Madhulika Tripathi

P. Kakkar, M. Tripathi, B. Singh, 2009, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

Early induction of hepatic deiodinase type 1 inhibits hepatosteatosis during NAFLD progression

B. Singh, A. Boelen, P. Yen, 2021, bioRxiv.

Spermidine-mediated hypusination of translation factor EIF5A improves mitochondrial fatty acid oxidation and prevents non-alcoholic steatohepatitis progression

E. Petretto, A. Diehl, P. Yen, 2022, Nature Communications.

Region‐specific dysregulation of glycogen synthase kinase‐3β and β‐catenin in the postmortem brains of subjects with bipolar disorder and schizophrenia

G. Pandey, H. Rizavi, G. Pandey, 2015, Bipolar disorders.

IL-11 neutralising therapies target hepatic stellate cell-induced liver inflammation and fibrosis in NASH

S. Cook, S. Albani, S. Chothani, 2018, bioRxiv.

Gut microbiota and their metabolites in the progression of non-alcoholic fatty liver disease

B. Singh, P. Yen, R. Sinha, 2021, Hepatoma research.

B. Bay, V. Giguère, B. Singh, 2021, bioRxiv.

Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH

S. Cook, S. Chothani, B. Singh, 2021, Nature Communications.

Inhibition of an immunometabolic axis of mTORC1 activation extends mammalian healthspan

N. Hübner, D. Carling, D. Withers, 2023, bioRxiv.

Natural Terpenes Prevent Mitochondrial Dysfunction, Oxidative Stress and Release of Apoptotic Proteins during Nimesulide-Hepatotoxicity in Rats

B. Singh, B. Chaudhari, P. Pandey, 2012, PloS one.

Pharmacological inhibition of CFTR attenuates nonalcoholic steatohepatitis (NASH) progression in mice.

Pratima Gupta, Sangam Rajak, B. Chakravarti, 2023, Biochimica et biophysica acta. Molecular basis of disease.

Decreased autophagy and fuel switching occur in a senescent hepatic cell model system

B. Singh, P. Yen, Jin Zhou, 2020, Aging.

Alteration in mitochondrial thiol enhances calcium ion dependent membrane permeability transition and dysfunction in vitro: a cross-talk between mtThiol, Ca2+, and ROS

B. Singh, P. Pandey, P. Kakkar, 2011, Molecular and Cellular Biochemistry.

Nimesulide aggravates redox imbalance and calcium dependent mitochondrial permeability transition leading to dysfunction in vitro.

B. Singh, P. Pandey, P. Kakkar, 2010, Toxicology.

Development of an in vitro senescent hepatic cell model for metabolic studies in aging

B. Singh, P. Yen, Jin Zhou, 2020, bioRxiv.

Role of AKR1B10 and AKR1B8 in the pathogenesis of non-alcoholic steatohepatitis (NASH) in mouse.

Pratima Gupta, Sangam Rajak, B. Singh, 2021, Biochimica et biophysica acta. Molecular basis of disease.

Systemic impacts of metabolic dysfunction-associated steatotic liver disease (MASLD) and metabolic dysfunction-associated steatohepatitis (MASH) on heart, muscle, and kidney related diseases

Reddemma Sandireddy, B. Singh, Jatin Behari, 2024, Frontiers in cell and developmental biology.

Vitamin B12 and folate decrease inflammation and fibrosis in NASH by preventing Syntaxin 17 homocysteinylation.

A. Suzuki, B. Singh, P. Yen, 2022, Journal of hepatology.

Inhibition of IL-11 signalling extends mammalian healthspan and lifespan

S. Chothani, S. Schafer, B. Corden, 2024, Nature.

Esrra regulates Rplp1-mediated translation of lysosome proteins suppressed in non-alcoholic steatohepatitis and reversed by alternate day fasting

P. Chow, Jin Zhou, Reddemma Sandireddy, 2021, bioRxiv.

Lower hepatic CBS and PEMT expression in advanced NAFLD: inferencing strategies to lower homocysteine with a mathematical model

M. Abdelmalek, B. Singh, Madhulika Tripathi, 2024, Metabolism and Target Organ Damage.