Jennie W. Taylor

Clusterin promotes amyloid plaque formation and is critical for neuritic toxicity in a mouse model of Alzheimer's disease

Bruce J Aronow, David M Holtzman, D. Holtzman, 2002, Proceedings of the National Academy of Sciences of the United States of America.

ApoAI deficiency results in marked reductions in plasma cholesterol but no alterations in amyloid-beta pathology in a mouse model of Alzheimer's disease-like cerebral amyloidosis.

A. Fagan, D. Holtzman, S. Paul, 2004, The American journal of pathology.

ApoE and Clusterin Cooperatively Suppress Aβ Levels and Deposition Evidence that ApoE Regulates Extracellular Aβ Metabolism In Vivo

Bruce J Aronow, D. Holtzman, S. Paul, 2004, Neuron.

In Vivo Assessment of Brain Interstitial Fluid with Microdialysis Reveals Plaque-Associated Changes in Amyloid-β Metabolism and Half-Life

D. Holtzman, S. Paul, J. Cirrito, 2003, The Journal of Neuroscience.

Apolipoprotein E Markedly Facilitates Age-Dependent Cerebral Amyloid Angiopathy and Spontaneous Hemorrhage in Amyloid Precursor Protein Transgenic Mice

D. Holtzman, S. Paul, K. Bales, 2003, The Journal of Neuroscience.

Evidence for peripheral clearance of cerebral Aβ protein following chronic, active Aβ immunization in PSAPP mice

D. Holtzman, D. Selkoe, C. Lemere, 2003, Neurobiology of Disease.

Evidence for peripheral clearance of cerebral Abeta protein following chronic, active Abeta immunization in PSAPP mice.

D. Holtzman, D. Selkoe, C. Lemere, 2003, Neurobiology of disease.