Christopher J. Carbone

发表

Eliminative Signaling by Janus Kinases: Role in the Downregulation of Associated Receptors

S. Fuchs, C. Carbone, Serge Y. Fuchs, 2014, Journal of cellular biochemistry.

Inactivation of Interferon Receptor Promotes the Establishment of Immune Privileged Tumor Microenvironment.

Amy R. Peck, H. Rui, E. Puré, 2017, Cancer cell.

Triggering ubiquitination of IFNAR1 protects tissues from inflammatory injury

H. Rui, A. Rustgi, Angela K. Brice, 2014, EMBO molecular medicine.

Vascular endothelial growth factor-induced elimination of the type 1 interferon receptor is required for efficient angiogenesis.

D. Baker, S. Fuchs, N. A. Leu, 2011, Blood.

Site-specific ubiquitination exposes a linear motif to promote interferon-α receptor endocytosis

G. Lukács, D. Baker, S. Fuchs, 2007, The Journal of cell biology.

Prolactin stimulates ubiquitination, initial internalization, and degradation of its receptor via catalytic activation of Janus kinase 2.

S. Fuchs, K. G. S. Kumar, S. Frank, 2008, The Journal of endocrinology.

Pathogen Recognition Receptor Signaling Accelerates Phosphorylation-Dependent Degradation of IFNAR1

D. Baker, S. Fuchs, N. A. Leu, 2011, PLoS pathogens.

Eliminative Signaling by Janus Kinases: Role in the Downregulation of Associated Receptors

Serge Y. Fuchs, Christopher J. Carbone, 2014, Journal of cellular biochemistry.

Site-specific ubiquitination exposes a linear motif to promote interferon-α receptor endocytosis

G. Lukács, D. Baker, Junmin Peng, 2007, The Journal of cell biology.