X. Guli

Stereotactically Injected Kv1.2 and CASPR2 Antisera Cause Differential Effects on CA1 Synaptic and Cellular Excitability, but Both Enhance the Vulnerability to Pro-epileptic Conditions

R. Köhling, M. Wittstock, A. Storch, 2020, Frontiers in Synaptic Neuroscience.

Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner

R. Köhling, T. Kirschstein, T. Tokay, 2016, Neural plasticity.

Differentially Altered NMDAR Dependent and Independent Long-Term Potentiation in the CA3 Subfield in a Model of Anti-NMDAR Encephalitis

R. Köhling, C. Bien, T. Kirschstein, 2018, Front. Synaptic Neurosci..

Stereotactic injection of cerebrospinal fluid from anti-NMDA receptor encephalitis into rat dentate gyrus impairs NMDA receptor function

R. Köhling, C. Bien, T. Kirschstein, 2016, Brain Research.

Functional Metaplasticity of Hippocampal Schaffer Collateral-CA1 Synapses Is Reversed in Chronically Epileptic Rats

R. Köhling, T. Kirschstein, T. Tokay, 2017, Neural plasticity.

Bidirectional shift of group III metabotropic glutamate receptor-mediated synaptic depression in the epileptic hippocampus

R. Köhling, T. Kirschstein, K. Porath, 2017, Epilepsy Research.

ZD7288 Enhances Long-Term Depression at Early Postnatal Medial Perforant Path-Granule Cell Synapses

R. Köhling, T. Kirschstein, R. Bender, 2012, Neural plasticity.

CK2 Inhibition Prior to Status Epilepticus Persistently Enhances KCa2 Function in CA1 Which Slows Down Disease Progression

Till W. Riffert, R. Köhling, Felix-Michael Schulze, 2020, Frontiers in Cellular Neuroscience.