Karin Hehenberger

Impaired proliferation and increased L‐lactate production of dermal fibroblasts in the GK‐rat, a spontaneous model of non‐insulin dependent diabetes mellitus

A. Hansson, K. Hehenberger, K. Brismar, 1999, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

Dose‐dependent hyperbaric oxygen stimulation of human fibroblast proliferation

K. Hehenberger, K. Brismar, G. Kratz, 1997, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

Dose-dependent hyperbaricoxygen stimulation ofhuman fibroblast proliferation

K. Brismar, G. Kratz, F. Lind, 1997 .

Fibroblasts derived from human chronic diabetic wounds have a decreased proliferation rate, which is recovered by the addition of heparin.

A. Hansson, K. Hehenberger, K. Brismar, 1998, Journal of dermatological science (Amsterdam).

Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of L‐lactate

A. Hansson, K. Hehenberger, K. Brismar, 1998, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

High glucose‐induced growth factor resistance in human fibroblasts can be reversed by antioxidants and protein kinase C‐inhibitors

A. Hansson, Karin Hehenberger, 1997, Cell biochemistry and function.

Long‐term treatment of swiss 3T3 fibroblasts with dexamethasone attenuates MAP kinase activation induced by insulin‐like growth factor‐I (IGF‐I)

A. Hansson, K. Hehenberger, M. Thorén, 1996, Cell biochemistry and function.