B. Brown

发表

Incomplete lysis of thrombus in the moderate underlying atherosclerotic lesion during intracoronary infusion of streptokinase for acute myocardial infarction: quantitative angiographic observations.

E L Bolson, E. Bolson, B. G. Brown, 1986, Circulation.

Antioxidant Supplements Block the Response of HDL to Simvastatin-Niacin Therapy in Patients With Coronary Artery Disease and Low HDL

B. G. Brown, A. Chait, J. Albers, 2001, Arteriosclerosis, thrombosis, and vascular biology.

Usefulness of recanalization to luminal diameter of 0.6 millimeter or more with intracoronary streptokinase during acute myocardial infarction in predicting "normal" perfusion status, continued arterial patency and survival at one year.

E. Bolson, B. G. Brown, H. Dodge, 1987, The American journal of cardiology.

Lipid Lowering and Plaque Regression New Insights Into Prevention of Plaque Disruption and Clinical Events in Coronary Disease

J. Albers, B. Greg Brown, X. Zhao, 1993, Circulation.

Simultaneous low-density lipoprotein-C lowering and high-density lipoprotein-C elevation for optimum cardiovascular disease prevention with various drug classes, and their combinations: a meta-analysis of 23 randomized lipid trials

B. G. Brown, X. Zhao, Xue-Qiao Zhao, 2006, Current opinion in lipidology.

Effect of intensive compared with moderate lipid-lowering therapy on progression of coronary atherosclerosis: a randomized controlled trial.

Paul Schoenhagen, Tim Crowe, C. Grines, 2004, JAMA.

Is there any hope for vitamin E?

B. G. Brown, J. Crowley, B. Brown, 2005, JAMA.

Variability in the measurement of regional left ventricular wall motion from contrast angiograms.

E L Bolson, F H Sheehan, F. Sheehan, 1983, Circulation.

Influence of coronary collateral vessels on myocardial infarct size in humans. Results of phase I thrombolysis in myocardial infarction (TIMI) trial. The TIMI Investigators.

B. G. Brown, R. Bolli, R. Roberts, 1991, Circulation.

Effects of combination lipid therapy on coronary stenosis progression and clinical cardiovascular events in coronary disease patients with metabolic syndrome: a combined analysis of the Familial Atherosclerosis Treatment Study (FATS), the HDL-Atherosclerosis Treatment Study (HATS), and the Armed For

B. G. Brown, A. Chait, R. Krasuski, 2009, The American journal of cardiology.

Comparison of four methods of analysis of lipoprotein particle subfractions for their association with angiographic progression of coronary artery disease.

Paul T. Williams, R. Krauss, B. G. Brown, 2014, Atherosclerosis.

Antioxidant Vitamins and Lipid Therapy

B. G. Brown, A. Chait, X. Zhao, 2002 .

Myocardial Infarct Size in Humans

B. G. Brown, R. Bolli, R. Roberts, 2005 .

Effects of niacin combination therapy with statin or bile acid resin on lipoproteins and cardiovascular disease.

B. G. Brown, J. Brunzell, A. Zambon, 2014, The American journal of cardiology.

Maximizing coronary disease risk reduction using nicotinic acid combined with LDL-lowering therapy

B. G. Brown, B. Brown, 2005 .

Effects of Intensive Lipid‐Lowering Therapy on the Coronary Arteries of Asymptomatic Subjects With Elevated Apolipoprotein B

B. G. Brown, L. Fisher, J. Albers, 1993, Circulation.

Lipid Accumulation and Plaque Disruption: Processes Triggering Clinical Instability in Coronary Disease

B. G. Brown, J. Albers, X. Zhao, 1995 .

Arteriographic view of treatment to achieve regression of coronary atherosclerosis and to prevent plaque disruption and clinical cardiovascular events.

B. G. Brown, J. Albers, D. Sacco, 1993, British heart journal.

Regression of coronary artery disease as a result of intensive lipid-lowering therapy.

B. G. Brown, L. Garcia-Buñuel, J. Albers, 1991, The New England journal of medicine.

Secondary prevention of heart disease amongst patients with lipid abnormalities: practice and trends in the United States

B. G. Brown, J. Albers, X. Zhao, 1997, Journal of internal medicine.

Benefits of lipid-lowering therapy in men with elevated apolipoprotein B are not confined to those with very high low density lipoprotein cholesterol.

