N. Ale‐Agha

发表

Protective Effects of Curcumin in Cardiovascular Diseases—Impact on Oxidative Stress and Mitochondria

M. Grandoch, J. Altschmied, J. Haendeler, 2022, Cells.

The imbalanced redox status in senescent endothelial cells is due to dysregulated Thioredoxin-1 and NADPH oxidase 4

A. Shah, J. Altschmied, J. Haendeler, 2014, Experimental Gerontology.

Role of HuR and p38MAPK in Ultraviolet B-induced Post-transcriptional Regulation of COX-2 Expression in the Human Keratinocyte Cell Line HaCaT*

Jean Krutmann, Stefanie Galban, L. Klotz, 2009, The Journal of Biological Chemistry.

Extra-Nuclear Functions of the Transcription Factor Grainyhead-Like 3 in the Endothelium—Interaction with Endothelial Nitric Oxide Synthase

J. Altschmied, J. Haendeler, N. Ale-Agha, 2021, Antioxidants.

Aryl Hydrocarbon Receptor-Dependent and -Independent Pathways Mediate Curcumin Anti-Aging Effects

X. Coumoul, C. Esser, N. Ventura, 2022, bioRxiv.

Aryl hydrocarbon receptor-dependent and -independent pathways 1 mediate curcumin anti-aging effects 2

X. Coumoul, C. Esser, N. Ventura, 2022 .

The Aryl Hydrocarbon Receptor (AhR) in the Aging Process: Another Puzzling Role for This Highly Conserved Transcription Factor

N. Ventura, J. Haendeler, N. Ale-Agha, 2020, Frontiers in Physiology.

CDKN1B/p27 is localized in mitochondria and improves respiration-dependent processes in the cardiovascular system—New mode of action for caffeine

K. Köhrer, M. Grandoch, J. Fischer, 2018, PLoS biology.

The aryl hydrocarbon receptor promotes aging phenotypes across species

A. Eckers, C. Esser, N. Ventura, 2016, Scientific Reports.

2-Methyl-1,4-naphthoquinone, vitamin K(3), decreases gap-junctional intercellular communication via activation of the epidermal growth factor receptor/extracellular signal-regulated kinase cascade.

L. Klotz, H. Sies, P. Gerber, 2002, Cancer research.

Mitochondrial Telomerase Reverse Transcriptase Protects From Myocardial Ischemia/Reperfusion Injury by Improving Complex I Composition and Function

M. Grandoch, G. Heusch, K. Unfried, 2021, Circulation.

Ultraviolet A induced modulation of gap junctional intercellular communication by P38 MAPK activation in human keratinocytes

M. Picardo, H. Sies, S. Briganti, 2008, Experimental dermatology.

Non-Antioxidant Properties of Carotenoids

M. Polidori, W. Stahl, N. Ale-Agha, 2002, Biological chemistry.

(-)-Epicatechin effects in rat liver epithelial cells: stimulation of gap junctional communication and counteraction of its loss due to the tumor promoter 12-O-tetradecanoylphorbol-13-acetate.

W. Stahl, H. Sies, N. Ale-Agha, 2002, Biochemical pharmacology.

CD133+ hepatic stellate cells are progenitor cells.

D. Häussinger, H. Klonowski-Stumpe, V. Keitel, 2007, Biochemical and biophysical research communications.

Hydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system.

P. Eaton, S. Oka, J. Burgoyne, 2013, Antioxidants & redox signaling.

Endothelial hyaluronan synthase 3 aggravates acute colitis in an experimental model of inflammatory bowel disease.

M. Grandoch, J. Fischer, U. Flögel, 2021, Matrix biology : journal of the International Society for Matrix Biology.

Non-Canonical Activation of the Epidermal Growth Factor Receptor by Carbon Nanoparticles

A. Kraegeloh, K. Unfried, J. Haendeler, 2018, Nanomaterials.

Signalling-Dependent Adverse Health Effects of Carbon Nanoparticles Are Prevented by the Compatible Solute Mannosylglycerate (Firoin) In Vitro and In Vivo

C. Suschek, K. Unfried, C. Albrecht, 2014, PloS one.

The Anti-Apoptotic Properties of APEX1 in the Endothelium Require the First 20 Amino Acids and Converge on Thioredoxin-1.

A. Eckers, N. Leitinger, J. Altschmied, 2017, Antioxidants & redox signaling.

Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to carbon or silica-based nanoparticles

L. Klotz, C. Albrecht, N. Ale-Agha, 2010, Biological chemistry.

Modulation of homologous gap junctional intercellular communication of human dermal fibroblasts via a paracrine factor(s) generated by squamous tumor cells.

H. Steinbrenner, H. Sies, P. Brenneisen, 2003, Carcinogenesis.

Non-canonical functions of Telomerase Reverse Transcriptase – Impact on redox homeostasis

J. Rosen, J. Altschmied, J. Haendeler, 2020, Redox biology.

Lycopene oxidation product enhances gap junctional communication.

W. Stahl, H. Sies, H. Wollersen, 2003, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

Unhealthy diet and ultrafine carbon black particles induce senescence and disease associated phenotypic changes

K. Unfried, J. Altschmied, J. Haendeler, 2013, Experimental Gerontology.

Selenoprotein T Protects Endothelial Cells against Lipopolysaccharide-Induced Activation and Apoptosis

J. Altschmied, H. Schaal, J. Haendeler, 2021, Antioxidants.

Cellular functions of the dual-targeted catalytic subunit of telomerase, telomerase reverse transcriptase — Potential role in senescence and aging

J. Altschmied, J. Haendeler, Nadine Dyballa-Rukes, 2014, Experimental Gerontology.

Role of Telomerase in the Cardiovascular System

J. Altschmied, J. Haendeler, Stefanie Kohlgrüber, 2016, Genes.

HuR regulates gap junctional intercellular communication by controlling β‐catenin levels and adherens junction integrity

Kotb Abdelmohsen, M. Gorospe, L. Klotz, 2009, Hepatology.

Epidermal growth factor- and stress-induced loss of gap junctional communication is mediated by ERK-1/ERK-2 but not ERK-5 in rat liver epithelial cells.

L. Klotz, Elisabeth Sauerbier, H. Sies, 2007, Biochemical and biophysical research communications.

Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to quartz particles.

L. Klotz, C. Albrecht, N. Ale-Agha, 2009, Biochemical and biophysical research communications.

Induction of a senescent like phenotype and loss of gap junctional intercellular communication by carbon nanoparticle exposure of lung epithelial cells

K. Unfried, J. Altschmied, J. Haendeler, 2019, Experimental Gerontology.

High Concentration of Low-Density Lipoprotein Results in Disturbances in Mitochondrial Transcription and Functionality in Endothelial Cells

W. Kaisers, J. Altschmied, H. Schaal, 2019, Oxidative medicine and cellular longevity.