T. Barrett

发表

Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs

Jianxin Shi, Susmita Datta, Aribert Rothenberger, 2013, Nature Genetics.

Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4

Manuel A. R. Ferreira, John P. Rice, Edward G. Jones, 2011, Nature Genetics.

Joint Analysis of Psychiatric Disorders Increases Accuracy of Risk Prediction for Schizophrenia, Bipolar Disorder, and Major Depressive Disorder

Laura J. Scott, Naomi R. Wray, Margaret A. Pericak-Vance, 2015, American journal of human genetics.

Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways

Laura J. Scott, Naomi R. Wray, Margaret A. Pericak-Vance, 2015, Nature Neuroscience.

Evidence that a single nucleotide polymorphism in the promoter of the G protein receptor kinase 3 gene is associated with bipolar disorder

J. Kelsoe, P. Keck, S. McElroy, 2003, Molecular Psychiatry.

Singleton deletions throughout the genome increase risk of bipolar disorder

John P. Rice, Daniel L. Koller, S. Christian, 2009, Molecular Psychiatry.

Infusion of platelet-derived growth factor or basic fibroblast growth factor induces selective glomerular mesangial cell proliferation and matrix accumulation in rats.

C. Alpers, J. Floege, T. Barrett, 1993, The Journal of clinical investigation.

sis (platelet-derived growth factor B chain) gene transcript levels are elevated in human atherosclerotic lesions compared to normal artery.

T. Barrett, E. Benditt, T. Barrett, 1987, Proceedings of the National Academy of Sciences of the United States of America.

Expression of the sis gene by endothelial cells in culture and in vivo.

S. Schwartz, C. Gajdusek, J. McDougall, 1984, Proceedings of the National Academy of Sciences of the United States of America.

A significant part of macrophage-derived growth factor consists of at least two forms of PDGF

R. Ross, E. Raines, D. Madtes, 1985, Cell.

Combined analysis from eleven linkage studies of bipolar disorder provides strong evidence of susceptibility loci on chromosomes 6q and 8q.

M. Daly, S. Cichon, M. Rietschel, 2005, American journal of human genetics.

Genome-wide significant association between a ‘negative mood delusions' dimension in bipolar disorder and genetic variation on chromosome 3q26.1

John P. Rice, Daniel L. Koller, Pamela B. Mahon, 2012, Translational Psychiatry.

Genome-Wide Association of Bipolar Disorder Suggests an Enrichment of Replicable Associations in Regions near Genes

Daniel L. Koller, Pamela B. Mahon, F. McMahon, 2011, PLoS genetics.

CD40 signaling activates CD11a/CD18 (LFA-1)-mediated adhesion in B cells.

E. Clark, T. Barrett, G. Shu, 1991, Journal of immunology.

Genome-wide association study of 40,000 individuals identifies two novel loci associated with bipolar disorder

Janice M. Fullerton, Daniel L. Koller, Pamela B. Mahon, 2016, bioRxiv.

Particulate-cell interactions and pulmonary cytokine expression.

J. Finkelstein, T. Barrett, C. Johnston, 1997, Environmental health perspectives.

Suggestive evidence for association of the circadian genes PERIOD3 and ARNTL with bipolar disorder

C. Nievergelt, J. Kelsoe, N. Schork, 2006, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.

Allele specific analysis of the ADRBK2 gene in lymphoblastoid cells from bipolar disorder patients.

E. Turner, M. McCarthy, J. Kelsoe, 2010, Journal of psychiatric research.

Promoter Variant in the GRK3 Gene Associated with Bipolar Disorder Alters Gene Expression

J. Kelsoe, T. Barrett, Xianjin Zhou, 2008, Biological Psychiatry.

Association analysis of GRK3 gene promoter variants in cocaine abuse

D. Collier, J. Kelsoe, G. Breen, 2007, Psychiatric genetics.

Regional expression of the platelet-derived growth factor and its receptors in a primate graft model of vessel wall assembly.

R. Ross, J. Wilcox, E. Raines, 1993, The Journal of clinical investigation.

psychosis: a convergent functional genomics approach Identifying a series of candidate genes for mania and

J. Kelsoe, D. Segal, H. Akil, 2005 .

Signaling through CD19, Fc receptors or transforming growth factor‐β: each inhibits the activation of resting human B cells differently

E. Clark, T. Barrett, K. Draves, 1990, European journal of immunology.

Platelet-derived growth factor gene expression in human atherosclerotic plaques and normal artery wall.

T. Barrett, E. Benditt, T. Barrett, 1988, Proceedings of the National Academy of Sciences of the United States of America.

The Pharmacogenetics of Lithium Response Depends upon Clinical Co-Morbidity

J. Kelsoe, T. Barrett, Tatyana Shehktman, 2012, Molecular Diagnosis & Therapy.

Viruses in the etiology of atherosclerosis.

J. McDougall, T. Barrett, E. Benditt, 1983, Proceedings of the National Academy of Sciences of the United States of America.

Further evidence for association of GRK3 to bipolar disorder suggests a second disease mutation

E. Eskin, C. Nievergelt, J. Kelsoe, 2007, Psychiatric genetics.

Platelet-derived growth factor gene expression in human atherosclerotic plaques and normal artery wall (atherogenesis/smooth muscle cell/macrophage/endothelial cell/cell proliferation)

E. Benditt, T. Barrett, 2016 .

Regulation of platelet‐derived growth factor receptor expression by cell context overrides regulation by cytokines

T. Barrett, D. Bowen-Pope, R. Seifert, 1996, Journal of cellular physiology.

Mitogenic effect of platelet‐derived growth factor in human glomerular mesangial cells: modulation and/or suppression by inflammatory cytokines

J. Floege, K. Resch, T. Barrett, 1991, Clinical and experimental immunology.

psychosis: a convergent functional genomics approach Identifying a series of candidate genes for mania and

J. Kelsoe, D. Segal, H. Akil, 2005 .

Differential Regulation of Immediate-Early Gene Expression in the Prefrontal Cortex of Rats with a High vs Low Behavioral Response to Methamphetamine

J. Kelsoe, D. Segal, P. Shilling, 2006, Neuropsychopharmacology.