J. Soria

发表

Fibrinogen Bastia (γ 318 Asp → Tyr) a Novel Abnormal Fibrinogen Characterized by Defective Fibrin Polymerization

K. Lounes, J. Koopman, C. Soria, 1999, Thrombosis and Haemostasis.

Hemostasis in Patients Undergoing Extracorporeal Circulation: The Effect of Aprotinin (Trasylol)

J. Soria, C. Soria, He Lu, 1991, Thrombosis and Haemostasis.

Abnormal Fibrin Clot Architecture in Nephrotic Patients Is Related to Hypofibrinolysis: Influence of Plasma Biochemical Modifications

J. Soria, C. Soria, J. Collet, 1999, Thrombosis and Haemostasis.

Cerivastatin, an Inhibitor of HMG-CoA Reductase, Inhibits Urokinase/Urokinase-receptor Expression and MMP-9 Secretion by Peripheral Blood Monocytes

Z Mishal, J. Beaudeux, J Soria, 2000, Thrombosis and Haemostasis.

Postoperative Hemostasis and Fibrinolysis in Patients Undergoing Cardiopulmonary Bypass with or without Aprotinin Therapy

J. Soria, C. Soria, He Lu, 1994, Thrombosis and Haemostasis.

Role of Active Center and Lysine Binding Sites of Plasmin in Plasmin-Induced Platelet Activation and Disaggregation

Hong Li, J. Soria, C. Soria, 1991, Thrombosis and Haemostasis.

Increased Expression of u-PA and u-PAR on Monocytes by LDL and Lp(a) Lipoproteins – Consequences for Plasmin Generation and Monocyte Adhesion

J. Soria, C. Soria, J. Collet, 1999, Thrombosis and Haemostasis.

The plasminogen‐activation system in ovarian tumors

J. Soria, C. Soria, He Lu, 1993, International journal of cancer.

Malignant hematopoietic cells induce an increased expression of VEGFR-1 and VEGFR-3 on bone marrow endothelial cells via AKT and mTOR signalling pathways.

J. Soria, C. Soria, A. Faussat, 2006, Biochemical and biophysical research communications.

SDF-1 activity on microvascular endothelial cells: consequences on angiogenesis in in vitro and in vivo models.

J. Soria, C. Soria, Hong Li, 2000, Thrombosis research.

Anti-RhoA and anti-RhoC siRNAs inhibit the proliferation and invasiveness of MDA-MB-231 breast cancer cells in vitro and in vivo.

P. Opolon, Hong Li, J. Soria, 2005, Molecular therapy : the journal of the American Society of Gene Therapy.

Cerivastatin, an inhibitor of HMG-CoA reductase, inhibits the signaling pathways involved in the invasiveness and metastatic properties of highly invasive breast cancer cell lines: an in vitro study.

J. Soria, C. Soria, M. Körner, 2001, Carcinogenesis.

Recombinant hirudin: a specific thrombin inhibiting anticoagulant for hemodialysis.

J. Soria, C. Soria, R. Vanholder, 1994, Kidney international.

New insights into the actions of bisphosphonate zoledronic acid in breast cancer cells by dual RhoA-dependent and -independent effects

J. Soria, C. Soria, J. Vannier, 2003, British Journal of Cancer.

Successful Progressive Anticoagulation in a Severe Protein C Deficiency and Previous Skin Necrosis at the Initiation of Oral Anticoagulant Treatment

J. Soria, J. Conard, M. Samama, 1984, Thrombosis and Haemostasis.

Molecular basis for fibrinogen Dusart (A alpha 554 Arg-->Cys) and its association with abnormal fibrin polymerization and thrombophilia.

J. Soria, C. Legrand, C. Soria, 1993, The Journal of clinical investigation.

A Pilot Study of Subcutaneous Recombinant Hirudin (HBW 023) in the Treatment of Deep Vein Thrombosis

J. Soria, C. Soria, F. Schiele, 1994, Thrombosis and Haemostasis.

Adenovirus-mediated delivery of a uPA/uPAR antagonist suppresses angiogenesis-dependent tumor growth and dissemination in mice

P. Opolon, J. Soria, C. Soria, 1998, Gene Therapy.

Intravenous delivery of anti-RhoA small interfering RNA loaded in nanoparticles of chitosan in mice: safety and efficacy in xenografted aggressive breast cancer.

P. Opolon, J. Soria, C. Soria, 2006, Human gene therapy.

Combined effects of radiotherapy and angiostatin gene therapy in glioma tumor model.

P. Opolon, J. Soria, C. Soria, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Dysfibrinogenemia (Fibrinogen Dusard) Associated with Impaired Fibrin-Enhanced Plasminogen Activation

J. Soria, C. Soria, D. Collen, 1984, Thrombosis and Haemostasis.

Angiostatin gene transfer: inhibition of tumor growth in vivo by blockage of endothelial cell proliferation associated with a mitosis arrest.

P. Opolon, Hong Li, J. Soria, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Disaggregation of In Vitro Preformed Platelet-Rich Clots by Abciximab Increases Fibrin Exposure and Promotes Fibrinolysis

J. Soria, C. Soria, G. Montalescot, 2001, Arteriosclerosis, thrombosis, and vascular biology.