B. G. Brown, L. Fisher, J. Albers, 1994, Journal of the American College of Cardiology.

A maximum confidence approach for measuring progression and regression of coronary artery disease in clinical trials.

B. G. Brown, L. Fisher, D. Sacco, 1993, Circulation.

Atherosclerosis regression, plaque disruption, and cardiovascular events: a rationale for lipid lowering in coronary artery disease.

B. G. Brown, J. Albers, D. Sacco, 1993, Annual review of medicine.

Quantitative computer techniques for analyzing coronary arteriograms.

E L Bolson, E. Bolson, B. G. Brown, 1986, Progress in cardiovascular diseases.

Quantitative Coronary Arteriography: Estimation of Dimensions, Hemodynamic Resistance, and Atheroma Mass of Coronary Artery Lesions Using the Arteriogram and Digital Computation

E. Bolson, B. G. Brown, H. Dodge, 1977, Circulation.

Very intensive lipid therapy with lovastatin, niacin, and colestipol for prevention of death and myocardial infarction A 10-year Familial Atherosclerosis Treatment Study follow-up

B. G. Brown, X. Zhao, A. Dowdy, 1998 .

Niacin plus Simvastatin Reduces Coronary Stenosis Progression Among Patients with Metabolic Syndrome Despite a Modest Increase in Insulin Resistance: A Subgroup Analysis of the HDL-Atherosclerosis Treatment Study (HATS).

B. G. Brown, A. Chait, X. Zhao, 2007, Journal of clinical lipidology.

Altered particle size distribution of apolipoprotein A-I-containing lipoproteins in subjects with coronary artery disease.

B. G. Brown, J. Albers, M. Cheung, 1991, Journal of lipid research.

Effects of lowering elevated LDL cholesterol on the cardiovascular risk of lipoprotein(a).

B. G. Brown, S. Marcovina, J. Albers, 1995, JAMA.

Tracking atherosclerosis regression: a clinical tool in preventive cardiology.

L. Shaw, Z. Fayad, Allen J. Taylor, 2005, Atherosclerosis.

Intravenous dipyridamole combined with isometric handgrip for near maximal acute increase in coronary flow in patients with coronary artery disease.

E. Bolson, B. G. Brown, H. Dodge, 1981, The American journal of cardiology.

Noninvasive detection and localization of coronary stenoses in patients: comparison of resting dipyridamole and exercise thallium-201 myocardial perfusion imaging.

B. G. Brown, H. Hecht, M. Josephson, 1982, American heart journal.

Reflex constriction of significant coronary stenosis as a mechanism contributing to ischemic left ventricular dysfunction during isometric exercise.

E. Bolson, B. G. Brown, H. Dodge, 1984, Circulation.

Importance of endothelial function in mediating the benefits of lipid-lowering therapy.

B. G. Brown, X. Zhao, X. Q. Zhao, 1998, The American journal of cardiology.

34th Bethesda Conference: Can atherosclerosis imaging techniques improve the detection of patients at risk for ischemic heart disease?

L. Shaw, D. Mark, R. Virmani, 2003 .

Arteriographic Assessment of Coronary Atherosclerosis

E. Bolson, B. G. Brown, H. Dodge, 2005 .

Arteriographic assessment of coronary atherosclerosis. Review of current methods, their limitations, and clinical applications.

E L Bolson, E. Bolson, B. G. Brown, 1982, Arteriosclerosis.

A direct comparison of intravascular ultrasound and quantitative coronary arteriography: implications for measures of atherosclerosis as clinical surrogates.

B. G. Brown, B. Brown, 2007, Circulation.

Dynamic mechanisms in human coronary stenosis.

E L Bolson, E. Bolson, B. G. Brown, 1984, Circulation.

Does ENHANCE diminish confidence in lowering LDL or in ezetimibe?

Allen J. Taylor, B. G. Brown, B. Brown, 2008, The New England journal of medicine.

Improved graft patency in patients treated with platelet-inhibiting therapy after coronary bypass surgery.

