Benjamin Wayment

发表

Contribution of Impaired Myocardial Insulin Signaling to Mitochondrial Dysfunction and Oxidative Stress in the Heart

E. Abel, S. Boudina, X. Sheng, 2009, Circulation.

PGC-1&bgr; Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy

Torsten Doenst, Antonio Vidal-Puig, C. Lelliott, 2011, Circulation research.

Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome.

E. Abel, Curtis D. Olsen, S. Litwin, 2015, Journal of molecular and cellular cardiology.

A conserved role for phosphatidylinositol 3-kinase but not Akt signaling in mitochondrial adaptations that accompany physiological cardiac hypertrophy.

M. Birnbaum, S. Izumo, E. Abel, 2007, Cell metabolism.

Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses.

E. Abel, B. O’Neill, S. Litwin, 2017, American journal of physiology. Heart and circulatory physiology.

Genetic loss of insulin receptors worsens cardiac efficiency in diabetes.

B. Weimer, E. Abel, S. Boudina, 2012, Journal of molecular and cellular cardiology.

Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarction.

E. Abel, Curtis D. Olsen, S. Litwin, 2009, Journal of molecular and cellular cardiology.

Insulin-like growth factor I receptor signaling is required for exercise-induced cardiac hypertrophy.

D. Leroith, M. Holzenberger, E. Abel, 2008, Molecular endocrinology.

Mechanistic Target of Rapamycin (Mtor) Is Essential for Murine Embryonic Heart Development and Growth

G. Thomas, S. Kozma, E. Abel, 2013, PloS one.

Type 1 Diabetic Akita Mouse Hearts Are Insulin Sensitive but Manifest Structurally Abnormal Mitochondria That Remain Coupled Despite Increased Uncoupling Protein 3

Sheldon E. Litwin, E. Dale Abel, E. Abel, 2008, Diabetes.

Integrative PGC-1 (cid:1) Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy hypertrophy by preserving glucose metabolism and preventing oxidative stress.

C. Lelliott, E. Abel, A. Vidal-Puig, 2011 .

Contribution of Impaired Myocardial Insulin Signaling to Mitochondrial Dysfunction and Oxidative Stress in the Heart

S. Boudina, X. Sheng, S. Litwin, 2009, Circulation.

Integrative PGC-1 (cid:1) Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy hypertrophy by preserving glucose metabolism and preventing oxidative stress.

C. Lelliott, G. Medina-Gómez, Curtis D. Olsen, 2011 .

Abstract 3559: Insulin Receptor Substrates (IRS) Signaling are Essential Regulators of Mitochondrial Function and Cardiomyocyte Survival

M. White, X. Dong, E. Abel, 2008 .