Jennifer E. Skeen

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak.

C. Thompson, N. Chandel, N. Hay, 2004, Molecular cell.

Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2.

Jennifer Skeen, N. Hay, K. Coleman, 2003, Genes & development.

Akt Activates the Mammalian Target of Rapamycin by Regulating Cellular ATP Level and AMPK Activity*

N. Sonenberg, N. Hay, V. Nogueira, 2005, Journal of Biological Chemistry.

Activation of Akt/Protein Kinase B Overcomes a G2/M Cell Cycle Checkpoint Induced by DNA Damage

P. Pandolfi, A. Gartel, E. Kandel, 2002, Molecular and Cellular Biology.

Inhibition of Chk1 by Activated PKB/Akt

N. Hay, Jennifer E Skeen, E. Shtivelman, 2004, Cell cycle.

Akt deficiency impairs normal cell proliferation and suppresses oncogenesis in a p53-independent and mTORC1-dependent manner.

N. Hay, H. Kiyokawa, V. Nogueira, 2006, Cancer cell.