Francisco Javier Fernández-Sánchez

β−Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR‐α, PPAR‐γ and TRPV1, but not CB1 or CB2 receptors

B. Cravatt, J. Martínez-Orgado, J. Romero, 2012, British journal of pharmacology.

Endocannabinoid regulation of amyloid-induced neuroinflammation

B. Cravatt, M. T. Grande, C. Hillard, 2015, Neurobiology of Aging.

β−Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR‐α, PPAR‐γ and TRPV1, but not CB1 or CB2 receptors

B. Cravatt, J. Martínez-Orgado, J. Romero, 2012, British journal of pharmacology.