genes following unilateral traumatic brain injury

M. LaPlaca, B. Ford, Gregory D. Ford, 2016, BMC Genomics.

Analysis of transcriptional profiles and functional clustering of global cerebellar gene expression in PCD3J mice.

P. MacLeish, B. Ford, D. Hood, 2008, Biochemical and biophysical research communications.

Neuroprotective effects of neuregulin-1 on B35 neuronal cells following ischemia

B. Ford, Gregory D. Ford, Alicia S Gates, 2008, Brain Research.

Corrigendum to “Neuroprotection by neuregulin-1 following focal stroke is associated with the attenuation of ischemia-induced pro-inflammatory and stress gene expression” [Neurobiol. Dis. 19 (2005) 461–470]

B. Ford, Gregory D. Ford, Ju Jiang, 2008, Neurobiology of Disease.