N. B. Thomas

发表

Targeted therapies in myeloid leukemia.

G. Mufti, N. S. Thomas, R. Padua, 2004, Seminars in cancer biology.

BCL-2 and mutant NRAS interact physically and functionally in a mouse model of progressive myelodysplasia.

I. Weissman, D. Felsher, G. Mufti, 2007, Cancer research.

Mimicking the tumour microenvironment: three different co‐culture systems induce a similar phenotype but distinct proliferative signals in primary chronic lymphocytic leukaemia cells

C. Fegan, P. Brennan, E. Hamilton, 2012, British journal of haematology.

Genetic instability as an area of potential therapeutic intervention

G. Mufti, N. Lea, T. Gaymes, 2007 .

p130, p107, and pRb Are Differentially Regulated in Proliferating Cells and during Cell Cycle Arrest by α-Interferon*

A. Pizzey, N. S. Thomas, C. Williams, 1998, The Journal of Biological Chemistry.

Antiapoptotic Microenvironment of Acute Myeloid Leukemia

G. Mufti, D. Milojković, N. Westwood, 2004, The Journal of Immunology.

Myeloid Leukemia Antiapoptotic Microenvironment of Acute

G. Mufti, D. Milojković, N. S. Thomas, 2004 .

CDKN2B methylation status and isolated chromosome 7 abnormalities predict responses to treatment with 5-azacytidine

G. Mufti, K. Raj, C. Chronis, 2007, Leukemia.

Deep phenotyping of Tregs identifies an immune signature for idiopathic aplastic anemia and predicts response to treatment.

G. Mufti, N. Young, S. Heck, 2016, Blood.

Interaction with vascular endothelium enhances survival in primary chronic lymphocytic leukemia cells via NF-kappaB activation and de novo gene transcription.

G. Mufti, C. Fegan, C. Pepper, 2010, Cancer research.

Cancer esearch cular and Cellular Pathobiology raction with Vascular Endothelium Enhances Survival in ary Chronic Lymphocytic Leukemia Cells via NF-κ B R ivation and De novo Gene Transcription

C. Fegan, C. Pepper, N. S. Thomas, 2010 .

raction with Vascular Endothelium Enhances Survival in ary Chronic Lymphocytic Leukemia Cells via NF-κB

C. Fegan, C. Pepper, N. S. Thomas, 2010 .

Reactive oxygen species, DNA damage, and error-prone repair: a model for genomic instability with progression in myeloid leukemia?

G. Mufti, N. Lea, M. Pla, 2007, Cancer research.

Myeloid Leukemia? Repair: A Model for Genomic Instability with Progression in Reactive Oxygen Species, DNA Damage, and Error-Prone

G. Mufti, N. Lea, M. Pla, 2007 .

Cyclin D2 controls B cell progenitor numbers

E. Lam, G. Mufti, J. Glassford, 2003, Journal of leukocyte biology.

Immunotherapy of myeloid leukaemia

K. Mills, A. Mohamedali, B. Guinn, 2007, Cancer Immunology, Immunotherapy.

Microenvironment Produced by Acute Myeloid Leukemia Cells Prevents T Cell Activation and Proliferation by Inhibition of NF-κB, c-Myc, and pRb Pathways

G. Mufti, M. Arno, N. Lea, 2001, The Journal of Immunology.

JAK inhibition induces silencing of T Helper cytokine secretion and a profound reduction in T regulatory cells

G. Mufti, C. Harrison, D. Mclornan, 2015, British journal of haematology.

BCR-ABL and Interleukin 3 Promote Haematopoietic Cell Proliferation and Survival through Modulation of Cyclin D2 and p27Kip1 Expression*

E. Lam, E. Lam, C. Rivas, 2001, The Journal of Biological Chemistry.

The retinoblastoma family member, p107, is active in non‐dividing monocyte‐derived dendritic cells

K. Ardeshna, S. Devereux, F. Ahmed, 2001, British journal of haematology.

High efficiency protein transduction of quiescent and proliferating primary hematopoietic cells.

G. Mufti, N. Lea, S. Orr, 2003, Journal of biochemical and biophysical methods.