Hanning You

发表

c‐Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinoma

W. Ding, C. Rountree, H. You, 2011, Hepatology.

CD133+ liver cancer stem cells from methionine adenosyl transferase 1A–deficient mice demonstrate resistance to transforming growth factor (TGF)‐β–induced apoptosis

Shelly C. Lu, M. Mouzaki, J. Mato, 2009, Hepatology.

miR-200b restoration and DNA methyltransferase inhibitor block lung metastasis of mesenchymal-phenotype hepatocellular carcinoma

R. Albert, J. Liao, B. Stiles, 2012, Oncogenesis.

Diabetic nephropathy is resistant to oral L-arginine or L-citrulline supplementation.

S. Morris, T. Cooper, A. Awad, 2014, American journal of physiology. Renal physiology.

Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor‐β

W. Ding, C. Rountree, H. You, 2010, Hepatology.

Epithelial‐to‐mesenchymal transition of murine liver tumor cells promotes invasion

Shelly C. Lu, B. Stiles, W. Ding, 2010, Hepatology.

The EGFR/ErbB3 Pathway Acts as a Compensatory Survival Mechanism upon c-Met Inhibition in Human c-Met+ Hepatocellular Carcinoma

S. Steinway, W. Ding, C. Rountree, 2015, PloS one.

Macrophage-derived Tumor Necrosis Factor-α mediates diabetic renal injury

J. Vacher, S. Nedospasov, W. B. Reeves, 2015, Kidney international.

Delayed Treatment with a Small Pigment Epithelium Derived Factor (PEDF) Peptide Prevents the Progression of Diabetic Renal Injury

A. Gvritishvili, T. Cooper, J. Tombran-Tink, 2015, PloS one.

Protective role of small pigment epithelium-derived factor (PEDF) peptide in diabetic renal injury.

A. Gvritishvili, W. B. Reeves, T. Cooper, 2013, American journal of physiology. Renal physiology.

Macrophages directly mediate diabetic renal injury.

T. Cooper, A. Awad, H. You, 2013, American journal of physiology. Renal physiology.

Arginase inhibition mediates renal tissue protection in diabetic nephropathy by a nitric oxide synthase 3-dependent mechanism.

S. Morris, Ting Gao, Timothy Cooper, 2013, Kidney international.

Arginase inhibition mediates renal tissue protection in diabetic nephropathy by a nitric oxide synthase 3-dependent mechanism

S. Morris, T. Cooper, A. Awad, 2013, Kidney international.

Distinct roles of arginases 1 and 2 in diabetic nephropathy.

J. Vacher, S. Morris, T. Cooper, 2017, American journal of physiology. Renal physiology.

Podocyte-specific chemokine (C-C motif) receptor 2 overexpression mediates diabetic renal injury in mice.

W. B. Reeves, T. Cooper, S. Bronson, 2017, Kidney international.

Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathy.

S. Morris, T. Cooper, A. Awad, 2015, American journal of physiology. Renal physiology.

Clinical application for the preservation of phospho-proteins through in-situ tissue stabilization

H. D. Vanguilder, W. Freeman, C. Rountree, 2010, Proteome Science.

c‐Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinoma

C. Rountree, Yixing Jiang, Hien Dang, 2011, Hepatology.

Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathy.

S. Morris, T. Cooper, Ting Gao, 2015, American journal of physiology. Renal physiology.

knocking down liver ccaat/enhancer-binding protein alpha by adenovirus-transduced silent interfering ribonucleic acid improves hepatic gluconeogenesis and lipid homeostasis in db/db mice.

P. MacLean, J. Schaack, J. Shao, 2006, Endocrinology.