W. Sun

AML-2 is a potential target for transcriptional regulation by the t(8;21) and t(12;21) fusion proteins in acute leukemia.

N. Lenny, S. Hiebert, Weili Sun, 1996, Oncogene.

The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription

T. Golub, J. Downing, S. Shurtleff, 1996, Molecular and cellular biology.

Induction and regulation of the carcinogen-metabolizing enzyme CYP1A1 by marijuana smoke and delta (9)-tetrahydrocannabinol.

M. Roth, O. Hankinson, D. Tashkin, 2001, American journal of respiratory cell and molecular biology.

An uncommon phenotype of poor inducibility of CYP1A1 in human lung is not ascribable to polymorphisms in the AHR, ARNT, or CYP1A1 genes.

O. Hankinson, S. Anttila, H. Vainio, 2000, Pharmacogenetics.

Activation of transcription factors of nuclear factor kappa B, activator protein-1 and octamer factors in hyperalgesia.

J. Lin, S. Lin-Shiau, W. Sun, 2000, European journal of pharmacology.

Identification of novel dioxin-responsive genes by representational difference analysis

O. Hankinson, S. Saarikoski, W. Sun, 2007, Xenobiotica; the fate of foreign compounds in biological systems.

Identification of a nuclear matrix targeting signal in the leukemia and bone-related AML/CBF-alpha transcription factors.

J. Lawrence, G. Stein, J. Lian, 1997, Proceedings of the National Academy of Sciences of the United States of America.