Hans Michael Piper

Transcription activator protein 1 (AP‐1) mediates NO‐induced apoptosis of adult cardiomyocytes

H. Piper, A. Rakow, G. Taimor, 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Angiotensin II‐dependent loss of cardiac function: Mechanisms and pharmacological targets attenuating this effect

K. Schlüter, H. Piper, G. Euler, 2008, Journal of cellular physiology.

Hypertrophic responsiveness of cardiomyocytes to α‐ or β‐adrenoceptor stimulation requires sodium‐proton‐exchanger‐1 (NHE‐1) activation but not cellular alkalization

C. Schäfer, K. Schlüter, H. Piper, 2002 .

Influence of pH iand Creatine Phosphate on a-Adrenoceptor-mediated Cardiac Hypertrophy

K. Schlüter, M. Schäfer, G. Taimor, 1998 .

p38 MAP‐kinase in cultured adult rat ventricular cardiomyocytes: expression and involvement in hypertrophic signalling

K. Schlüter, H. Piper, S. Wenzel, 2005, European journal of heart failure.

A N‐terminal PTHrP peptide fragment void of a PTH/PTHrP‐receptor binding domain activates cardiac ETA receptors

K. Schlüter, H. Piper, C. Katzer, 2001, British journal of pharmacology.

Temporary contractile blockade prevents hypercontracture in anoxic-reoxygenated cardiomyocytes.

B. Siegmund, P. Schwartz, H. Piper, 1990, The American journal of physiology.