X. Jiang

发表

Increased prebeta-high density lipoprotein, apolipoprotein AI, and phospholipid in mice expressing the human phospholipid transfer protein and human apolipoprotein AI transgenes.

A. Tall, J. Breslow, A. Walsh, 1996, The Journal of clinical investigation.

Probucol enhances selective uptake of HDL-associated cholesteryl esters in vitro by a scavenger receptor B-I-dependent mechanism.

A. Tall, X. Jiang, R. Ramakrishnan, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Increased sphingomyelin content of plasma lipoproteins in apolipoprotein E knockout mice reflects combined production and catabolic defects and enhances reactivity with mammalian sphingomyelinase.

A. Tall, H. Pownall, X. Jiang, 1998, The Journal of clinical investigation.

IFN-gamma potentiates atherosclerosis in ApoE knock-out mice.

A. Tall, C. Schindler, S. Gupta, 1997, The Journal of clinical investigation.

Decreased early atherosclerotic lesions in hypertriglyceridemic mice expressing cholesteryl ester transfer protein transgene.

E. Rubin, A. Tall, J. Breslow, 1995, The Journal of clinical investigation.

Genetic cholesteryl ester transfer protein deficiency caused by two prevalent mutations as a major determinant of increased levels of high density lipoprotein cholesterol.

H. Mabuchi, X. Jiang, Akihiro Inazu, 1994, The Journal of clinical investigation.

A missense mutation in the cholesteryl ester transfer protein gene with possible dominant effects on plasma high density lipoproteins.

S. Yamashita, P. Kussie, X. Jiang, 1993, The Journal of clinical investigation.

Decreased cholesteryl ester transfer protein (CETP) mRNA and protein and increased high density lipoprotein following lipopolysaccharide administration in human CETP transgenic mice.

A. Tall, J. Breslow, A. Walsh, 1995, The Journal of clinical investigation.

Dietary cholesterol increases transcription of the human cholesteryl ester transfer protein gene in transgenic mice. Dependence on natural flanking sequences.

A. Tall, J. Breslow, A. Walsh, 1992, The Journal of clinical investigation.

Targeted mutation of plasma phospholipid transfer protein gene markedly reduces high-density lipoprotein levels.

A. Tall, X. Jiang, M. Lin, 1999, The Journal of clinical investigation.

1999 George Lyman Duff memorial lecture: lipid transfer proteins, HDL metabolism, and atherogenesis.

A. Tall, Y. Luo, X. Jiang, 2000, Arteriosclerosis, thrombosis, and vascular biology.

L-arginine prevents xanthoma development and inhibits atherosclerosis in LDL receptor knockout mice.

R. Michler, P. Cannon, X. Jiang, 1997, Circulation.

Remodeling of HDL by CETP in vivo and by CETP and hepatic lipase in vitro results in enhanced uptake of HDL CE by cells expressing scavenger receptor B-I.

A. Tall, X. Jiang, H. Oliveira, 1999, Journal of lipid research.

Profound induction of hepatic cholesteryl ester transfer protein transgene expression in apolipoprotein E and low density lipoprotein receptor gene knockout mice. A novel mechanism signals changes in plasma cholesterol levels.

A. Tall, J. Breslow, A. Walsh, 1996, The Journal of clinical investigation.

Reduced high density lipoprotein cholesterol in human cholesteryl ester transfer protein transgenic mice.

A. Tall, J. Breslow, A. Walsh, 1991, The Journal of biological chemistry.

Mammalian adipose tissue and muscle are major sources of lipid transfer protein mRNA.

A. Tall, I. Goldberg, P. Moulin, 1991, The Journal of biological chemistry.

Structure-function relationships of human cholesteryl ester transfer protein: analysis using monoclonal antibodies.

E. Rassart, A. Tall, P. Kussie, 1996, Journal of lipid research.

Down-regulation of mRNA for the low density lipoprotein receptor in transgenic mice containing the gene for human cholesteryl ester transfer protein. Mechanism to explain accumulation of lipoprotein B particles.

A. Tall, J. Breslow, A. Walsh, 1993, The Journal of biological chemistry.

Phospholipid transfer protein gene knock-out mice have low high density lipoprotein levels, due to hypercatabolism, and accumulate apoA-IV-rich lamellar lipoproteins.

A. Tall, X. Jiang, M. Lin, 2000, Journal of lipid research.

ApoA-II maintains HDL levels in part by inhibition of hepatic lipase. Studies In apoA-II and hepatic lipase double knockout mice.

A. Tall, J. Breslow, X. Jiang, 1999, Journal of lipid research.

Point mutagenesis of positively charged amino acids of cholesteryl ester transfer protein: conserved residues within the lipid transfer/lipopolysaccharide binding protein gene family essential for function.

M. Boguski, A. Tall, X. Jiang, 1995, Biochemistry.

The CCAAT/enhancer-binding protein trans-activates the human cholesteryl ester transfer protein gene promoter.

A. Tall, X. Jiang, L. Agellon, 1992, The Journal of biological chemistry.

Increased high density lipoprotein (HDL), defective hepatic catabolism of ApoA-I and ApoA-II, and decreased ApoA-I mRNA in ob/ob mice. Possible role of leptin in stimulation of HDL turnover.

A. Tall, X. Jiang, D. Silver, 1999, The Journal of biological chemistry.

Genetic cholesteryl ester transfer protein deficiency caused by two prevalent mutations as a major determinant of increased levels of high density lipoprotein cholesterol.

H. Mabuchi, Akihiro Inazu, J. Koizumi, 1994, The Journal of clinical investigation.