Lourdes Ruiz-Valdepeñas

β−Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR‐α, PPAR‐γ and TRPV1, but not CB1 or CB2 receptors

B. Cravatt, J. Martínez-Orgado, J. Romero, 2012, British journal of pharmacology.

Cannabidiol reduces lipopolysaccharide-induced vascular changes and inflammation in the mouse brain: an intravital microscopy study

J. Martínez-Orgado, J. Romero, C. Benito, 2011, Journal of Neuroinflammation.

The endocannabinoid system and amyloid-related diseases

J. Romero, C. Benito, R. Tolón, 2010, Experimental Neurology.

Endocannabinoid regulation of amyloid-induced neuroinflammation

B. Cravatt, M. T. Grande, C. Hillard, 2015, Neurobiology of Aging.

β−Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR‐α, PPAR‐γ and TRPV1, but not CB1 or CB2 receptors

B. Cravatt, J. Martínez-Orgado, J. Romero, 2012, British journal of pharmacology.