Tien‐Min Lin

发表

Enzymatically Biodegradable Polyrotaxane-Deferoxamine Conjugates for Iron Chelation.

May P. Xiong, Zhi Liu, Max Purro, 2016, ACS applied materials & interfaces.

In utero exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin causes accelerated terminal differentiation in fetal mouse skin.

R. Peterson, J. Loertscher, B. Allen-Hoffmann, 2002, Toxicological sciences : an official journal of the Society of Toxicology.

AHR signaling in prostate growth, morphogenesis, and disease.

R. Peterson, C. Vezina, Chad M. Vezina, 2009, Biochemical pharmacology.

A Constitutively Active Arylhydrocarbon Receptor Induces Growth Inhibition of Jurkat T Cells through Changes in the Expression of Genes Related to Apoptosis and Cell Cycle Arrest*

C. Tohyama, Y. Fujii‐Kuriyama, R. Peterson, 2004, Journal of Biological Chemistry.

Expression of novel genes linked to the androgen-induced, proliferative shutoff in prostate cancer cells

C. Sonnenschein, A. Soto, J. Szelei, 1997, The Journal of Steroid Biochemistry and Molecular Biology.

Androgenic regulation of ventral epithelial bud number and pattern in mouse urogenital sinus

Robert W. Moore, R. Peterson, Lisa L. Abler, 2010, Developmental dynamics : an official publication of the American Association of Anatomists.

WNT5A selectively inhibits mouse ventral prostate development.

Robert W. Moore, S. Chiu, R. Peterson, 2008, Developmental biology.

The aryl hydrocarbon receptor inhibits prostate carcinogenesis in TRAMP mice.

R. Cardiff, R. Peterson, Tien-Min Lin, 2006, Carcinogenesis.

2,3,7,8-tetrachlorodibenzo-p-dioxin inhibits prostatic epithelial bud formation by acting directly on the urogenital sinus.

Robert W. Moore, R. Peterson, R. Albrecht, 2004, The Journal of urology.

Region-specific inhibition of prostatic epithelial bud formation in the urogenital sinus of C57BL/6 mice exposed in utero to 2,3,7,8-tetrachlorodibenzo-p-dioxin.

Robert W. Moore, R. Peterson, R. Albrecht, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

Beta-catenin (CTNNB1) induces Bmp expression in urogenital sinus epithelium and participates in prostatic bud initiation and patterning.

Kimberly P Keil, M. Taketo, Xin Sun, 2013, Developmental biology.

Nanogel-DFO conjugates as a model to investigate pharmacokinetics, biodistribution, and iron chelation in vivo.

May P. Xiong, Zhi Liu, Yan Wang, 2018, International journal of pharmaceutics.

2,3,7,8-Tetrachlorodibenzo-p-dioxin has both pro-carcinogenic and anti-carcinogenic effects on neuroendocrine prostate carcinoma formation in TRAMP mice.

Robert W. Moore, S. Safe, R. Peterson, 2016, Toxicology and applied pharmacology.

Organic Chemicals Adsorbed onto Diesel Exhaust Particles Directly Alter the Differentiation of Fetal Thymocytes Through Arylhydrocarbon Receptor but Not Oxidative Stress Responses

C. Tohyama, R. Peterson, H. Nagai, 2006, Journal of immunotoxicology.

Aryl Hydrocarbon Receptor Regulates Growth, But Not Atresia, of Mouse Preantral and Antral Follicles1

R. Peterson, J. Flaws, C. Greenfeld, 2003, Biology of reproduction.

Behavioral Rhythmicity of Mice Lacking AhR and Attenuation of Light-Induced Phase Shift by 2,3,7,8-Tetrachlorodibenzo-p-Dioxin

R. Peterson, P. Cooke, M. Mukai, 2008, Journal of biological rhythms.

The aryl hydrocarbon receptor (AhR) inhibits vanadate-induced vascular endothelial growth factor (VEGF) production in TRAMP prostates.

Richard E Peterson, R. Peterson, Wayne A Fritz, 2008, Carcinogenesis.

Effects of aryl hydrocarbon receptor null mutation and in utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on prostate and seminal vesicle development in C57BL/6 mice.

Robert W. Moore, R. Peterson, K. Ko, 2002, Toxicological sciences : an official journal of the Society of Toxicology.

Estrogen signaling is not required for prostatic bud patterning or for its disruption by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

Robert W. Moore, R. Peterson, P. Cooke, 2009, Toxicology and applied pharmacology.

Abnormalities of sexual development in male rats with in utero and lactational exposure to the antiandrogenic plasticizer Di(2-ethylhexyl) phthalate.

Robert W. Moore, R. Peterson, K. Ko, 2001, Environmental health perspectives.

2,3,7,8-Tetrachlorodibenzo-p-dioxin has both pro-carcinogenic and anti-carcinogenic effects on neuroendocrine prostate carcinoma formation in TRAMP mice.

Robert W. Moore, S. Safe, R. Peterson, 2016, Toxicology and applied pharmacology.

The selective aryl hydrocarbon receptor modulator 6-methyl-1,3,8-trichlorodibenzofuran inhibits prostate tumor metastasis in TRAMP mice.

Richard E. Peterson, Stephen Safe, Robert W. Moore, 2009, Biochemical pharmacology.

Tetrachlorodibenzo‐p‐dioxin exposure alters radial arm maze performance and hippocampal morphology in female AhR +/– mice

R. Peterson, S. Schantz, B. Powers, 2004, Genes, brain, and behavior.

Lack of expression of EGF and TGF-alpha in the fetal mouse alters formation of prostatic epithelial buds and influences the response to TCDD.

R. Peterson, R. Albrecht, B. Abbott, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

2,3,7,8-tetrachlorodibenzo-p-dioxin inhibits luminal cell differentiation and androgen responsiveness of the ventral prostate without inhibiting prostatic 5alpha-dihydrotestosterone formation or testicular androgen production in rat offspring.

R. Peterson, B. L. Roman, H. Theobald, 2000, Toxicological sciences : an official journal of the Society of Toxicology.