B. G. Brown, T. Derouen, J. Carey, 1985, Circulation.

Lumen Diameter of Normal Human Coronary Arteries: Influence of Age, Sex, Anatomic Variation, and Left Ventricular Hypertrophy or Dilation

E. Bolson, J. Dodge, B. G. Brown, 1992, Circulation.

Is intravascular ultrasound the gold standard surrogate for clinically relevant atherosclerosis progression?

B. G. Brown, X. Zhao, B. Brown, 2007, Journal of the American College of Cardiology.

Effects of niacin on glucose levels, coronary stenosis progression, and clinical events in subjects with normal baseline glucose levels (<100 mg/dl): a combined analysis of the Familial Atherosclerosis Treatment Study (FATS), HDL-Atherosclerosis Treatment Study (HATS), Armed Forces Regression Study

B. G. Brown, X. Zhao, B. A. Phan, 2013, The American journal of cardiology.

ApoA-I(Milano)/phospholipid complex: clinical implications of dose-response studies in rabbit atherosclerosis with intravascular ultrasound and magnetic resonance imaging.

B. G. Brown, X. Zhao, B. Brown, 2008, Journal of the American College of Cardiology.

Phospholipid transfer protein activity is associated with inflammatory markers in patients with cardiovascular disease.

B. G. Brown, K. O’Brien, J. Albers, 2006, Biochimica et biophysica acta.

Molecular genetics of apolipoproteins and coronary heart disease.

B. G. Brown, A. Motulsky, S. Deeb, 1986, Cold Spring Harbor symposia on quantitative biology.

Intimal fibromuscular hyperplasia at the venous anastomosis of PTFE grafts in hemodialysis patients. Clinical, immunocytochemical, light and electron microscopic assessment.

B. G. Brown, R. Sigley, S. Nicholls, 1989, Circulation.

Relationship between plasma phospholipid transfer protein activity and HDL subclasses among patients with low HDL and cardiovascular disease.

B. G. Brown, J. Albers, M. Cheung, 1999, Atherosclerosis.

Should both HDL-C and LDL-C be targets for lipid therapy? A review of current evidence.

B. G. Brown, X. Zhao, Xue-Qiao Zhao, 2007, Journal of clinical lipidology.

Coronary Collateral Size, Flow Capacity, and Growth: Estimates From the Angiogram in Patients With Obstructive Coronary Disease

B. G. Brown, B Greg Brown, Julia Rockstroh, 2002, Circulation.

Regression of atherosclerosis. Does it occur and does it have clinical meaning?

B. G. Brown, J. Albers, X. Q. Zhao, 1995, European heart journal.

Postoperative treadmill performance and graft patency after myocardial revascularization.

B. G. Brown, J. Carey, R. Cukingnan, 1983, The Annals of thoracic surgery.

Effectiveness of revascularization in the Emory angioplasty versus surgery trial. A randomized comparison of coronary angioplasty with bypass surgery.

H. Barnhart, B. G. Brown, W. Weintraub, 1996, Circulation.

The Mechanisms of Nitroglycerin Action: Stenosis Vasodilatation as a Major Component of the Drug Response

E. Bolson, B. G. Brown, H. Dodge, 1981, Circulation.

Lipid-lowering therapy for the stabilization of the vulnerable atherosclerotic plaque

B. Brown, 1993 .

The Fate and Fibrogenic Potential of Subintimal Implants of Crystalline Lipid in the Canine Aorta: Quantitative Histological and Autoradiographic Studies

D. L. Fry, B. G. Brown, B. Brown, 1978, Circulation research.

Lipid-lowering therapy prevents instability among mild and moderate atherosclerotic plaques: Observations from fats

L. Fisher, J. Albers, X. Zhao, 1992 .

Unstable angina: guidelines for therapy based on the last decade of clinical observations.

B. G. Brown, H. Dodge, B. Brown, 1982, Annals of internal medicine.

Perianastomotic arteriosclerosis in grafted human coronary arteries: Prevention with platelet-inhibiting therapy

E. Bolson, B. G. Brown, H. Dodge, 1982 .

Improved graft patency with antiplatelet drugs in patients treated for one year following coronary bypass surgery

J. Roth, B. G. Brown, J. Wittig, 1981 .

In vitro and in situ magnetic resonance imaging signal features of atherosclerotic plaque-associated lipids.

C Yuan, C. Yuan, T. Hatsukami, 1997, Arteriosclerosis, thrombosis, and vascular biology.

PATHOPHYSIOLOGY AND NATURAL HISTORY

E. Bolson, B. G. Brown, H. Dodge, 2005 .

Moderate dose, three-drug therapy with niacin, lovastatin, and colestipol to reduce low-density lipoprotein cholesterol <100 mg/dl in patients with hyperlipidemia and coronary artery disease.

L A Hillger, J J Albers, B. G. Brown, 1997, The American journal of cardiology.

Quantitative and qualitative coronary arteriography

Edward L. Bolson, Harold T. Dodge, E. Bolson, 1991 .

Progression of coronary atherosclerosis in patients with probable familial hypercholesterolemia. Quantitative arteriographic assessment of patients in NHLBI type II study.

B. G. Brown, H. Dodge, S. Kelsey, 1989, Arteriosclerosis.

Reproducibility of carotid atherosclerotic lesion type characterization using high resolution multicontrast weighted cardiovascular magnetic resonance.

Chun Yuan, Niranjan Balu, Baocheng Chu, 2006, Journal of cardiovascular magnetic resonance : official journal of the Society for Cardiovascular Magnetic Resonance.

Intrathoracic spatial location of specified coronary segments on the normal human heart. Applications in quantitative arteriography, assessment of regional risk and contraction, and anatomic display.

E. Bolson, J. Dodge, B. G. Brown, 1988, Circulation.

Regression of Atherosclerosis in Man: Current Data and Their Methodological Limitations

E. Bolson, B. G. Brown, H. Dodge, 1984 .

Comparison of immediate invasive, delayed invasive, and conservative strategies after tissue-type plasminogen activator. Results of the Thrombolysis in Myocardial Infarction (TIMI) Phase II-A trial.

J. Gore, W. Rogers, F. Sheehan, 1990, Circulation.

Use of niacin, statins, and resins in patients with combined hyperlipidemia.

B. G. Brown, J. Brunzell, A. Zambon, 1998, The American journal of cardiology.

Quantitative Coronary Arteriography

B. G. Brown, H. Dodge, B. H. Smith, 1979 .

Response of normal and diseased epicardial coronary arteries to vasoactive drugs: quantitative arteriographic studies.

B. G. Brown, B G Brown, B. Brown, 1985, The American journal of cardiology.

Diltiazem-induced blockade of sympathetically mediated constriction of normal and diseased coronary arteries: lack of epicardial coronary dilatory effect in humans.

B. G. Brown, H. Dodge, D. Stewart, 1984, Circulation.

Effects of verapamil on coronary hemodynamic function and vasomobility relative to its mechanism of antianginal action.

B. G. Brown, R. Petersen, C. Pierce, 1983, The American journal of cardiology.

Mechanism of action of verapamil in ischemic heart disease: observations on changes in systemic and coronary hemodynamics and coronary vasomobility.

H. Dodge, H. Hecht, P. Shah, 1980, Clinical and investigative medicine. Medecine clinique et experimentale.

Levels of Cholesterol in Small LDL Particles Predict Atherosclerosis Progression and Incident CHD in the HDL-Atherosclerosis Treatment Study (HATS)

Paul T. Williams, R. Krauss, B. G. Brown, 2013, PloS one.

Dynamic responses of human coronary stenoses.

B. G. Brown, B. Brown, 1986, Australian and New Zealand journal of medicine.

Coronary Angioplasty Performed Within the Thrombolysis in Myocardial Infarction II Study

E. Braunwald, B. G. Brown, D. Baim, 1992, Circulation.

Plaque regression and clinical events in coronary disease

J. Albers, X. Zhao, B. Brown, 1992 .

Percutaneous transluminal coronary angioplasty and subsequent restenosis: quantitative and qualitative methodology for their assessment.

E. Bolson, B. G. Brown, H. Dodge, 1987, The American journal of cardiology.